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Citation: O. Chibafalek et al., THE MOLECULAR-BASIS OF DISEASE VARIABILITY AMONG CYSTIC-FIBROSIS PATIENTS CARRYING THE 3849-]T MUTATION(10 KB C), Genomics (San Diego, Calif.), 53(3), 1998, pp. 276-283
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Citation: M. Aviram et al., INTERACTIONS OF PLATELETS, MACROPHAGES, AND LIPOPROTEINS IN HYPERCHOLESTEROLEMIA - ANTIATHEROGENIC EFFECTS OF HMG-COA REDUCTASE INHIBITOR THERAPY, Journal of cardiovascular pharmacology, 31(1), 1998, pp. 39-45
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Citation: Pa. Belinky et al., THE ANTIOXIDATIVE EFFECTS OF THE ISOFLAVAN GLABRIDIN ON ENDOGENOUS CONSTITUENTS OF LDL DURING ITS OXIDATION, Atherosclerosis, 137(1), 1998, pp. 49-61
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Citation: Ly. Zhu et al., INDUCTION OF PPAR-GAMMA-1 EXPRESSION IN HUMAN THP-1 MONOCYTIC LEUKEMIA-CELLS BY 9-CIS-RETINOIC ACID IS ASSOCIATED WITH CELLULAR GROWTH SUPPRESSION, Biochemical and biophysical research communications (Print), 251(3), 1998, pp. 842-848
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Citation: H. Mandel et al., PLASMALOGEN PHOSPHOLIPIDS ARE INVOLVED IN HDL-MEDIATED CHOLESTEROL EFFLUX - INSIGHTS FROM INVESTIGATIONS WITH PLASMALOGEN-DEFICIENT CELLS, Biochemical and biophysical research communications (Print), 250(2), 1998, pp. 369-373
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Citation: T. Hayek et al., REDUCED PROGRESSION OF ATHEROSCLEROSIS IN APOLIPOPROTEIN E-DEFICIENT MICE FOLLOWING CONSUMPTION OF RED WINE, OR ITS POLYPHENOLS QUERCETIN OR CATECHIN, IS ASSOCIATED WITH REDUCED SUSCEPTIBILITY OF LDL TO OXIDATION AND AGGREGATION, Arteriosclerosis, thrombosis, and vascular biology, 17(11), 1997, pp. 2744-2752
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Citation: E. Kerem et al., A CYSTIC-FIBROSIS TRANSMEMBRANE CONDUCTANCE REGULATOR SPLICE VARIANT WITH PARTIAL PENETRANCE ASSOCIATED WITH VARIABLE CYSTIC-FIBROSIS PRESENTATIONS, American journal of respiratory and critical care medicine, 155(6), 1997, pp. 1914-1920
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Citation: B. Fuhrman et al., TOMATO LYCOPENE AND BETA-CAROTENE INHIBIT LOW-DENSITY-LIPOPROTEIN OXIDATION AND THIS EFFECT DEPENDS ON THE LIPOPROTEIN VITAMIN-E CONTENT, NMCD. Nutrition Metabolism and Cardiovascular Diseases, 7(6), 1997, pp. 433-443
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Citation: B. Fuhrman et al., INCREASED UPTAKE OF LDL BY OXIDIZED MACROPHAGES IS THE RESULT OF AN INITIAL ENHANCED LDL-RECEPTOR ACTIVITY AND OF A FURTHER PROGRESSIVE OXIDATION OF LDL, Free radical biology & medicine, 23(1), 1997, pp. 34-46
Citation: O. Hussein et al., DIETARY SELENIUM INCREASES CELLULAR GLUTATHIONE-PEROXIDASE ACTIVITY AND REDUCES THE ENHANCED SUSCEPTIBILITY TO LIPID-PEROXIDATION OF PLASMAAND LOW-DENSITY-LIPOPROTEIN IN KIDNEY-TRANSPLANT RECIPIENTS, Transplantation, 63(5), 1997, pp. 679-685
Citation: B. Fuhrman et al., LYCOPENE AND BETA-CAROTENE INHIBIT CHOLESTEROL-SYNTHESIS AND AUGMENT LDL RECEPTOR ACTIVITY IN MACROPHAGES, Atherosclerosis, 134(1-2), 1997, pp. 55-55
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Citation: Y. Levy et al., IS IT POSSIBLE TO REDUCE LDL OXIDIZABILITY IN NORMOLIPIDEMIC SUBJECTSBY ANTIOXIDANT AND FAT MODIFICATION OF EGGS, Atherosclerosis, 134(1-2), 1997, pp. 205-205