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Results: 1-16 |
Results: 16
TRUNCATED PRESENILIN-2 DERIVED FROM DIFFERENTIALLY SPLICED MESSENGER-RNAS DOES NOT AFFECT THE RATIO OF AMYLOID BETA-PEPTIDE-1-42 1-40/
Authors: GRUNBERG J WALTER J ECKMAN C CAPELL A SCHINDZIELORZ A YOUNKIN S MEHTA N HARDY J HAASS C
Citation: J. Grunberg et al., TRUNCATED PRESENILIN-2 DERIVED FROM DIFFERENTIALLY SPLICED MESSENGER-RNAS DOES NOT AFFECT THE RATIO OF AMYLOID BETA-PEPTIDE-1-42 1-40/, NeuroReport, 9(14), 1998, pp. 3293-3299
PROTEOLYTIC PROCESSING OF ALZHEIMERS-DISEASE ASSOCIATED PROTEINS
Authors: HAASS C GRUNBERG J CAPELL A WILDBODE C LEIMER U WALTER J YAMAZAKI T IHARA I ZWECKBRONNER I JAKUBEK C BAUMEISTER R
Citation: C. Haass et al., PROTEOLYTIC PROCESSING OF ALZHEIMERS-DISEASE ASSOCIATED PROTEINS, Journal of neural transmission. Supplementum, (53), 1998, pp. 159-167
MUTANT PRESENILIN-2 TRANSGENIC MOUSE - EFFECT ON AN AGE-DEPENDENT INCREASE OF AMYLOID BETA-PROTEIN-42 IN THE BRAIN
Authors: OYAMA F SAWAMURA N KOBAYASHI K MORISHIMAKAWASHIMA M KURAMOCHI T ITO M TOMITA T MARUYAMA K SAIDO TC IWATSUBO T CAPELL A WALTER J GRUNBERG L UEYAMA Y HAASS C IHARA Y
Citation: F. Oyama et al., MUTANT PRESENILIN-2 TRANSGENIC MOUSE - EFFECT ON AN AGE-DEPENDENT INCREASE OF AMYLOID BETA-PROTEIN-42 IN THE BRAIN, Journal of neurochemistry, 71(1), 1998, pp. 313-322
THE PROTEOLYTIC FRAGMENTS OF THE ALZHEIMERS-DISEASE-ASSOCIATED PRESENILIN-1 FORM HETERODIMERS AND OCCUR AS A 100-150-KDA MOLECULAR-MASS COMPLEX
Authors: CAPELL A GRUNBERG J PESOLD B DIEHLMANN A CITRON M NIXON R BEYREUTHER K SELKOE DJ HAASS C
Citation: A. Capell et al., THE PROTEOLYTIC FRAGMENTS OF THE ALZHEIMERS-DISEASE-ASSOCIATED PRESENILIN-1 FORM HETERODIMERS AND OCCUR AS A 100-150-KDA MOLECULAR-MASS COMPLEX, The Journal of biological chemistry, 273(6), 1998, pp. 3205-3211
EXPRESSION OF ALZHEIMERS-DISEASE-ASSOCIATED PRESENILIN-1 IS CONTROLLED BY PROTEOLYTIC DEGRADATION AND COMPLEX-FORMATION
Authors: STEINER H CAPELL A PESOLD B CITRON M KLOETZEL PM SELKOE DJ ROMIG H MENDLA K HAASS C
Citation: H. Steiner et al., EXPRESSION OF ALZHEIMERS-DISEASE-ASSOCIATED PRESENILIN-1 IS CONTROLLED BY PROTEOLYTIC DEGRADATION AND COMPLEX-FORMATION, The Journal of biological chemistry, 273(48), 1998, pp. 32322-32331
THE PRESENILIN-2 MUTATION (N141I) LINKED TO FAMILIAL ALZHEIMER-DISEASE (VOLGA GERMAN FAMILIES) INCREASES THE SECRETION OF AMYLOID-BETA PROTEIN ENDING AT THE 42ND (OR 43RD) RESIDUE
