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Results: 1-25 | 26-50 | 51-52
Results: 1-25/52
THE ANTIINFLAMMATORY EFFECTS OF METHOTREXATE ARE MEDIATED BY ADENOSINE IN THE ADJUVANT ARTHRITIS MODEL OF RHEUMATOID-ARTHRITIS
Authors: MONTESINOS MC YAP J MCCRARY C CRONSTEIN BN
Citation: Mc. Montesinos et al., THE ANTIINFLAMMATORY EFFECTS OF METHOTREXATE ARE MEDIATED BY ADENOSINE IN THE ADJUVANT ARTHRITIS MODEL OF RHEUMATOID-ARTHRITIS, Journal of investigative medicine, 46(3), 1998, pp. 229-229
METHOTREXATE AND SULFASALAZINE PROMOTE ADENOSINE RELEASE BY A MECHANISM THAT REQUIRES ECTO-5'-NUCLEOTIDASE-MEDIATED CONVERSION OF ADENINE-NUCLEOTIDES
Authors: MORABITO L MONTESINOS MC SCHREIBMAN DM BALTER L THOMPSON LF RESTA R CARLIN G HUIE MA CRONSTEIN BN
Citation: L. Morabito et al., METHOTREXATE AND SULFASALAZINE PROMOTE ADENOSINE RELEASE BY A MECHANISM THAT REQUIRES ECTO-5'-NUCLEOTIDASE-MEDIATED CONVERSION OF ADENINE-NUCLEOTIDES, The Journal of clinical investigation, 101(2), 1998, pp. 295-300
OCCUPANCY OF C1Q RECEPTORS ON ENDOTHELIAL-CELLS (EC) BY IMMUNE-COMPLEXES (IC) DOWN-REGULATES MESSENGER-RNA FOR STEROL 27-HYDROXYLASE (27-OH'ASE), THE MAJOR MEDIATOR OF EXTRAHEPATIC CHOLESTEROL-METABOLISM
Authors: REISS AB MALHOTRA S JAVITT NB GROSSI EA GALLOWAY AC MONTESINOS MC CRONSTEIN BN
Citation: Ab. Reiss et al., OCCUPANCY OF C1Q RECEPTORS ON ENDOTHELIAL-CELLS (EC) BY IMMUNE-COMPLEXES (IC) DOWN-REGULATES MESSENGER-RNA FOR STEROL 27-HYDROXYLASE (27-OH'ASE), THE MAJOR MEDIATOR OF EXTRAHEPATIC CHOLESTEROL-METABOLISM, Arthritis and rheumatism, 41(9), 1998, pp. 281-281
HIGH-DOSE ASPIRIN (ASA) AND SODIUM-SALICYLATE (SAL) ARE ANTIINFLAMMATORY IN NF-KAPPA-B KNOCKOUT AND WILD-TYPE MICE BY AN ADENOSINE-DEPENDENT MECHANISM
Authors: CRONSTEIN BN MONTESINOS MC WEISSMANN G
Citation: Bn. Cronstein et al., HIGH-DOSE ASPIRIN (ASA) AND SODIUM-SALICYLATE (SAL) ARE ANTIINFLAMMATORY IN NF-KAPPA-B KNOCKOUT AND WILD-TYPE MICE BY AN ADENOSINE-DEPENDENT MECHANISM, Arthritis and rheumatism, 41(9), 1998, pp. 393-393
SULFASALAZINE (SSA) IS ANTIINFLAMMATORY BY A MECHANISM WHICH IS INDEPENDENT OF NF-KAPPA-B
Authors: MONTESINOS MC DESAI A CRONSTEIN BN
Citation: Mc. Montesinos et al., SULFASALAZINE (SSA) IS ANTIINFLAMMATORY BY A MECHANISM WHICH IS INDEPENDENT OF NF-KAPPA-B, Arthritis and rheumatism, 41(9), 1998, pp. 395-395
