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Citation: E. Kirazov et al., Ontogenetic changes in protein level of amyloid precursor protein (APP) ingrowth cones and synaptosomes from rat brain and prenatal expression pattern of APP mRNA isoforms in developing rat embryo, INT J DEV N, 19(3), 2001, pp. 287-296
Citation: S. Rossner et al., Developmentally induced microencephalopathy in guinea pigs - embryonic glial cell activation marks selective neuronal death, INT J DEV N, 19(3), 2001, pp. 313-318
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Citation: S. Rossner et al., Neuronal and glial beta-secretase (BACE) protein expression in transgenic Tg2576 mice with amyloid plaque pathology, J NEUROSC R, 64(5), 2001, pp. 437-446
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Citation: W. Hartig et al., Hyperphosphorylated protein tau is restricted to neurons devoid of perineuronal nets in the cortex of aged bison, NEUROBIOL A, 22(1), 2001, pp. 25-33
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Citation: S. Rossner et al., Constitutive overactivation of protein kinase C in guinea pig brain increases alpha-secretory APP processing without decreasing beta-amyloid generation, EUR J NEURO, 12(9), 2000, pp. 3191-3200
Citation: S. Rossner et al., Intracerebroventricular infusion of CHO5, a rat monoclonal antibody directed against mouse low-affinity nerve growth factor receptor (p75NTR), specifically labels basal forebrain cholinergic neurons in mouse brain, METAB BRAIN, 15(1), 2000, pp. 17-27
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Citation: M. Beck et al., Guinea-pig primary cell cultures provide a model to study expression and amyloidogenic processing of endogenous amyloid precursor protein, NEUROSCIENC, 95(1), 2000, pp. 243-254
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Citation: M. Holzer et al., Modulation of APP processing and secretion by okadaic acid in primary guinea pig neurons, J NEURAL TR, 107(4), 2000, pp. 451-461
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Citation: M. Bigl et al., Altered phosphofructokinase mRNA levels but unchanged isoenzyme pattern inbrains from patients with Alzheimer's disease, MOL BRAIN R, 76(2), 2000, pp. 411-414
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Citation: W. Hartig et al., Abnormally phosphorylated protein tau in the cortex of aged individuals ofvarious mammalian orders, ACT NEUROP, 100(3), 2000, pp. 305-312
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Citation: E. Reisner et al., Acute effect of KA-872, a putative cognitive enhancer, on neurotransmitterreceptor binding in mouse brain, NEUROSCI L, 274(3), 1999, pp. 187-190
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Citation: S. Rossner et al., Regulated secretion of amyloid precursor protein by TrkA receptor stimulation in rat pheochromocytoma-12 cells is mitogen activated protein kinase sensitive, NEUROSCI L, 271(2), 1999, pp. 97-100
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Citation: M. Bigl et al., Activities of key glycolytic enzymes in the brains of patients with Alzheimer's disease, J NEURAL TR, 106(5-6), 1999, pp. 499-511
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Citation: C. Janke et al., Phylogenetic diversity of the expression of the microtubule-associated protein tau: implications for neurodegenerative disorders, MOL BRAIN R, 68(1-2), 1999, pp. 119-128
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Citation: S. Rossner et al., Cerebrospinal fluid cholinesterases - Markers for loss of cholinergic basal forebrain neurons?, INT J DEV N, 16(7-8), 1998, pp. 669-673
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Citation: G. Mehlhorn et al., Glucose metabolism in cholinoceptive cortical rat brain regions after basal forebrain cholinergic lesion, INT J DEV N, 16(7-8), 1998, pp. 675-690
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Citation: T. Stahl et al., Long-term basal forebrain cholinergic-rich grafts derived from trisomy 16 mice do not develop beta-amyloid pathology and neurodegeneration but demonstrate neuroinflammatory responses, INT J DEV N, 16(7-8), 1998, pp. 763-775
Citation: V. Bigl et R. Schliebs, Simulation of cortical cholinergic deficits - a novel experimental approach to study pathogenetic aspects of Alzheimer's disease, J NEURAL TR, 1998, pp. 237-247
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Bigl, V
Citation: S. Rossner et al., Neurotrophin binding to the p75 neurotrophin receptor is necessary but notsufficient to mediate NGF-effects on APP secretion in PC-12 cells, J NEURAL TR, 1998, pp. 279-285