Authors:
CONDORELLI G
VIGLIOTTA G
IAVARONE C
CARUSO M
TOCCHETTI CG
ANDREOZZI F
CAFIERI A
TECCE MF
FORMISANO P
BEGUINOT L
BEGUINOT F
Citation: G. Condorelli et al., PED PEA-15 GENE CONTROLS GLUCOSE-TRANSPORT AND IS OVEREXPRESSED IN TYPE-2 DIABETES-MELLITUS/, EMBO journal (Print), 17(14), 1998, pp. 3858-3866
Authors:
KORNILOVA E
SORKINA T
BEGUINOT L
SORKIN A
Citation: E. Kornilova et al., LYSOSOMAL TARGETING OF EPIDERMAL GROWTH-FACTOR RECEPTORS VIA A KINASE-DEPENDENT PATHWAY IS MEDIATED BY THE RECEPTOR CARBOXYL-TERMINAL RESIDUES-1022-1123, The Journal of biological chemistry, 271(48), 1996, pp. 30340-30346
Authors:
NICOLINI G
MILOSO M
MORONI MC
BEGUINOT L
SCOTTO L
Citation: G. Nicolini et al., POSTTRANSCRIPTIONAL CONTROL REGULATES TRANSFORMING GROWTH-FACTOR-ALPHA IN THE HUMAN CARCINOMA KB CELL-LINE, The Journal of biological chemistry, 271(47), 1996, pp. 30290-30296
Authors:
SORKIN A
MAZZOTTI M
SORKINA T
SCOTTO L
BEGUINOT L
Citation: A. Sorkin et al., EPIDERMAL GROWTH-FACTOR RECEPTOR INTERACTION WITH CLATHRIN ADAPTERS IS MEDIATED BY THE TYR(974)-CONTAINING INTERNALIZATION MOTIF, The Journal of biological chemistry, 271(23), 1996, pp. 13377-13384
Authors:
BEGUINOT L
YAMAZAKI H
PASTAN I
JOHNSON AC
Citation: L. Beguinot et al., BIOCHEMICAL-CHARACTERIZATION OF HUMAN GCF TRANSCRIPTION FACTOR IN TUMOR-CELLS, Cell growth & differentiation, 6(6), 1995, pp. 699-706
Citation: L. Beguinot et al., SHC IS CONSTITUTIVELY ACTIVATED BY A TRANSFORMING EGF RECEPTOR MUTANTWITH A POINT MUTATION IN THE TRANSMEMBRANE DOMAIN, Journal of cellular biochemistry, 1995, pp. 11-11
Citation: M. Miloso et al., SHC AND GRB-2 ARE CONSTITUTIVELY ACTIVATED BY AN EPIDERMAL GROWTH-FACTOR RECEPTOR WITH A POINT MUTATION IN THE TRANSMEMBRANE DOMAIN, The Journal of biological chemistry, 270(33), 1995, pp. 19557-19562
Citation: Cv. Alvarez et al., STRUCTURAL REQUIREMENTS OF THE EPIDERMAL GROWTH-FACTOR RECEPTOR FOR TYROSINE PHOSPHORYLATION OF EPS8 AND EPS15, SUBSTRATES LACKING SRC SH2 HOMOLOGY DOMAINS, The Journal of biological chemistry, 270(27), 1995, pp. 16271-16276
Authors:
MIELE C
FORMISANO P
SOHN KJ
CARUSO M
PIANESE M
PALUMBO G
BEGUINOT L
BEGUINOT F
Citation: C. Miele et al., DECREASED PHOSPHORYLATION OF MUTANT INSULIN-RECEPTOR BY PROTEIN-KINASE-C AND PROTEIN-KINASE-A, The Journal of biological chemistry, 270(26), 1995, pp. 15844-15852
Authors:
SOLER C
ALVAREZ CV
BEGUINOT L
CARPENTER G
Citation: C. Soler et al., POTENT SHC TYROSINE PHOSPHORYLATION BY EPIDERMAL GROWTH-FACTOR AT LOWRECEPTOR DENSITY OR IN THE ABSENCE OF RECEPTOR AUTOPHOSPHORYLATION SITES, Oncogene, 9(8), 1994, pp. 2207-2215
Authors:
JOHNSON AC
YAMAZAKI H
PIEBENGA E
BEGUINOT L
Citation: Ac. Johnson et al., EXPRESSION AND CHARACTERIZATION OF THE HUMAN TRANSCRIPTIONAL REPRESSOR GCF IN CANCER CELL-LINES, The FASEB journal, 8(7), 1994, pp. 10001397-10001397
Authors:
FORMISANO P
DENOVELLIS G
MIELE C
TRIPODI F
CARUSO M
PALUMBO G
BEGUINOT L
BEGUINOT F
Citation: P. Formisano et al., INTERNALIZATION OF THE CONSTITUTIVELY ACTIVE ARGININE-1152-]GLUTAMINEINSULIN-RECEPTOR OCCURS INDEPENDENTLY OF INSULIN AT AN ACCELERATED RATE, The Journal of biological chemistry, 269(23), 1994, pp. 16242-16246
Citation: C. Soler et al., INDIVIDUAL EPIDERMAL GROWTH-FACTOR RECEPTOR AUTOPHOSPHORYLATION SITESDO NOT STRINGENTLY DEFINE ASSOCIATION MOTIFS FOR SEVERAL SH2-CONTAINING PROTEINS, The Journal of biological chemistry, 269(16), 1994, pp. 12320-12324
Authors:
PETRUZZIELLO A
FORMISANO P
MIELE C
DIFINIZIO B
RICCARDI G
FERRARA A
BEGUINOT L
BEGUINOT F
Citation: A. Petruzziello et al., DEFECTIVE INSULIN ACTION IN FIBROBLASTS FROM NONINSULIN-DEPENDENT DIABETES-MELLITUS PATIENTS WITH GLN(1152) INSULIN-RECEPTOR MUTATION, The Journal of clinical endocrinology and metabolism, 77(2), 1993, pp. 409-412
Citation: C. Soler et al., TYROSINE PHOSPHORYLATION OF RAS GTPASE-ACTIVATING PROTEIN DOES NOT REQUIRE ASSOCIATION WITH THE EPIDERMAL GROWTH-FACTOR RECEPTOR, The Journal of biological chemistry, 268(29), 1993, pp. 22010-22019
Authors:
FORMISANO P
MIELE C
PETRUZZIELLO A
DIFINIZIO B
BEGUINOT L
BEGUINOT F
Citation: P. Formisano et al., A BASIC-AMINO-ACID MOTIF REGULATES INSULIN-RECEPTOR (I(R)) KINASE-ACTIVITY AND METABOLIC SIGNALING, Diabetes, 42, 1993, pp. 129-129