AN ADENOSINE A2A RECEPTOR AGONIST (CGS-21680) PROMOTES WOUND CLOSURE IN NORMAL AND UROKINASE PLASMINOGEN-ACTIVATOR KNOCKOUT (UPA-KO) BUT NOT TISSUE-PLASMINOGEN ACTIVATOR KNOCKOUT (TPA-KO) MICE
Authors: MONTESINOS MC SHAPIRO R DESAI A CRONSTEIN BN
Citation: Mc. Montesinos et al., AN ADENOSINE A2A RECEPTOR AGONIST (CGS-21680) PROMOTES WOUND CLOSURE IN NORMAL AND UROKINASE PLASMINOGEN-ACTIVATOR KNOCKOUT (UPA-KO) BUT NOT TISSUE-PLASMINOGEN ACTIVATOR KNOCKOUT (TPA-KO) MICE, Arthritis and rheumatism, 41(9), 1998, pp. 951-951
MYCOPHENOLIC-ACID (MPA), THE PHARMACOLOGICALLY ACTIVE METABOLITE OF MYCOPHENOLATE-MOFETIL, INHIBITS MONOCYTE DIFFERENTIATION IN-VITRO
Authors: MERRILL JT ZHANG HW DESAI A LAHITA RG DUMONT E CRONSTEIN BN
Citation: Jt. Merrill et al., MYCOPHENOLIC-ACID (MPA), THE PHARMACOLOGICALLY ACTIVE METABOLITE OF MYCOPHENOLATE-MOFETIL, INHIBITS MONOCYTE DIFFERENTIATION IN-VITRO, Arthritis and rheumatism, 41(9), 1998, pp. 1705-1705
THE MECHANISM OF ACTION OF METHOTREXATE
Authors: CRONSTEIN BN
Citation: Bn. Cronstein, THE MECHANISM OF ACTION OF METHOTREXATE, Rheumatic diseases clinics of North America, 23(4), 1997, pp. 739
WOUND-HEALING IS ACCELERATED BY AGONISTS OF ADENOSINE A(2) (G(ALPHA-S)-LINKED) RECEPTORS
Authors: MONTESINOS MC GADANGI P LONGAKER M SUNG J LEVINE J NILSEN D REIBMAN J LI M JIANG CK HIRSCHHORN R RECHT PA OSTAD E LEVIN RI CRONSTEIN BN
Citation: Mc. Montesinos et al., WOUND-HEALING IS ACCELERATED BY AGONISTS OF ADENOSINE A(2) (G(ALPHA-S)-LINKED) RECEPTORS, The Journal of experimental medicine, 186(9), 1997, pp. 1615-1620
THE ANTIINFLAMMATORY EFFECTS OF METHOTREXATE ARE MEDIATED BY ADENOSINE IN THE ARTHRITIS MODEL OF RHEUMATOID-ARTHRITIS
Authors: YAP J MONTESINOS MC MCCRARY C CRONSTEIN BN
Citation: J. Yap et al., THE ANTIINFLAMMATORY EFFECTS OF METHOTREXATE ARE MEDIATED BY ADENOSINE IN THE ARTHRITIS MODEL OF RHEUMATOID-ARTHRITIS, Arthritis and rheumatism, 40(9), 1997, pp. 407-407
SIGNALING AT ENDOTHELIAL-CELL C1Q RECEPTORS IS MEDIATED BY THE INTRACELLULAR CALCIUM-INDEPENDENT ACTIVATION OF PROTEIN-KINASE-C
Authors: MCCRARY CT MONTESINOS MC CRONSTEIN BN
Citation: Ct. Mccrary et al., SIGNALING AT ENDOTHELIAL-CELL C1Q RECEPTORS IS MEDIATED BY THE INTRACELLULAR CALCIUM-INDEPENDENT ACTIVATION OF PROTEIN-KINASE-C, Arthritis and rheumatism, 40(9), 1997, pp. 593-593
A NOVEL FUNCTION FOR ECTO-5' NUCLEOTIDASE (5'NT) - TRANSDUCTION OF THE ANTIINFLAMMATORY EFFECTS OF METHOTRERATE(MTX) IN-VITRO AND IN-VIVO
