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SMITH G
HANHAM I
CHRISTMAS T
BANKS L
EPSTEIN RJ
Citation: G. Smith et al., CHANGES IN BONE-DENSITY DURING PROSTATE-CANCER PROGRESSION AND TREATMENT QUANTIFIED USING WHOLE-BODY DXA DENSITOMETRIC SCANNING, Annals of oncology, 9, 1998, pp. 277-277
Authors:
STOREY A
THOMAS M
KALITA A
HARWOOD C
GARDIOL D
MANTOVANI F
BREUER J
LEIGH IM
MATLASHEWSKI G
BANKS L
Citation: A. Storey et al., ROLE OF A P53 POLYMORPHISM IN THE DEVELOPMENT OF HUMAN PAPILLOMAVIRUS-ASSOCIATED CANCER, Nature, 393(6682), 1998, pp. 229-234
Authors:
STOREY A
HARWOOD C
MATLASHEWSKI G
THOMAS M
BREUER J
LEIGH I
GARDIOL D
KALITA A
BANKS L
Citation: A. Storey et al., A GENETIC-POLYMORPHISM DETERMINES SUSCEPTIBILITY TO HUMAN-PAPILLOMAVIRUS ASSOCIATED CANCERS, Journal of investigative dermatology, 110(4), 1998, pp. 485-485
Citation: D. Gardiol et L. Banks, COMPARISON OF HUMAN-PAPILLOMAVIRUS TYPE-18 (HPV-18) E6-MEDIATED DEGRADATION OF P53 IN-VITRO AND IN-VIVO REVEALS SIGNIFICANT DIFFERENCES BASED ON P53 STRUCTURE AND CELL-TYPE BUT LITTLE DIFFERENCE WITH RESPECT TO MUTANTS OF HPV-18 E6, Journal of General Virology, 79, 1998, pp. 1963-1970
Authors:
DOHERTY A
SMITH G
BANKS L
CHRISTMAS TJ
EPSTEIN RJ
Citation: A. Doherty et al., A PATHOGENETIC ROLE FOR PROSTATE-SPECIFIC ANTIGEN (PSA) IN OSTEOBLASTIC BONE METASTASIS - IMPLICATIONS FOR DRUG-TREATMENT OF HORMONE-RESISTANT DISEASE, European journal of cancer, 33, 1997, pp. 113-113
Citation: D. Pim et al., ALTERNATIVELY SPLICED HPV-18 E6-ASTERISK PROTEIN INHIBITS E6 MEDIATEDDEGRADATION OF P53 AND SUPPRESSES TRANSFORMED-CELL GROWTH, Oncogene, 15(3), 1997, pp. 257-264
Citation: A. Piccini et al., REGULATION OF HUMAN-PAPILLOMAVIRUS TYPE-16 DNA-REPLICATION BY E2, GLUCOCORTICOID HORMONE AND EPIDERMAL GROWTH-FACTOR, Journal of General Virology, 78, 1997, pp. 1963-1970
Citation: P. Massimi et al., HUMAN-PAPILLOMAVIRUS TYPE-16 E7 BINDS TO THE CONSERVED CARBOXY-TERMINAL REGION OF THE TATA BOX-BINDING PROTEIN AND THIS CONTRIBUTES TO E7 TRANSFORMING ACTIVITY, Journal of General Virology, 78, 1997, pp. 2607-2613
Citation: V. Bouvard et al., CHARACTERIZATION OF CELLULAR-CHANGES WHICH INFLUENCE PROGRESSION OF HUMAN PAPILLOMAVIRUS TYPE-16 IMMORTALIZED KERATINOCYTES TO ANCHORAGE-INDEPENDENT PHENOTYPE, International journal of oncology, 8(1), 1996, pp. 159-167
Citation: M. Thomas et al., HPV-18 E6 INHIBITS P53 DNA-BINDING ACTIVITY REGARDLESS OF THE OLIGOMERIC STATE OF P53 OR THE EXACT P53 RECOGNITION SEQUENCE, Oncogene, 13(3), 1996, pp. 471-480
