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Results: 1-11 |
Results: 11
REGULATORY ROLES OF COMPLEXINS IN NEUROTRANSMITTER RELEASE FROM MATURE PRESYNAPTIC NERVE-TERMINALS
Authors: ONO S BAUX G SEKIGUCHI M FOSSIER P MOREL NF NIHONMATSU I HIRATA K AWAJI T TAKAHASHI S TAKAHASHI M
Citation: S. Ono et al., REGULATORY ROLES OF COMPLEXINS IN NEUROTRANSMITTER RELEASE FROM MATURE PRESYNAPTIC NERVE-TERMINALS, European journal of neuroscience, 10(6), 1998, pp. 2143-2152
CONTROL OF THE CALCIUM-CONCENTRATION INVOLVED IN ACETYLCHOLINE-RELEASE AND ITS FACILITATION - AN ADDITIONAL ROLE FOR SYNAPTIC VESICLES
Authors: FOSSIER P DIEBLER MF MOTHET JP ISRAEL M TAUC L BAUX G
Citation: P. Fossier et al., CONTROL OF THE CALCIUM-CONCENTRATION INVOLVED IN ACETYLCHOLINE-RELEASE AND ITS FACILITATION - AN ADDITIONAL ROLE FOR SYNAPTIC VESICLES, Neuroscience, 85(1), 1998, pp. 85-91
CYCLIC ADP-RIBOSE AND CALCIUM-INDUCED CALCIUM-RELEASE REGULATE NEUROTRANSMITTER RELEASE AT A CHOLINERGIC SYNAPSE OF APLYSIA
Authors: MOTHET JP FOSSIER P MEUNIER FM STINNAKRE J TAUC L BAUX G
Citation: Jp. Mothet et al., CYCLIC ADP-RIBOSE AND CALCIUM-INDUCED CALCIUM-RELEASE REGULATE NEUROTRANSMITTER RELEASE AT A CHOLINERGIC SYNAPSE OF APLYSIA, Journal of physiology, 507(2), 1998, pp. 405-414
OPPOSITE EFFECTS OF NITRIC-OXIDE ON IDENTIFIED INHIBITORY AND EXCITATORY CHOLINERGIC SYNAPSES OF APLYSIA-CALIFORNICA
Authors: MOTHET JP FOSSIER P SCHIRAR A TAUC L BAUX G
Citation: Jp. Mothet et al., OPPOSITE EFFECTS OF NITRIC-OXIDE ON IDENTIFIED INHIBITORY AND EXCITATORY CHOLINERGIC SYNAPSES OF APLYSIA-CALIFORNICA, Physiological Research, 45(3), 1996, pp. 177-183
OPPOSITE ACTIONS OF NITRIC-OXIDE ON CHOLINERGIC SYNAPSES - WHICH PATHWAYS
Authors: MOTHET JP FOSSIER P TAUC L BAUX G
Citation: Jp. Mothet et al., OPPOSITE ACTIONS OF NITRIC-OXIDE ON CHOLINERGIC SYNAPSES - WHICH PATHWAYS, Proceedings of the National Academy of Sciences of the United Statesof America, 93(16), 1996, pp. 8721-8726
NO DECREASES EVOKED QUANTAL ACH RELEASE AT A SYNAPSE OF APLYSIA BY A MECHANISM INDEPENDENT OF CA2-KINASE-G( INFLUX AND PROTEIN)
Authors: MOTHET JP FOSSIER P TAUC L BAUX G
Citation: Jp. Mothet et al., NO DECREASES EVOKED QUANTAL ACH RELEASE AT A SYNAPSE OF APLYSIA BY A MECHANISM INDEPENDENT OF CA2-KINASE-G( INFLUX AND PROTEIN), Journal of physiology, 493(3), 1996, pp. 769-784
A NITRIC-OXIDE SYNTHASE ACTIVITY IS INVOLVED IN THE MODULATION OF ACETYLCHOLINE-RELEASE IN APLYSIA GANGLION NEURONS - A HISTOLOGICAL, VOLTAMMETRIC AND ELECTROPHYSIOLOGICAL STUDY
Authors: MEULEMANS A MOTHET JP SCHIRAR A FOSSIER P TAUC L BAUX G
Citation: A. Meulemans et al., A NITRIC-OXIDE SYNTHASE ACTIVITY IS INVOLVED IN THE MODULATION OF ACETYLCHOLINE-RELEASE IN APLYSIA GANGLION NEURONS - A HISTOLOGICAL, VOLTAMMETRIC AND ELECTROPHYSIOLOGICAL STUDY, Neuroscience, 69(3), 1995, pp. 985-995
A SYNTAXIN-RELATED PROTEIN CONTROLS ACETYLCHOLINE-RELEASE BY DIFFERENT MECHANISMS IN APLYSIA
Authors: SMIRNOVA T FOSSIER P STINNAKRE J MALLET J BAUX G
Citation: T. Smirnova et al., A SYNTAXIN-RELATED PROTEIN CONTROLS ACETYLCHOLINE-RELEASE BY DIFFERENT MECHANISMS IN APLYSIA, Neuroscience, 68(1), 1995, pp. 125-133
INHIBITION OF ACH RELEASE AT AN APLYSIA SYNAPSE BY NEUROTOXIC PHOSPHOLIPASES A(2) - SPECIFIC RECEPTORS AND MECHANISMS OF ACTION
Authors: FOSSIER P LAMBEAU G LAZDUNSKI M BAUX G
Citation: P. Fossier et al., INHIBITION OF ACH RELEASE AT AN APLYSIA SYNAPSE BY NEUROTOXIC PHOSPHOLIPASES A(2) - SPECIFIC RECEPTORS AND MECHANISMS OF ACTION, Journal of physiology, 489(1), 1995, pp. 29-40
PRESYNAPTIC MECHANISMS REGULATING CA2-RELEASE AT AN IDENTIFIED NEURO-NEURONAL SYNAPSE OF APLYSIA( CONCENTRATION TRIGGERING ACETYLCHOLINE)
Authors: FOSSIER P BAUX G TAUC L
Citation: P. Fossier et al., PRESYNAPTIC MECHANISMS REGULATING CA2-RELEASE AT AN IDENTIFIED NEURO-NEURONAL SYNAPSE OF APLYSIA( CONCENTRATION TRIGGERING ACETYLCHOLINE), Neuroscience, 63(2), 1994, pp. 405-414
N-TYPE AND P-TYPE CA2-RELEASE AT A NEURONEURONAL SYNAPSE - ONLY THE N-TYPE CHANNEL IS THE TARGET OF NEUROMODULATORS( CHANNELS ARE INVOLVED IN ACETYLCHOLINE)
Authors: FOSSIER P BAUX G TAUC L
Citation: P. Fossier et al., N-TYPE AND P-TYPE CA2-RELEASE AT A NEURONEURONAL SYNAPSE - ONLY THE N-TYPE CHANNEL IS THE TARGET OF NEUROMODULATORS( CHANNELS ARE INVOLVED IN ACETYLCHOLINE), Proceedings of the National Academy of Sciences of the United Statesof America, 91(11), 1994, pp. 4771-4775
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