Authors: MORABITO L MONTESINOS MC THOMPSON L RESTA R BALTER L SCHREIBMAN D CRONSTEIN BN
Citation: L. Morabito et al., A NOVEL FUNCTION FOR ECTO-5' NUCLEOTIDASE (5'NT) - TRANSDUCTION OF THE ANTIINFLAMMATORY EFFECTS OF METHOTRERATE(MTX) IN-VITRO AND IN-VIVO, Arthritis and rheumatism, 40(9), 1997, pp. 602-602
ADENOSINE A(1) RECEPTOR PROMOTION OF MULTINUCLEATED GIANT-CELL FORMATION BY HUMAN MONOCYTES - A MECHANISM FOR METHOTREXATE-INDUCED NODULOSIS IN RHEUMATOID-ARTHRITIS
Authors: MERRILL JT SHEN C SCHREIBMAN D COFFEY D ZAKHARENKO O FISHER R LAHITA RG SALMON J CRONSTEIN BN
Citation: Jt. Merrill et al., ADENOSINE A(1) RECEPTOR PROMOTION OF MULTINUCLEATED GIANT-CELL FORMATION BY HUMAN MONOCYTES - A MECHANISM FOR METHOTREXATE-INDUCED NODULOSIS IN RHEUMATOID-ARTHRITIS, Arthritis and rheumatism, 40(7), 1997, pp. 1308-1315
MECHANISM OF RHEUMATOID NODULOSIS - METHOTREXATE-ENANCED MONOCYTE FUSION REQUIRES PROTEIN-SYNTHESIS AND INTACT MICROTUBULES
Authors: MERRILL JT SHEN C GOODMAN S MITNICK H FISHER R LAHITA RG CRONSTEIN BN
Citation: Jt. Merrill et al., MECHANISM OF RHEUMATOID NODULOSIS - METHOTREXATE-ENANCED MONOCYTE FUSION REQUIRES PROTEIN-SYNTHESIS AND INTACT MICROTUBULES, Journal of investigative medicine, 44(3), 1996, pp. 220-220
ADENOSINE A(2) RECEPTOR OCCUPANCY PROMOTES FIBROBLAST AND ENDOTHELIAL-CELL MIGRATION AND WOUND-HEALING
Authors: CRONSTEIN BN MONTESINOS C GADANGI P LONGAKER M OSTAD E RECHT PA LEVIN RI
Citation: Bn. Cronstein et al., ADENOSINE A(2) RECEPTOR OCCUPANCY PROMOTES FIBROBLAST AND ENDOTHELIAL-CELL MIGRATION AND WOUND-HEALING, Journal of investigative medicine, 44(3), 1996, pp. 226-226
ADENOSINE A(2) RECEPTOR OCCUPANCY REGULATES NEUTROPHIL FUNCTION VIA THE CAMP-INDEPENDENT ACTIVATION OF A SERINE-THREONINE PROTEIN PHOSPHATASE
Authors: CRONSTEIN BN REVAN S MONTESINOS MC NAIME D LANDAU S
Citation: Bn. Cronstein et al., ADENOSINE A(2) RECEPTOR OCCUPANCY REGULATES NEUTROPHIL FUNCTION VIA THE CAMP-INDEPENDENT ACTIVATION OF A SERINE-THREONINE PROTEIN PHOSPHATASE, Journal of investigative medicine, 44(3), 1996, pp. 268-268
PURINERGIC MECHANISMS IN INFLAMMATION
Authors: CRONSTEIN BN BOUMA MG BECKER BF
Citation: Bn. Cronstein et al., PURINERGIC MECHANISMS IN INFLAMMATION, Drug development research, 39(3-4), 1996, pp. 426-435
THE ANTIINFLAMMATORY MECHANISM OF SULFASALAZINE IS RELATED TO ADENOSINE RELEASE AT INFLAMED SITES
