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Citation: S. Parlato et al., Primary HIV-1 infection of human CD4+T cells passaged into SCID mice leadsto selection of chronically infected cells through a massive Fas-mediated autocrine suicide of uninfected cells, CELL DEAT D, 7(1), 2000, pp. 37-47
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Citation: G. Schiavoni et al., Cyclophosphamide induces type I interferon and augments the number of CD44(hi) T lymphocytes in mice: implications for strategies of chemoimmunotherapy of cancer, BLOOD, 95(6), 2000, pp. 2024-2030
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Citation: A. Albini et al., Inhibition of angiogenesis and vascular tumor growth by interferon-producing cells - A gene therapy approach, AM J PATH, 156(4), 2000, pp. 1381-1393
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Citation: L. Conti et al., Role of the HIV-1 Vpr protein in the cellular response to viral infection:Possible implications for the pathogenesis of AIDS, FIFTH EUROPEAN CONFERENCE ON EXPERIMENTAL AIDS RESEARCH (ECEAR 2000), 2000, pp. 49-53
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Citation: G. D'Agostino et al., Type I consensus IFN (IFN-con1) gene transfer into KSHV/HHV-8-infected BCBL-1 cells causes inhibition of viral lytic cycle activation via induction of apoptosis and abrogates tumorigenicity in SCID mice, J INTERF CY, 19(11), 1999, pp. 1305-1316
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Citation: C. Rozera et al., Murine interferon-alpha(1) gene-transduced ESb tumor cells are rejected byhost-mediated mechanisms despite resistance of the parental tumor to interferon-alpha/beta therapy, CANC GENE T, 6(3), 1999, pp. 246-253
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Citation: C. Lapenta et al., Type I interferon is a powerful inhibitor of in vivo HIV-1 infection and preserves human CD4(+) T cells from virus-induced depletion in SCID mice transplanted with human cells, VIROLOGY, 263(1), 1999, pp. 78-88
Authors:
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Citation: E. Guillemard et al., Inhibitory activity of constitutive nitric oxide on the expression of alpha/beta interferon genes in murine peritoneal macrophages, J VIROLOGY, 73(9), 1999, pp. 7328-7333