Authors: TOMITA T MARUYAMA K SAIDO TC KUME H SHINOZAKI K TOKUHIRO S CAPELL A WALTER J GRUNBERG J HAASS C IWATSUBO T OBATA K
Citation: T. Tomita et al., THE PRESENILIN-2 MUTATION (N141I) LINKED TO FAMILIAL ALZHEIMER-DISEASE (VOLGA GERMAN FAMILIES) INCREASES THE SECRETION OF AMYLOID-BETA PROTEIN ENDING AT THE 42ND (OR 43RD) RESIDUE, Proceedings of the National Academy of Sciences of the United Statesof America, 94(5), 1997, pp. 2025-2030
PROTEOLYTIC PROCESSING OF THE ALZHEIMER DISEASE-ASSOCIATED PRESENILIN-1 GENERATES AN IN-VIVO SUBSTRATE FOR PROTEIN-KINASE
Authors: WALTER J GRUNBERG J CAPELL A PESOLD B SCHINDZIELORZ A CITRON M MENDLA K STGEORGEHYSLOP P MULTHAUP G SELKOE DJ HAASS C
Citation: J. Walter et al., PROTEOLYTIC PROCESSING OF THE ALZHEIMER DISEASE-ASSOCIATED PRESENILIN-1 GENERATES AN IN-VIVO SUBSTRATE FOR PROTEIN-KINASE, Proceedings of the National Academy of Sciences of the United Statesof America, 94(10), 1997, pp. 5349-5354
CELLULAR EXPRESSION AND PROTEOLYTIC PROCESSING OF PRESENILIN PROTEINSIS DEVELOPMENTALLY-REGULATED DURING NEURONAL DIFFERENTIATION
Authors: CAPELL A SAFFRICH R OLIVO JC MEYN L WALTER J GRUNBERG J MATHEWS P NIXON R DOTTI C HAASS C
Citation: A. Capell et al., CELLULAR EXPRESSION AND PROTEOLYTIC PROCESSING OF PRESENILIN PROTEINSIS DEVELOPMENTALLY-REGULATED DURING NEURONAL DIFFERENTIATION, Journal of neurochemistry, 69(6), 1997, pp. 2432-2440
ECTODOMAIN PHOSPHORYLATION OF BETA-AMYLOID PRECURSOR PROTEIN AT 2 DISTINCT CELLULAR LOCATIONS
Authors: WALTER J CAPELL A HUNG AY LANGEN H SCHNOLZER M THINAKARAN G SISODIA SS SELKOE DJ HAASS C
Citation: J. Walter et al., ECTODOMAIN PHOSPHORYLATION OF BETA-AMYLOID PRECURSOR PROTEIN AT 2 DISTINCT CELLULAR LOCATIONS, The Journal of biological chemistry, 272(3), 1997, pp. 1896-1903
INTRACELLULAR GENERATION AND ACCUMULATION OF AMYLOID BETA-PEPTIDE TERMINATING AT AMINO-ACID-42
Authors: WILDBODE C YAMAZAKI T CAPELL A LEIMER U STEINER H IHARA Y HAASS C
Citation: C. Wildbode et al., INTRACELLULAR GENERATION AND ACCUMULATION OF AMYLOID BETA-PEPTIDE TERMINATING AT AMINO-ACID-42, The Journal of biological chemistry, 272(26), 1997, pp. 16085-16088
SUBSTITUTION OF PHENYLALANINE BY PROLINE AT POSITION-19 OF AMYLOID-BETA PEPTIDE RESULTS IN AN INCREASED PRODUCTION OF AMYLOID-BETA PEPTIDESWITH ALTERNATIVE N-TERMINI AFTER PROTEIN-KINASE-C STIMULATION
Authors: CAPELL A TEPLOW DB CITRON M SELKOE DJ HAASS C
