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Results:
1-24
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Results: 24
Authors:
Varma, N
Eberli, FR
Apstein, CS
Citation: N. Varma et al., Left ventricular diastolic dysfunction during demand ischemia: Rigor underlies increased stiffness without calcium-mediated tension. Amelioration by glycolytic substrate, J AM COL C, 37(8), 2001, pp. 2144-2153
Authors:
Podesser, BK
Siwik, DA
Eberli, FR
Sam, F
Ngoy, S
Lambert, J
Ngo, K
Apstein, CS
Colucci, WS
Citation: Bk. Podesser et al., ETA-receptor blockade prevents matrix metalloproteinase activation late postmyocardial infarction in the rat, AM J P-HEAR, 280(3), 2001, pp. H984-H991
Authors:
Guazzi, M
Brenner, DA
Apstein, CS
Saupe, KW
Citation: M. Guazzi et al., Exercise intolerance in rats with hypertensive heart disease is associatedwith impaired diastolic relaxation, HYPERTENSIO, 37(2), 2001, pp. 204-208
Authors:
Saupe, KW
Eberli, FR
Ingwall, JS
Apstein, CS
Citation: Kw. Saupe et al., Metabolic support as an adjunct to inotropic support in the hypoperfused heart, J MOL CEL C, 33(2), 2001, pp. 261-269
Authors:
Sam, F
Sawyer, DB
Xie, ZL
Chang, DLF
Ngoy, S
Brenner, DA
Siwik, DA
Singh, K
Apstein, CS
Colucci, WS
Citation: F. Sam et al., Mice lacking inducible nitric oxide synthase have improved left ventricular contractile function and reduced apoptotic cell death late after myocardial infarction, CIRCUL RES, 89(4), 2001, pp. 351-356
Authors:
Trueblood, NA
Xie, ZL
Communal, C
Sam, F
Ngoy, S
Liaw, L
Jenkins, AW
Wang, J
Sawyer, DB
Bing, OHL
Apstein, CS
Colucci, WS
Singh, K
Citation: Na. Trueblood et al., Exaggerated left ventricular dilation and reduced collagen deposition after myocardial infarction in mice lacking osteopontin, CIRCUL RES, 88(10), 2001, pp. 1080-1087
Authors:
Brenner, DA
Apstein, CS
Saupe, KW
Citation: Da. Brenner et al., Exercise training attenuates age-associated diastolic dysfunction in rats, CIRCULATION, 104(2), 2001, pp. 221-226
Authors:
Liao, RL
Jain, M
Teller, P
Connors, LH
Ngoy, S
Skinner, M
Falk, RH
Apstein, CS
Citation: Rl. Liao et al., Infusion of light chains from patients with cardiac amyloidosis causes diastolic dysfunction in isolated mouse hearts, CIRCULATION, 104(14), 2001, pp. 1594-1597
Authors:
Jain, M
DerSimonian, H
Brenner, DA
Ngoy, S
Teller, P
Edge, ASB
Zawadzka, A
Wetzel, K
Sawyer, DB
Colucci, WS
Apstein, CS
Liao, RL
Citation: M. Jain et al., Cell therapy attenuates deleterious ventricular remodeling and improves cardiac performance after myocardial infarction, CIRCULATION, 103(14), 2001, pp. 1920-1927
Authors:
Nagata, K
Communal, C
Lim, CC
Jain, M
Suter, TM
Eberli, FR
Satoh, N
Colucci, WS
Apstein, CS
Liao, RL
Citation: K. Nagata et al., Altered beta-adrenergic signal transduction in nonfailing hypertrophied myocytes from Dahl salt-sensitive rats, AM J P-HEAR, 279(5), 2000, pp. H2502-H2508
Authors:
Sam, F
Sawyer, DB
Chang, DLF
Eberli, FR
Ngoy, S
Jain, M
Amin, J
Apstein, CS
Colucci, WS
Citation: F. Sam et al., Progressive left ventricular remodeling and apoptosis late after myocardial infarction in mouse heart, AM J P-HEAR, 279(1), 2000, pp. H422-H428
