Authors:
BROWNELL HL
LYDON NB
SCHAEFER E
ROBERTS TM
RAPTIS L
Citation: Hl. Brownell et al., INHIBITION OF EPIDERMAL GROWTH FACTOR-MEDIATED ERK1 2 ACTIVATION BY IN-SITU ELECTROPORATION OF NONPERMEANT [(ALKYLAMINO)METHYL]ACRYLOPHENONE DERIVATIVES/, DNA and cell biology, 17(3), 1998, pp. 265-274
Authors:
TOMAI E
BROWNELL HL
TUFESCU TV
RAPTIS S
REID K
CAMPLING BG
RAPTIS L
Citation: E. Tomai et al., A FUNCTIONAL ASSAY FOR INTERCELLULAR, JUNCTIONAL COMMUNICATION IN CULTURED HUMAN LUNG-CARCINOMA CELLS, Laboratory investigation, 78(5), 1998, pp. 639-640
Citation: Hl. Brownell et L. Raptis, A GRB2-SH2 BINDING PEPTIDE INHIBITS ERK ACTIVATION BY EGF OR PDGF IN NIH 3T3 CELLS, Molecular biology of the cell, 8, 1997, pp. 1424-1424
Citation: E. Tomais et al., A FUNCTIONAL ASSAY FOR INTERCELLULAR, JUNCTIONAL COMMUNICATION IN HUMAN LUNG-CARCINOMA CELLS, Molecular biology of the cell, 8, 1997, pp. 2009-2009
Authors:
RAPTIS L
BROWNELL HL
CORBLEY MJ
WOOD KW
WANG D
HALIOTIS T
Citation: L. Raptis et al., CELLULAR RAS GENE ACTIVITY IS REQUIRED FOR FULL NEOPLASTIC TRANSFORMATION BY THE LARGE TUMOR-ANTIGEN OF SV40, Cell growth & differentiation, 8(8), 1997, pp. 891-901
Authors:
BROWNELL HL
FIRTH KL
KAWAUCHI K
DELOVITCH T
RAPTIS L
Citation: Hl. Brownell et al., A NOVEL TECHNIQUE FOR THE STUDY OF RAS ACTIVITY - ELECTROPORATION OF [ALPHA-P-32]GTP, DNA and cell biology, 16(1), 1997, pp. 103-110
Citation: Hl. Brownell et al., ELIMINATION OF INTERCELLULAR JUNCTIONAL COMMUNICATION REQUIRES LOWER RAS(LEU61) LEVELS THAN STIMULATION OF ANCHORAGE-INDEPENDENT PROLIFERATION, Cancer detection and prevention, 21(4), 1997, pp. 289-294
Authors:
RAPTIS L
BROWNELL HL
LU Y
PRESTON T
NARSIMHAN RP
ANDERSON S
SCHAEFER E
HALIOTIS T
Citation: L. Raptis et al., V-RAS AND V-RAF BLOCK DIFFERENTIATION OF TRANSFORMABLE C3H10T1 2-DERIVED PREADIPOCYTES AT LOWER LEVELS THAN REQUIRED FOR NEOPLASTIC TRANSFORMATION/, Experimental cell research, 235(1), 1997, pp. 188-197
Authors:
BROWNELL HL
NARSIMHAN RP
CORBLEY MJ
MANN VM
WHITFIELD JF
RAPTIS L
Citation: Hl. Brownell et al., RAS IS INVOLVED IN GAP JUNCTION CLOSURE IN PROLIFERATING FIBROBLASTS OR PREADIPOCYTES BUT NOT IN DIFFERENTIATED ADIPOCYTES, DNA and cell biology, 15(6), 1996, pp. 443-451
Citation: Hl. Brownell et al., CELLULAR RAS PARTLY MEDIATES GAP JUNCTION CLOSURE BY THE POLYOMA-VIRUS MIDDLE TUMOR-ANTIGEN, Cancer letters, 103(1), 1996, pp. 99-106
Citation: Lh. Raptis et al., A NOVEL TECHNIQUE FOR THE STUDY OF INTERCELLULAR, JUNCTIONAL COMMUNICATION - ELECTROPORATION OF ADHERENT CELLS ON A PARTLY CONDUCTIVE SLIDE, DNA and cell biology, 13(9), 1994, pp. 963-975