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Results: 1-25/27
Authors:
Heindl, B
Becker, BF
Citation: B. Heindl et Bf. Becker, Aspirin, but not the more selective cyclooxygenase (COX)-2 inhibitors meloxicam and SC 58125, aggravates postischaemic cardiac dysfunction, independent of COX function, N-S ARCH PH, 363(2), 2001, pp. 233-240
Authors:
Kastenbauer, S
Koedel, U
Becker, BF
Pfister, HW
Citation: S. Kastenbauer et al., Experimental meningitis in the rat: protection by uric acid at human physiological blood concentrations, EUR J PHARM, 425(2), 2001, pp. 149-152
Authors:
Massoudy, P
Zahler, S
Becker, BF
Braun, SL
Barankay, A
Meisner, H
Citation: P. Massoudy et al., Evidence for inflammatory responses of the lungs during coronary artery bypass grafting with cardiopulmonary bypass, CHEST, 119(1), 2001, pp. 31-36
Authors:
Kupatt, C
Wichels, R
Becker, BF
Boekstegers, P
Citation: C. Kupatt et al., Inhibition of post-ischaemic inflammation as a therapeutic approach to myocardial ischaemia reperfusion injury, EXPERT OP T, 10(9), 2000, pp. 1395-1403
Authors:
Kupatt, C
Habazettl, H
Hanusch, P
Wichels, R
Hahnel, D
Becker, BF
Boekstegers, P
Citation: C. Kupatt et al., c7E3Fab reduces postischemic leukocyte-thrombocyte interaction mediated byfibrinogen - Implications for myocardial reperfusion injury, ART THROM V, 20(10), 2000, pp. 2226-2232
Authors:
Massoudy, P
Zahler, S
Tassani, P
Becker, BF
Richter, JA
Pfauder, M
Lange, R
Meisner, H
Citation: P. Massoudy et al., Reduction of pro-inflammatory cytokine levels and cellular adhesion in CABG procedures with separated pulmonary and systemic extracorporeal circulation without an oxygenator, EUR J CAR-T, 17(6), 2000, pp. 729-735
Authors:
Zahler, S
Kupatt, C
Becker, BF
Citation: S. Zahler et al., Endothelial preconditioning by transient oxidative stress reduces inflammatory responses of cultured endothelial cells to TNF-alpha, FASEB J, 14(3), 2000, pp. 555-564
Authors:
Becker, BF
Kupatt, C
Massoudy, P
Zahler, S
Citation: Bf. Becker et al., Reactive oxygen species and nitric oxide in myocardial ischemia and reperfusion, Z KARDIOL, 89, 2000, pp. 88-91
Authors:
Kupatt, C
Habazettl, H
Becker, BF
Boekstegers, P
Citation: C. Kupatt et al., Endothelial activation - a strategic event during postischemic myocardial inflammation, Z KARDIOL, 89, 2000, pp. 96-100
Authors:
Becker, BF
Heindl, B
Kupatt, C
Zahler, S
Citation: Bf. Becker et al., Endothelial function and hemostasis, Z KARDIOL, 89(3), 2000, pp. 160-167
Authors:
Massoudy, P
Zahler, S
Freyholdt, T
Henze, R
Barankay, A
Becker, BF
Braun, SL
Meisner, H
Citation: P. Massoudy et al., Sodium nitroprusside in patients with compromised left ventricular function undergoing coronary bypass: Reduction of cardiac proinflammatory substances, J THOR SURG, 119(3), 2000, pp. 566-574
Authors:
Szekely, A
Heindl, B
Zahler, S
Conzen, PF
Becker, BF
Citation: A. Szekely et al., Nonuniform behavior of intravenous anesthetics on postischemic adhesion ofneutrophils in the guinea pig heart, ANESTH ANAL, 90(6), 2000, pp. 1293-1300
Authors:
Heindl, B
Becker, BF