Authors:
Saupe, KW
Lim, CC
Ingwall, JS
Apstein, CS
Eberli, FR
Citation: Kw. Saupe et al., Comparison of hearts with 2 types of pressure-overload left ventricular hypertrophy, HYPERTENSIO, 35(5), 2000, pp. 1167-1172
Authors:
Lim, CC
Apstein, CS
Colucci, WS
Liao, RL
Citation: Cc. Lim et al., Impaired cell shortening and relengthening with increased pacing frequencyare intrinsic to the senescent mouse cardiomyocyte, J MOL CEL C, 32(11), 2000, pp. 2075-2082
Authors:
Eberli, FR
Stromer, H
Ferrell, MA
Varma, N
Morgan, JP
Neubauer, S
Apstein, CS
Citation: Fr. Eberli et al., Lack of direct role for calcium in ischemic diastolic dysfunction in isolated hearts, CIRCULATION, 102(21), 2000, pp. 2643-2649
Authors:
Varma, N
Eberli, FR
Apstein, CS
Citation: N. Varma et al., Increased diastolic chamber stiffness during demand ischemia - Response toquick length change differentiates rigor-activared from calcium-activated tension, CIRCULATION, 101(18), 2000, pp. 2185-2192
Authors:
Cave, AC
Ingwall, JS
Friedrich, J
Liao, RL
Saupe, KW
Apstein, CS
Eberli, FR
Citation: Ac. Cave et al., ATP synthesis during low-flow ischemia - Influence of increased glycolyticsubstrate, CIRCULATION, 101(17), 2000, pp. 2090-2096
Authors:
Jain, M
Liao, R
Ngoy, S
Whittaker, P
Apstein, CS
Eberli, FR
Citation: M. Jain et al., Angiotensin II receptor blockade attenuates the deleterious effects of exercise training on post-MI ventricular remodelling in rats, CARDIO RES, 46(1), 2000, pp. 66-72
Authors:
Apstein, CS
Citation: Cs. Apstein, Increased glycolytic substrate protection improves ischemic cardiac dysfunction and reduces injury, AM HEART J, 139(2), 2000, pp. S107-S114
Authors:
Apstein, CS
Citation: Cs. Apstein, Metabolic approaches in ischaemic heart disease, EUR H J SUP, 1(O), 1999, pp. O1-O10
Authors:
Apstein, CS
Opie, LH
Citation: Cs. Apstein et Lh. Opie, Glucose-insulin-potassium (GIK) for acute myocardial infarction: A negative study with a positive value, CARDIO DRUG, 13(3), 1999, pp. 185-189
Authors:
Saupe, KW
Eberli, FR
Ingwall, JS
Apstein, CS
Citation: Kw. Saupe et al., Hypoperfusion-induced contractile failure does not require changes in cardiac energetics, AM J P-HEAR, 45(5), 1999, pp. H1715-H1723
Authors:
Lim, CC
Liao, RL
Varma, N
Apstein, CS
Citation: Cc. Lim et al., Impaired lusitropy-frequency in the aging mouse: role of Ca2+-handling proteins and effects of isoproterenol, AM J P-HEAR, 277(5), 1999, pp. H2083-H2090
Authors:
McKenney, PA
Apstein, CS
Mendes, LA
Connelly, GP
Aldea, GS
Shemin, RJ
Davidoff, R
Citation: Pa. Mckenney et al., Immediate effect of aortic valve replacement for aortic stenosis on left ventricular diastolic chamber stiffness, AM J CARD, 84(8), 1999, pp. 914-918
Authors:
Kondo, RP
Apstein, CS
Eberli, FR
Tillotson, DL
Suter, TM
Citation: Rp. Kondo et al., Increased calcium loading and inotropy without greater cell death in hypoxic rat cardiomyocytes, AM J P-HEAR, 44(6), 1998, pp. H2272-H2282
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