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Results: 1-3 |
Results: 3
PREMALIGNANT HUMAN KERATINOCYTES EXHIBITING EXTENDED REPLICATIVE LIFE-SPANS AND EGF-INDEPENDENCE CULTURED FROM DYSPLASTIC ORAL EPITHELIAL LESIONS
Authors: RHEINWALD J PALANISAMY A BENWOOD J FELDMAN R SAUTER E
Citation: J. Rheinwald et al., PREMALIGNANT HUMAN KERATINOCYTES EXHIBITING EXTENDED REPLICATIVE LIFE-SPANS AND EGF-INDEPENDENCE CULTURED FROM DYSPLASTIC ORAL EPITHELIAL LESIONS, Journal of investigative dermatology, 110(4), 1998, pp. 522-522
ORAL KERATINOCYTES FORM A HYPERKERATOTIC EPITHELIUM AND EXPRESS KERATINS K1 K10 IN ORGANOTYPIC CULTURE WITH EITHER DERMAL OR ORAL FIBROBLASTS, AN ABNORMAL PHENOTYPE THAT IS RESISTANT TO RETINOIC ACID MODULATION/
Authors: SCHON M BENWOOD J RHEINWALD JG
Citation: M. Schon et al., ORAL KERATINOCYTES FORM A HYPERKERATOTIC EPITHELIUM AND EXPRESS KERATINS K1 K10 IN ORGANOTYPIC CULTURE WITH EITHER DERMAL OR ORAL FIBROBLASTS, AN ABNORMAL PHENOTYPE THAT IS RESISTANT TO RETINOIC ACID MODULATION/, Journal of investigative dermatology, 108(4), 1997, pp. 517-517
KERATIN K19 IS INDUCED IN EPIDERMAL-KERATINOCYTES BY ACTIVATORS OF PROTEIN-KINASE-C, REVEALING A K19 REGULATORY MECHANISM DISTINCT FROM THAT INVOLVING RETINOIDS AND THEIR RECEPTORS
Authors: SCHON M BENWOOD J RHEINWALD JG
Citation: M. Schon et al., KERATIN K19 IS INDUCED IN EPIDERMAL-KERATINOCYTES BY ACTIVATORS OF PROTEIN-KINASE-C, REVEALING A K19 REGULATORY MECHANISM DISTINCT FROM THAT INVOLVING RETINOIDS AND THEIR RECEPTORS, Journal of investigative dermatology, 106(4), 1996, pp. 413-413
Risultati: 1-3 |