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POUPON R
BEREZIAT G
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GASC JM
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BERTAGNA X
Citation: G. Arnaldi et al., VARIABLE EXPRESSION OF THE V1 VASOPRESSIN RECEPTOR MODULATES THE PHENOTYPIC RESPONSE OF STEROID-SECRETING ADRENOCORTICAL TUMORS, The Journal of clinical endocrinology and metabolism, 83(6), 1998, pp. 2029-2035
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PERDEREAU D
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Citation: A. Kasusjacobi et al., IDENTIFICATION OF THE RAT ADAPTER GRB14 AS AN INHIBITOR OF INSULIN ACTIONS, The Journal of biological chemistry, 273(40), 1998, pp. 26026-26035
Citation: B. Vianello et al., FUNCTIONAL INTERACTIONS OF L-162,313 WITH ANGIOTENSIN-II RECEPTOR SUBTYPES AND MUTANTS, European journal of pharmacology, 347(1), 1998, pp. 113-118
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Citation: E. Davies et al., SOMATIC MUTATIONS OF THE ANGIOTENSIN-II (AT(1)) RECEPTOR GENE ARE NOTPRESENT IN ALDOSTERONE-PRODUCING ADENOMA, The Journal of clinical endocrinology and metabolism, 82(2), 1997, pp. 611-615
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Citation: S. Conchon et al., THE C-TERMINAL 3RD INTRACELLULAR LOOP OF THE RAT AT(1A) ANGIOTENSIN RECEPTOR PLAYS A KEY ROLE IN G-PROTEIN COUPLING SPECIFICITY AND TRANSDUCTION OF THE MITOGENIC SIGNAL, The Journal of biological chemistry, 272(41), 1997, pp. 25566-25572
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Citation: B. Corman et al., ANGIOTENSIN-II POTENTIATES THE VASOPRESSIN DEPENDENT CAMP ACCUMULATION IN CHO CELLS, Journal of the American Society of Nephrology, 7(9), 1996, pp. 87-87
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Citation: C. Klingler et al., EVIDENCES FOR PHOSPHODIESTERASE INHIBITION IN THE INTERACTIONS BETWEEN VASOPRESSIN AND ANGIOTENSIN-II RECEPTORS, Kidney international, 50(5), 1996, pp. 1803-1803
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BERTAGNA X
Citation: Y. Dekeyzer et al., THE PITUITARY V3 VASOPRESSIN RECEPTOR AND THE CORTICOTROPH PHENOTYPE IN ECTOPIC ACTH SYNDROME, The Journal of clinical investigation, 97(5), 1996, pp. 1311-1318
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Citation: C. Monnot et al., POLAR RESIDUES IN THE TRANSMEMBRANE DOMAINS OF THE TYPE-1 ANGIOTENSIN-II RECEPTOR ARE REQUIRED FOR BINDING AND COUPLING - RECONSTITUTION OFTHE BINDING-SITE BY COEXPRESSION OF 2 DEFICIENT MUTANTS, The Journal of biological chemistry, 271(3), 1996, pp. 1507-1513
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Citation: Ap. Gimenezroqueplo et al., THE NATURAL MUTATION Y248C OF HUMAN ANGIOTENSINOGEN LEADS TO ABNORMALGLYCOSYLATION AND ALTERED IMMUNOLOGICAL RECOGNITION OF THE PROTEIN, The Journal of biological chemistry, 271(16), 1996, pp. 9838-9844
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PASCOE L
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CORVOL P
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Citation: Km. Curnow et al., ALTERNATIVELY SPLICED HUMAN TYPE-1 ANGIOTENSIN-II RECEPTOR MESSENGER-RNAS ARE TRANSLATED AT DIFFERENT EFFICIENCIES AND ENCODE 2 RECEPTOR ISOFORMS, Molecular endocrinology, 9(9), 1995, pp. 1250-1262
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Citation: Ys. Wen et al., LIPID SIGNAL-TRANSDUCTION PATHWAYS IN ANGIOTENSIN-II TYPE-1 RECEPTOR-TRANSFECTED FIBROBLASTS, American journal of physiology. Cell physiology, 38(2), 1995, pp. 435-442
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LLORENSCORTES C
CLAUSER E
CORVOL P
GASC JM
Citation: S. Shanmugam et al., EXPRESSION OF ANGIOTENSIN-II AT(2) RECEPTOR MESSENGER-RNA DURING DEVELOPMENT OF RAT-KIDNEY AND ADRENAL-GLAND, American journal of physiology. Renal, fluid and electrolyte physiology, 37(5), 1995, pp. 922-930
Citation: I. Leconte et E. Clauser, 2 SEQUENCES FLANKING THE MAJOR AUTOPHOSPHORYLATION SITE OF THE INSULIN-RECEPTOR ARE ESSENTIAL FOR TYROSINE KINASE ACTIVATION, Biochemical journal, 306, 1995, pp. 465-472