ACNP - Italian Periodicals Catalogue

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Results: 1-25 |

Results: 25

ANALYSIS OF OXYGEN-CONSUMPTION AND MITOCHONDRIAL PERMEABILITY WITH AGE IN MICE

Authors: GOODELL S CORTOPASSI G

Citation: S. Goodell et G. Cortopassi, ANALYSIS OF OXYGEN-CONSUMPTION AND MITOCHONDRIAL PERMEABILITY WITH AGE IN MICE, Mechanism of ageing and development, 101(3), 1998, pp. 245-256

MUTATIONS IN THE FRATAXIN GENE OF FRIEDREICHS ATAXIA CONFER SENSITIVITY TO OXIDANT STRESS

Authors: WONG A TARONI F GELLERA C CORTOPASSI G

Citation: A. Wong et al., MUTATIONS IN THE FRATAXIN GENE OF FRIEDREICHS ATAXIA CONFER SENSITIVITY TO OXIDANT STRESS, Neurology, 50(4), 1998, pp. 53002-53002

Authors: LIU YF CORTOPASSI G STEINGRIMSDOTTIR H WAUGH APW BEARE DM GREEN MHL ROBINSON DR COLE J

Citation: Yf. Liu et al., CORRELATED MUTAGENESIS OF BCL2 AND HPRT LOCI IN BLOOD-LYMPHOCYTES, Environmental and molecular mutagenesis, 29(1), 1997, pp. 36-45

MITOCHONDRIA-MEDIATED CELL INJURY

Authors: WALLACE KB EELLS JT MADEIRA VMC CORTOPASSI G JONES DP

Citation: Kb. Wallace et al., MITOCHONDRIA-MEDIATED CELL INJURY, Fundamental and applied toxicology, 38(1), 1997, pp. 23-37

THE RATE OF MITOCHONDRIAL MUTAGENESIS IS FASTER IN MICE THAN HUMANS

Authors: WANG ED WONG A CORTOPASSI G

Citation: Ed. Wang et al., THE RATE OF MITOCHONDRIAL MUTAGENESIS IS FASTER IN MICE THAN HUMANS, Mutation research, 377(2), 1997, pp. 157-166

FAMILIAL STREPTOMYCIN OTOTOXICITY IN A SOUTH-AFRICAN FAMILY - A MITOCHONDRIAL DISORDER

Authors: GARDNER JC GOLIATH R VILJOEN D SELLARS S CORTOPASSI G HUTCHIN T GREENBERG J BEIGHTON P

Citation: Jc. Gardner et al., FAMILIAL STREPTOMYCIN OTOTOXICITY IN A SOUTH-AFRICAN FAMILY - A MITOCHONDRIAL DISORDER, Journal of Medical Genetics, 34(11), 1997, pp. 904-906

FREQUENCY OF BCL-2 REARRANGEMENTS IN PERIPHERAL-BLOOD OF HIV-INFECTEDINDIVIDUALS

Authors: LIU YF CORTOPASSI G GOEDERT JJ RABKIN CS

Citation: Yf. Liu et al., FREQUENCY OF BCL-2 REARRANGEMENTS IN PERIPHERAL-BLOOD OF HIV-INFECTEDINDIVIDUALS, British Journal of Haematology, 99(2), 1997, pp. 465-466

MTDNA MUTATIONS CONFER CELLULAR-SENSITIVITY TO OXIDANT STRESS THAT ISPARTIALLY RESCUED BY CALCIUM DEPLETION AND CYCLOSPORINE-A

Authors: WONG A CORTOPASSI G

Citation: A. Wong et G. Cortopassi, MTDNA MUTATIONS CONFER CELLULAR-SENSITIVITY TO OXIDANT STRESS THAT ISPARTIALLY RESCUED BY CALCIUM DEPLETION AND CYCLOSPORINE-A, Biochemical and biophysical research communications, 239(1), 1997, pp. 139-145

OCCUPATIONAL PESTICIDE EXPOSURE AND GENETIC INSTABILITY

Authors: MCDUFFIE HH NORDAL R KIRSCH A NAKAHARA K CORTOPASSI G PABELLO P UEDA A GIBSON A

Citation: Hh. Mcduffie et al., OCCUPATIONAL PESTICIDE EXPOSURE AND GENETIC INSTABILITY, American journal of human genetics, 61(4), 1997, pp. 2260-2260