Citation: M. Thomas et al., INDUCTION OF APOPTOSIS BY P53 IS INDEPENDENT OF ITS OLIGOMERIC STATE AND CAN BE ABOLISHED BY HPV-18 E6 THROUGH UBIQUITIN MEDIATED DEGRADATION, Oncogene, 13(2), 1996, pp. 265-273
Citation: A. Storey et al., CONDITIONAL IMMORTALIZATION OF PRIMARY-CELLS BY HUMAN PAPILLOMAVIRUS TYPE-18 E6 AND E5-RAS DEFINES AN E6 ACTIVITY IN G(0) G(1) PHASE WHICH CAN BE SUBSTITUTED FOR BY MUTATIONS IN P53 (VOL 11, PG 653, 1995)/, Oncogene, 12(4), 1996, pp. 945-945
Citation: A. Storey et al., CONDITIONAL IMMORTALIZATION OF PRIMARY-CELLS BY HUMAN PAPILLOMAVIRUS TYPE-18 E6 AND EJ-RAS DEFINES AN E6 ACTIVITY IN G(0) G(1) PHASE WHICH CAN BE SUBSTITUTED FOR MUTATIONS IN P53/, Oncogene, 11(4), 1995, pp. 653-661
Citation: M. Thomas et al., HPV-18 E6 MEDIATED INHIBITION OF P53 DNA-BINDING ACTIVITY IS INDEPENDENT OF E6 INDUCED DEGRADATION, Oncogene, 10(2), 1995, pp. 261-268
Citation: Gf. Valle et L. Banks, THE HUMAN PAPILLOMAVIRUS (HPV)-6 AND HPV-16 E5 PROTEINS COOPERATE WITH HPV-16 E7 IN THE TRANSFORMATION OF PRIMARY RODENT CELLS, Journal of General Virology, 76, 1995, pp. 1239-1245
Authors:
STOREY A
PICCINI A
MASSIMI P
BOUVARD V
BANKS L
Citation: A. Storey et al., MUTATIONS IN THE HUMAN PAPILLOMAVIRUS TYPE-16 E2 PROTEIN IDENTIFY A REGION OF THE PROTEIN INVOLVED IN BINDING TO E1 PROTEIN, Journal of General Virology, 76, 1995, pp. 819-826
Citation: A. Piccini et al., MUTATIONS IN THE HUMAN PAPILLOMAVIRUS TYPE-16 E2 PROTEIN IDENTIFY MULTIPLE REGIONS OF THE PROTEIN INVOLVED IN BINDING TO E1, Journal of General Virology, 76, 1995, pp. 2909-2913
Authors:
PIM D
STOREY A
THOMAS M
MASSIMI P
BANKS L
Citation: D. Pim et al., MUTATIONAL ANALYSIS OF HPV-18 E6 IDENTIFIES DOMAINS REQUIRED FOR P53 DEGRADATION IN-VITRO, ABOLITION OF P53 TRANSACTIVATION IN-VIVO AND IMMORTALIZATION OF PRIMARY BMK CELLS, Oncogene, 9(7), 1994, pp. 1869-1876
Authors:
GU ZM
PIM D
LABRECQUE S
BANKS L
MATLASHEWSKI G
Citation: Zm. Gu et al., DNA-DAMAGE INDUCED P53 MEDIATED TRANSCRIPTION IS INHIBITED BY HUMAN PAPILLOMAVIRUS TYPE-18 E6, Oncogene, 9(2), 1994, pp. 629-633
Citation: V. Bouvard et al., CHARACTERIZATION OF THE HUMAN PAPILLOMAVIRUS E2 PROTEIN - EVIDENCE OFTRANSACTIVATION AND TRANSREPRESSION IN CERVICAL KERATINOCYTES, EMBO journal, 13(22), 1994, pp. 5451-5459
Authors:
BOUVARD V
MATLASHEWSKI G
GU ZM
STOREY A
BANKS L
Citation: V. Bouvard et al., THE HUMAN PAPILLOMAVIRUS TYPE-16 E5 GENE COOPERATES WITH THE E7 GENE TO STIMULATE PROLIFERATION OF PRIMARY-CELLS AND INCREASES VIRAL GENE-EXPRESSION, Virology, 203(1), 1994, pp. 73-80