Citation: A. Capell et al., SUBSTITUTION OF PHENYLALANINE BY PROLINE AT POSITION-19 OF AMYLOID-BETA PEPTIDE RESULTS IN AN INCREASED PRODUCTION OF AMYLOID-BETA PEPTIDESWITH ALTERNATIVE N-TERMINI AFTER PROTEIN-KINASE-C STIMULATION, AMYLOID-INTERNATIONAL JOURNAL OF EXPERIMENTAL AND CLINICAL INVESTIGATION, 3(3), 1996, pp. 150-155
THE ALZHEIMERS-DISEASE-ASSOCIATED PRESENILINS ARE DIFFERENTIALLY PHOSPHORYLATED PROTEINS LOCATED PREDOMINANTLY WITHIN THE ENDOPLASMIC-RETICULUM
Authors: WALTER J CAPELL A GRUNBERG J PESOLD B SCHINDZIELORZ A PRIOR R PODLISNY MB FRASER P HYSLOP PS SELKOE DJ HAASS C
Citation: J. Walter et al., THE ALZHEIMERS-DISEASE-ASSOCIATED PRESENILINS ARE DIFFERENTIALLY PHOSPHORYLATED PROTEINS LOCATED PREDOMINANTLY WITHIN THE ENDOPLASMIC-RETICULUM, Molecular medicine, 2(6), 1996, pp. 673-691
INHIBITION OF AMYLOID BETA-PROTEIN PRODUCTION IN NEURAL CELLS BY THE SERINE-PROTEASE INHIBITOR AEBSF
Authors: CITRON M DIEHL TS CAPELL A HAASS C TEPLOW DB SELKOE DJ
Citation: M. Citron et al., INHIBITION OF AMYLOID BETA-PROTEIN PRODUCTION IN NEURAL CELLS BY THE SERINE-PROTEASE INHIBITOR AEBSF, Neuron, 17(1), 1996, pp. 171-179
THE SWEDISH MUTATION CAUSES EARLY-ONSET ALZHEIMERS-DISEASE BY BETA-SECRETASE CLEAVAGE WITHIN THE SECRETORY PATHWAY
Authors: HAASS C LEMERE CA CAPELL A CITRON M SEUBERT P SCHENK D LANNFELT L SELKOE DJ
Citation: C. Haass et al., THE SWEDISH MUTATION CAUSES EARLY-ONSET ALZHEIMERS-DISEASE BY BETA-SECRETASE CLEAVAGE WITHIN THE SECRETORY PATHWAY, Nature medicine, 1(12), 1995, pp. 1291-1296
POLARIZED SORTING OF BETA-AMYLOID PRECURSOR PROTEIN AND ITS PROTEOLYTIC PRODUCTS IN MDCK CELLS IS REGULATED BY 2 INDEPENDENT SIGNALS
Authors: HAASS C KOO EH CAPELL A TEPLOW DB SELKOE DJ
Citation: C. Haass et al., POLARIZED SORTING OF BETA-AMYLOID PRECURSOR PROTEIN AND ITS PROTEOLYTIC PRODUCTS IN MDCK CELLS IS REGULATED BY 2 INDEPENDENT SIGNALS, The Journal of cell biology, 128(4), 1995, pp. 537-547
THE VACUOLAR H-ATPASE INHIBITOR BAFILOMYCIN A1 DIFFERENTIALLY AFFECTSPROTEOLYTIC PROCESSING OF MUTANT AND WILD-TYPE BETA-AMYLOID PRECURSORPROTEIN()
Authors: HAASS C CAPELL A CITRON M TEPLOW DB SELKOE DJ
Citation: C. Haass et al., THE VACUOLAR H-ATPASE INHIBITOR BAFILOMYCIN A1 DIFFERENTIALLY AFFECTSPROTEOLYTIC PROCESSING OF MUTANT AND WILD-TYPE BETA-AMYLOID PRECURSORPROTEIN(), The Journal of biological chemistry, 270(11), 1995, pp. 6186-6192
Risultati: 1-16 |