DEGENERATION OF HUMAN ONCOGENES AND MITOCHONDRIAL GENES OCCURS IN CELLS THAT EXHIBIT AGE-RELATED PATHOLOGY

Authors: CORTOPASSI G LIU YF HUTCHIN T

Citation: G. Cortopassi et al., DEGENERATION OF HUMAN ONCOGENES AND MITOCHONDRIAL GENES OCCURS IN CELLS THAT EXHIBIT AGE-RELATED PATHOLOGY, Experimental gerontology, 31(1-2), 1996, pp. 253-265

LACK OF EVIDENCE FOR AN ASSOCIATION BETWEEN THE FREQUENCY OF MUTANTS OR TRANSLOCATIONS IN CIRCULATING LYMPHOCYTES AND EXPOSURE TO RADON GASIN THE HOME

Authors: COLE J GREEN MHL BRIDGES BA WAUGH APW BEARE DM HENSHAW D LAST R LIU YF CORTOPASSI G

Citation: J. Cole et al., LACK OF EVIDENCE FOR AN ASSOCIATION BETWEEN THE FREQUENCY OF MUTANTS OR TRANSLOCATIONS IN CIRCULATING LYMPHOCYTES AND EXPOSURE TO RADON GASIN THE HOME, Radiation research, 145(1), 1996, pp. 61-69

BCL-2 POTENTIATES THE MAXIMAL CALCIUM-UPTAKE CAPACITY OF NEURAL CELL MITOCHONDRIA

Authors: MURPHY AN BREDESEN DE CORTOPASSI G WANG E FISKUM G

Citation: An. Murphy et al., BCL-2 POTENTIATES THE MAXIMAL CALCIUM-UPTAKE CAPACITY OF NEURAL CELL MITOCHONDRIA, Proceedings of the National Academy of Sciences of the United Statesof America, 93(18), 1996, pp. 9893-9898

THE MUTATION-RATE OF THE HUMAN MTDNA DELETION MTDNA(4977)

Authors: SHENKAR R NAVIDI W TAVARE S DANG MH CHOMYN A ATTARDI G CORTOPASSI G ARNHEIM N

Citation: R. Shenkar et al., THE MUTATION-RATE OF THE HUMAN MTDNA DELETION MTDNA(4977), American journal of human genetics, 59(4), 1996, pp. 772-780

MODELING THE EFFECTS OF AGE-RELATED MTDNA MUTATION ACCUMULATION - COMPLEX-I DEFICIENCY, SUPEROXIDE AND CELL-DEATH

Authors: CORTOPASSI G WANG E

Citation: G. Cortopassi et E. Wang, MODELING THE EFFECTS OF AGE-RELATED MTDNA MUTATION ACCUMULATION - COMPLEX-I DEFICIENCY, SUPEROXIDE AND CELL-DEATH, Biochimica et biophysica acta. Molecular basis of disease, 1271(1), 1995, pp. 171-176

GENOTYPIC SELECTION OF MITOCHONDRIAL AND ONCOGENIC MUTATIONS IN HUMANTISSUE SUGGESTS MECHANISMS OF AGE-RELATED PATHOPHYSIOLOGY

Authors: CORTOPASSI G LIU YF

Citation: G. Cortopassi et Yf. Liu, GENOTYPIC SELECTION OF MITOCHONDRIAL AND ONCOGENIC MUTATIONS IN HUMANTISSUE SUGGESTS MECHANISMS OF AGE-RELATED PATHOPHYSIOLOGY, Mutation research. DNAging, 338(1-6), 1995, pp. 151-159

MITOCHONDRIAL-DNA HAPLOTYPE PREDICTS DEAFNESS RISK

Authors: HUTCHIN T CORTOPASSI G

Citation: T. Hutchin et G. Cortopassi, MITOCHONDRIAL-DNA HAPLOTYPE PREDICTS DEAFNESS RISK, American journal of medical genetics, 60(6), 1995, pp. 592-592

