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Citation: Pt. Sager, How to record high-quality monophasic action potential tracings, MONOPHASIC ACTION POTENTIALS: BRIDGING CELL AND BEDSIDE, 2000, pp. 121-134
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Citation: T. Satoh et Dp. Zipes, Tachycardia superimposed on bradycardia prolongs ventricular refractoriness and facilitates arrhythmia induction, MONOPHASIC ACTION POTENTIALS: BRIDGING CELL AND BEDSIDE, 2000, pp. 157-169
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Citation: I. Libbus et al., High-resolution measurement of ventricular repolarization using voltage-sensitive dyes, MONOPHASIC ACTION POTENTIALS: BRIDGING CELL AND BEDSIDE, 2000, pp. 291-306
Citation: M. Zabel et al., Effects of heart rate and antiarrhythmic drugs on dispersion of ventricular repolarization measured by multiple monophasic action potential recordings, MONOPHASIC ACTION POTENTIALS: BRIDGING CELL AND BEDSIDE, 2000, pp. 307-317
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Citation: M. Zabel et al., Validation of electrocardiographic variables of dispersion of ventricular repolarization with direct myocardial repolarization measurements, MONOPHASIC ACTION POTENTIALS: BRIDGING CELL AND BEDSIDE, 2000, pp. 337-350
Citation: L. Gepstein et al., Mapping of the spatial correlation between the activation and repolarization properties in the normal ventricle, MONOPHASIC ACTION POTENTIALS: BRIDGING CELL AND BEDSIDE, 2000, pp. 351
Citation: Mr. Franz et K. Bargheer, Abnormal relationship between activation and repolarization in human left ventricular hypertrophy, MONOPHASIC ACTION POTENTIALS: BRIDGING CELL AND BEDSIDE, 2000, pp. 363-378
Citation: L. Hondeghem, Frequency dependence of class I and class III antiarrhythmic agents explaining their antiarrhythmic and proarrhythmic properties, MONOPHASIC ACTION POTENTIALS: BRIDGING CELL AND BEDSIDE, 2000, pp. 381
Citation: Pf. Kirchhof et Cl. Fabritz, Effect of d-sotalol on the vulnerable window of ventricular fibrillation, and a definition of the "area of vulnerability", MONOPHASIC ACTION POTENTIALS: BRIDGING CELL AND BEDSIDE, 2000, pp. 395-421