Citation: B. Heindl et Bf. Becker, Sevoflurane and isoflurane do not enhance the pre- and postischemic eicosanoid production in guinea pig hearts, ANESTH ANAL, 90(1), 2000, pp. 17-24
Authors:
Massoudy, P
Mempel, T
Raschke, P
Becker, BF
Citation: P. Massoudy et al., Reduction of oxygen delivery during post-ischemic reperfusion protects theisolated guinea pig heart, BAS R CARD, 94(4), 1999, pp. 231-237
Authors:
Heindl, B
Conzen, PF
Becker, BF
Citation: B. Heindl et al., The volatile anesthetic sevoflurane mitigates cardiodepressive effects of platelets in reperfused hearts, BAS R CARD, 94(2), 1999, pp. 102-111
Authors:
Zahler, S
Kupatt, C
Becker, BF
Citation: S. Zahler et al., ACE-inhibition attenuates cardiac cell damage and preserves release of NO in the postischemic heart, IMMUNOPHARM, 44(1-2), 1999, pp. 27-33
Authors:
Habazettl, H
Kupatt, C
Zahler, S
Becker, BF
Messmer, K
Citation: H. Habazettl et al., Selectins and beta(2)-integrins mediate post-ischaemic venular adhesion ofpolymorphonuclear leukocytes, but not capillary plugging, in isolated hearts, PFLUG ARCH, 438(4), 1999, pp. 479-485
Authors:
Becker, BF
Mobert, J
Citation: Bf. Becker et J. Mobert, Low-dose calcium antagonists reduce energy demand and cellular damage of isolated hearts during both ischemia and reperfusion, N-S ARCH PH, 360(3), 1999, pp. 287-294
Authors:
Kupatt, C
Habazettl, H
Goedecke, A
Wolf, DA
Zahler, S
Boekstegers, P
Kelly, RA
Becker, BF
Citation: C. Kupatt et al., Tumor necrosis factor-alpha contributes to ischemia- and reperfusion-induced endothelial activation in isolated hearts, CIRCUL RES, 84(4), 1999, pp. 392-400
Authors:
Zahler, S
Massoudy, P
Hartl, H
Hahnel, C
Meisner, H
Becker, BF
Citation: S. Zahler et al., Acute cardiac inflammatory responses to postischemic reperfusion during cardiopulmonary bypass, CARDIO RES, 41(3), 1999, pp. 722-730
Sodium nitroprusside during coronary artery bypass grafting: Evidence for an antiinflammatory action
Authors:
Massoudy, P
Zahler, S
Barankay, A
Becker, BF
Richter, JA
Meisner, H
Citation: P. Massoudy et al., Sodium nitroprusside during coronary artery bypass grafting: Evidence for an antiinflammatory action, ANN THORAC, 67(4), 1999, pp. 1059-1064
Authors:
Heindl, B
Reichle, FM
Zahler, S
Conzen, PF
Becker, BF
Citation: B. Heindl et al., Sevoflurane and isoflurane protect the reperfused guinea pig heart by reducing postischemic adhesion of polymorphonuclear neutrophils, ANESTHESIOL, 91(2), 1999, pp. 521-530
Authors:
Mobert, J
Zahler, S
Becker, BF
Conzen, PF
Citation: J. Mobert et al., Inhibition of neutrophil activation by volatile anesthetics decreases adhesion to cultured human endothelial cells, ANESTHESIOL, 90(5), 1999, pp. 1372-1381
Authors:
Szekely, A
Heindl, B
Zahler, S
Conzen, PF
Becker, BF
Citation: A. Szekely et al., S(+)-ketamine, but not R(-)-ketamine, reduces postischemic adherence of neutrophils in the coronary system of isolated guinea pig hearts, ANESTH ANAL, 88(5), 1999, pp. 1017-1024
Authors:
Zahler, S
Heindl, B
Becker, BF
Citation: S. Zahler et al., Ketamine does not inhibit inflammatory responses of cultured human endothelial cells but reduces chemotactic activation of neutrophils, ACT ANAE SC, 43(10), 1999, pp. 1011-1016