A MITOCHONDRIAL-DNA CLONE IS ASSOCIATED WITH INCREASED RISK FOR ALZHEIMER-DISEASE

Authors: HUTCHIN T CORTOPASSI G

Citation: T. Hutchin et G. Cortopassi, A MITOCHONDRIAL-DNA CLONE IS ASSOCIATED WITH INCREASED RISK FOR ALZHEIMER-DISEASE, Proceedings of the National Academy of Sciences of the United Statesof America, 92(15), 1995, pp. 6892-6895

MITOCHONDRIAL AND APOE GENOTYPING AND RISK FOR ALZHEIMERS-DISEASE

Authors: HUTCHIN T PERICAKVANCE M CORTOPASSI G

Citation: T. Hutchin et al., MITOCHONDRIAL AND APOE GENOTYPING AND RISK FOR ALZHEIMERS-DISEASE, American journal of human genetics, 57(4), 1995, pp. 1401-1401

A MOLECULAR AND CELLULAR HYPOTHESIS FOR AMINOGLYCOSIDE-INDUCED DEAFNESS

Authors: CORTOPASSI G HUTCHIN T

Citation: G. Cortopassi et T. Hutchin, A MOLECULAR AND CELLULAR HYPOTHESIS FOR AMINOGLYCOSIDE-INDUCED DEAFNESS, Hearing research, 78(1), 1994, pp. 27-30

PROPOSED MOLECULAR AND CELLULAR MECHANISM FOR AMINOGLYCOSIDE OTOTOXICITY

Authors: HUTCHIN T CORTOPASSI G

Citation: T. Hutchin et G. Cortopassi, PROPOSED MOLECULAR AND CELLULAR MECHANISM FOR AMINOGLYCOSIDE OTOTOXICITY, Antimicrobial agents and chemotherapy, 38(11), 1994, pp. 2517-2520

PROPOSED MOLECULAR AND CELLULAR MECHANISM FOR AMINOGLYCOSIDE OTOTOXICITY

Authors: HUTCHIN T CORTOPASSI G

Citation: T. Hutchin et G. Cortopassi, PROPOSED MOLECULAR AND CELLULAR MECHANISM FOR AMINOGLYCOSIDE OTOTOXICITY, Antimicrobial agents and chemotherapy, 38(11), 1994, pp. 2517-2520

MICE WITH DUPLICATIONS AND DELETIONS AT THE TME LOCUS HAVE ALTERED MNSOD ACTIVITY

Authors: WANG ED CORTOPASSI G

Citation: Ed. Wang et G. Cortopassi, MICE WITH DUPLICATIONS AND DELETIONS AT THE TME LOCUS HAVE ALTERED MNSOD ACTIVITY, The Journal of biological chemistry, 269(36), 1994, pp. 22463-22465

A MOLECULAR-BASIS FOR HUMAN HYPERSENSITIVITY TO AMINOGLYCOSIDE ANTIBIOTICS

Authors: HUTCHIN T HAWORTH I HIGASHI K FISCHEGHODSIAN N STONEKING M SAHA N ARNOS C CORTOPASSI G

Citation: T. Hutchin et al., A MOLECULAR-BASIS FOR HUMAN HYPERSENSITIVITY TO AMINOGLYCOSIDE ANTIBIOTICS, Nucleic acids research, 21(18), 1993, pp. 4174-4179

ASSOCIATION OF A PARTICULAR POINT MUTATION OF THE MITOCHONDRIAL-DNA WITH AMINOGLYCOSIDE-INDUCED DEAFNESS

Authors: HUTCHIN TP STONEKING M QIU WQ FISCHELGHODSIAN N CORTOPASSI G

Citation: Tp. Hutchin et al., ASSOCIATION OF A PARTICULAR POINT MUTATION OF THE MITOCHONDRIAL-DNA WITH AMINOGLYCOSIDE-INDUCED DEAFNESS, American journal of human genetics, 53(3), 1993, pp. 20-20

SOMATIC MUTATIONS AT THE BCL-2-PROTO-ONCOGENE OCCUR MORE FREQUENTLY AMONG AGED THAN YOUNG HUMANS

Authors: CORTOPASSI G LIU YF

Citation: G. Cortopassi et Yf. Liu, SOMATIC MUTATIONS AT THE BCL-2-PROTO-ONCOGENE OCCUR MORE FREQUENTLY AMONG AGED THAN YOUNG HUMANS, American journal of human genetics, 53(3), 1993, pp. 288-288

Risultati: 1-25 |