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Results: 1-8 |
Results: 8
IGF-I TREATMENT OF NORMAL RATS SUPPRESSES LEPTIN MESSENGER-RNA IN FATTISSUE
Authors: BONISCHNETZLER M HAURI C ZAPF J
Citation: M. Bonischnetzler et al., IGF-I TREATMENT OF NORMAL RATS SUPPRESSES LEPTIN MESSENGER-RNA IN FATTISSUE, Diabetologia, 40, 1997, pp. 1069-1069
IN-FRAME EXON-2 DELETION IN INSULIN-RECEPTOR RNA IN A FAMILY WITH EXTREME INSULIN-RESISTANCE IN ASSOCIATION WITH DEFECTIVE INSULIN BINDING - A CASE-REPORT
Authors: MORITZ W BONISCHNETZLER M STEVENS W FROESCH ER LEVY JR
Citation: W. Moritz et al., IN-FRAME EXON-2 DELETION IN INSULIN-RECEPTOR RNA IN A FAMILY WITH EXTREME INSULIN-RESISTANCE IN ASSOCIATION WITH DEFECTIVE INSULIN BINDING - A CASE-REPORT, European journal of endocrinology, 135(3), 1996, pp. 357-363
A NOVEL POINT MUTATION IN THE TRANSLATION INITIATION CODON OF THE PRE-PRO-VASOPRESSIN-NEUROPHYSIN-II GENE - COSEGREGATION WITH MORPHOLOGICAL ABNORMALITIES AND CLINICAL SYMPTOMS IN AUTOSOMAL-DOMINANT NEUROHYPOPHYSEAL DIABETES-INSIPIDUS
Authors: RUTISHAUSER J BONISCHNETZLER M BONI J WICHMANN W HUISMAN T VALLOTTON MB FROESCH ER
Citation: J. Rutishauser et al., A NOVEL POINT MUTATION IN THE TRANSLATION INITIATION CODON OF THE PRE-PRO-VASOPRESSIN-NEUROPHYSIN-II GENE - COSEGREGATION WITH MORPHOLOGICAL ABNORMALITIES AND CLINICAL SYMPTOMS IN AUTOSOMAL-DOMINANT NEUROHYPOPHYSEAL DIABETES-INSIPIDUS, The Journal of clinical endocrinology and metabolism, 81(1), 1996, pp. 192-198
REDUCED OB MESSENGER-RNA IN HYPOPHYSECTOMIZED RATS IS NOT RESTORED BYGROWTH-HORMONE (GH), BUT FURTHER SUPPRESSED BY EXOGENOUSLY ADMINISTERED INSULIN-LIKE GROWTH-FACTOR (IGF)-I
Authors: BONISCHNETZLER M GOSTELIPETER MA MORITZ W FROESCH ER ZAPF J
Citation: M. Bonischnetzler et al., REDUCED OB MESSENGER-RNA IN HYPOPHYSECTOMIZED RATS IS NOT RESTORED BYGROWTH-HORMONE (GH), BUT FURTHER SUPPRESSED BY EXOGENOUSLY ADMINISTERED INSULIN-LIKE GROWTH-FACTOR (IGF)-I, Biochemical and biophysical research communications, 225(1), 1996, pp. 296-301
A NOVEL POINT MUTATION GLY1119-]ARG IN THE INSULIN-RECEPTOR GENE OF 2SISTERS WITH TYPE-A INSULIN-RESISTANCE
Authors: BONISCHNETZLER M MORITZ W FROESCH ER
Citation: M. Bonischnetzler et al., A NOVEL POINT MUTATION GLY1119-]ARG IN THE INSULIN-RECEPTOR GENE OF 2SISTERS WITH TYPE-A INSULIN-RESISTANCE, Diabetologia, 38, 1995, pp. 132-132
FUNCTIONAL-PROPERTIES OF A HETEROZYGOUS MUTATION (ARG(1174)-]GLN) IN THE TYROSINE KINASE DOMAIN OF THE INSULIN-RECEPTOR FROM A TYPE-A INSULIN-RESISTANT PATIENT
Authors: MORITZ W FROESCH ER BONISCHNETZLER M
Citation: W. Moritz et al., FUNCTIONAL-PROPERTIES OF A HETEROZYGOUS MUTATION (ARG(1174)-]GLN) IN THE TYROSINE KINASE DOMAIN OF THE INSULIN-RECEPTOR FROM A TYPE-A INSULIN-RESISTANT PATIENT, FEBS letters, 351(2), 1994, pp. 276-280
GROWTH-HORMONE AND PARATHYROID-HORMONE STIMULATE IGFBP-3 IN RAT OSTEOBLASTS
Authors: SCHMID C SCHLAPFER I PETER M BONISCHNETZLER M SCHWANDER J ZAPF J FROESCH ER
Citation: C. Schmid et al., GROWTH-HORMONE AND PARATHYROID-HORMONE STIMULATE IGFBP-3 IN RAT OSTEOBLASTS, The American journal of physiology, 267(2), 1994, pp. 50000226-50000233
REGULATION OF INSULIN-LIKE GROWTH FACTOR-I (IGF-I) AND IGF-BINDING PROTEINS BY GROWTH-HORMONE IN RAT WHITE ADIPOSE-TISSUE
Authors: PETER MA WINTERHALTER KH BONISCHNETZLER M FROESCH ER ZAPF J
Citation: Ma. Peter et al., REGULATION OF INSULIN-LIKE GROWTH FACTOR-I (IGF-I) AND IGF-BINDING PROTEINS BY GROWTH-HORMONE IN RAT WHITE ADIPOSE-TISSUE, Endocrinology, 133(6), 1993, pp. 2624-2631
Risultati: 1-8 |