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Results: 1-17 |
Results: 17
A pathogenic presenilin-1 deletion causes abberrant A beta 42 production in the absence of congophilic amyloid plaques
Authors: Steiner, H Revesz, T Neumann, M Romig, H Grim, MG Pesold, B Kretzschmar, HA Hardy, J Holton, JL Baumeister, R Houlden, H Haass, C
Citation: H. Steiner et al., A pathogenic presenilin-1 deletion causes abberrant A beta 42 production in the absence of congophilic amyloid plaques, J BIOL CHEM, 276(10), 2001, pp. 7233-7239
Presenilin-1 differentially facilitates endoproteolysis of the beta-amyloid precursor protein and Notch
Authors: Capell, A Steiner, H Romig, H Keck, S Baader, M Grim, MG Baumeister, R Haass, C
Citation: A. Capell et al., Presenilin-1 differentially facilitates endoproteolysis of the beta-amyloid precursor protein and Notch, NAT CELL BI, 2(4), 2000, pp. 205-211
Glycine 384 is required for presenilin-1 function and is conserved in bacterial polytopic aspartyl proteases
Authors: Steiner, H Kostka, M Romig, H Basset, G Pesold, B Hardy, J Capell, A Meyn, L Grim, ML Baumeister, R Fechteler, K Haass, C
Citation: H. Steiner et al., Glycine 384 is required for presenilin-1 function and is conserved in bacterial polytopic aspartyl proteases, NAT CELL BI, 2(11), 2000, pp. 848-851
Identification of amino acid residues in the Caenorhabditis elegans POU protein UNC-86 that mediate UNC-86-MEC-3-DNA ternary complex formation
Authors: Rockelein, I Rohrig, S Donhauser, R Eimer, S Baumeister, R
Citation: I. Rockelein et al., Identification of amino acid residues in the Caenorhabditis elegans POU protein UNC-86 that mediate UNC-86-MEC-3-DNA ternary complex formation, MOL CELL B, 20(13), 2000, pp. 4806-4813
Protein interaction surface of the POU transcription factor UNC-86 selectively used in touch neurons
Authors: Rohrig, S Rockelein, I Donhauser, P Baumeister, R
Citation: S. Rohrig et al., Protein interaction surface of the POU transcription factor UNC-86 selectively used in touch neurons, EMBO J, 19(14), 2000, pp. 3694-3703
Presenilin is required for proper morphology and function of neurons in C-elegans
Authors: Wittenburg, N Eimer, S Lakowski, B Rohrig, S Rudolph, C Baumeister, R
Citation: N. Wittenburg et al., Presenilin is required for proper morphology and function of neurons in C-elegans, NATURE, 406(6793), 2000, pp. 306-309
Separation of presenilin function in amyloid beta-peptide generation and endoproteolysis of Notch
Authors: Kulic, L Walter, J Multhaup, G Teplow, DB Baumeister, R Romig, H Capell, A Steiner, H Haass, C
Citation: L. Kulic et al., Separation of presenilin function in amyloid beta-peptide generation and endoproteolysis of Notch, P NAS US, 97(11), 2000, pp. 5913-5918
A loss of function mutant of the presenilin homologue SEL-12 undergoes aberrant endoproteolysis in Caenorhabditis elegans and increases A beta 42 generation in human cells
Authors: Okochi, M Eimer, S Bottcher, A Baumeister, R Romig, H Walter, J Capell, A Steiner, H Haass, C
Citation: M. Okochi et al., A loss of function mutant of the presenilin homologue SEL-12 undergoes aberrant endoproteolysis in Caenorhabditis elegans and increases A beta 42 generation in human cells, J BIOL CHEM, 275(52), 2000, pp. 40925-40932
The Caenorhabditis elegans Ldb/NLI/Clim orthologue ldb-1 is required for neuronal function
Authors: Cassata, G Rohrig, S Kuhn, F Hauri, HP Baumeister, R Burglin, TR
Citation: G. Cassata et al., The Caenorhabditis elegans Ldb/NLI/Clim orthologue ldb-1 is required for neuronal function, DEVELOP BIO, 226(1), 2000, pp. 45-56
The physiological role of presenilins in cellular differentiation: lessonsfrom model organisms
Authors: Baumeister, R
Citation: R. Baumeister, The physiological role of presenilins in cellular differentiation: lessonsfrom model organisms, EUR ARCH PS, 249(6), 1999, pp. 280-287
The biological and pathological function of presenilin proteins - simple cell systems and a worm in Alzheimer's disease research
Authors: Haass, C Baumeister, R
Citation: C. Haass et R. Baumeister, The biological and pathological function of presenilin proteins - simple cell systems and a worm in Alzheimer's disease research, EUR ARCH PS, 249, 1999, pp. 23-27
Thermal avoidance in Caenorhabditis elegans: An approach to the study of nociception
Authors: Wittenburg, N Baumeister, R
Citation: N. Wittenburg et R. Baumeister, Thermal avoidance in Caenorhabditis elegans: An approach to the study of nociception, P NAS US, 96(18), 1999, pp. 10477-10482
The influence of endoproteolytic processing of familial Alzheimer's disease presenilin 2 on A beta 42 amyloid peptide formation
Authors: Jacobsen, H Reinhardt, D Brockhaus, M Bur, D Kocyba, C Kurt, H Grim, MG Baumeister, R Loetscher, H
Citation: H. Jacobsen et al., The influence of endoproteolytic processing of familial Alzheimer's disease presenilin 2 on A beta 42 amyloid peptide formation, J BIOL CHEM, 274(49), 1999, pp. 35233-35239
A loss of function mutation of presenilin-2 interferes with amyloid beta-peptide production and notch signaling
Authors: Steiner, H Duff, K Capell, A Romig, H Grim, MG Lincoln, S Hardy, J Yu, X Picciano, M Fechteler, K Citron, M Kopan, R Pesold, B Keck, S Baader, M Tomita, T Iwatsubo, T Baumeister, R Haass, C
Citation: H. Steiner et al., A loss of function mutation of presenilin-2 interferes with amyloid beta-peptide production and notch signaling, J BIOL CHEM, 274(40), 1999, pp. 28669-28673
The biological and pathological function of the presenilin-1 Delta exon 9 mutation is independent of its defect to undergo proteolytic processing
Authors: Steiner, H Romig, H Grim, MG Philipp, U Pesold, B Citron, M Baumeister, R Haass, C
Citation: H. Steiner et al., The biological and pathological function of the presenilin-1 Delta exon 9 mutation is independent of its defect to undergo proteolytic processing, J BIOL CHEM, 274(12), 1999, pp. 7615-7618
Role of presenilin processing and caspases for amyloid beta-peptide generation and NOTCH signaling
Authors: Rohrig, S Brockhaus, M Steiner, H Capell, A Grunberg, J Walter, J Leimer, U Loetscher, H Wittenburg, N Jacobsen, H Baumeister, R Haass, C
Citation: S. Rohrig et al., Role of presenilin processing and caspases for amyloid beta-peptide generation and NOTCH signaling, ALZHEIMER'S DISEASE AND RELATED DISORDERS, 1999, pp. 353-362
What do we learn from a few familial Alzheimer's disease cases?
Authors: Haass, C Baumeister, R
Citation: C. Haass et R. Baumeister, What do we learn from a few familial Alzheimer's disease cases?, J NEURAL TR, 1998, pp. 137-145
Risultati: 1-17 |