Authors: GADANGI P LONGAKER M NAIME D LEVIN RI RECHT PA MONTESINOS MC BUCKLEY MT CARLIN G CRONSTEIN BN
Citation: P. Gadangi et al., THE ANTIINFLAMMATORY MECHANISM OF SULFASALAZINE IS RELATED TO ADENOSINE RELEASE AT INFLAMED SITES, The Journal of immunology, 156(5), 1996, pp. 1937-1941
TNF IL-1-INDUCIBLE PROTEIN TSG-6 POTENTIATES PLASMIN INHIBITION BY INTER-ALPHA-INHIBITOR AND EXERTS A STRONG ANTIINFLAMMATORY EFFECT IN-VIVO/
Authors: WISNIEWSKI HG HUA JC POPPERS DM NAIME D VILCEK J CRONSTEIN BN
Citation: Hg. Wisniewski et al., TNF IL-1-INDUCIBLE PROTEIN TSG-6 POTENTIATES PLASMIN INHIBITION BY INTER-ALPHA-INHIBITOR AND EXERTS A STRONG ANTIINFLAMMATORY EFFECT IN-VIVO/, The Journal of immunology, 156(4), 1996, pp. 1609-1615
ADENOSINE A(2) RECEPTOR OCCUPANCY REGULATES STIMULATED NEUTROPHIL FUNCTION VIA ACTIVATION OF A SERINE THREONINE PROTEIN PHOSPHATASE/
Authors: REVAN S MONTESINOS MC NAIME D LANDAU S CRONSTEIN BN
Citation: S. Revan et al., ADENOSINE A(2) RECEPTOR OCCUPANCY REGULATES STIMULATED NEUTROPHIL FUNCTION VIA ACTIVATION OF A SERINE THREONINE PROTEIN PHOSPHATASE/, The Journal of biological chemistry, 271(29), 1996, pp. 17114-17118
MECHANISMS OF THE EFFECTS OF METHOTREXATE
Authors: CRONSTEIN BN MERRILL JT
Citation: Bn. Cronstein et Jt. Merrill, MECHANISMS OF THE EFFECTS OF METHOTREXATE, Bulletin on rheumatic diseases, 45(5), 1996, pp. 6-8
SIGNAL-TRANSDUCTION IN NEUTROPHILS (PMN) - ADENOSINE INHIBITS AND AUGMENTS PROTEIN PHOSPHATIDASE ACTIVITY IN PMN CYTOSOL
Authors: HAINES KA OU GM MONTESINOS MC CRONSTEIN BN
Citation: Ka. Haines et al., SIGNAL-TRANSDUCTION IN NEUTROPHILS (PMN) - ADENOSINE INHIBITS AND AUGMENTS PROTEIN PHOSPHATIDASE ACTIVITY IN PMN CYTOSOL, Arthritis and rheumatism, 39(9), 1996, pp. 63-63
THE COMPLEMENT ACTIVATION PRODUCT C5A STIMULATES ENDOTHELIAL EXPRESSION OF A NOVEL ADHESION PROTEIN
Authors: CRONSTEIN BN LOZADA CM NAIME D MORABITO L MONTESINOS MC LEVIN RI
Citation: Bn. Cronstein et al., THE COMPLEMENT ACTIVATION PRODUCT C5A STIMULATES ENDOTHELIAL EXPRESSION OF A NOVEL ADHESION PROTEIN, Arthritis and rheumatism, 39(9), 1996, pp. 333-333
REVERSAL OF NEW BUT NOT OLD RHEUMATOID NODULES BY COLCHICINE - EVIDENCE FROM AN IN-VITRO MODEL AND CASE-REPORTS OF 14 PATIENTS
Authors: MERRILL JT CRONSTEIN BN SHEN C GOODMAN S PAGET S MITNICK H GREISMAN S BAUER B DIAKOLIOS C FISHER R LAHITA RG
Citation: Jt. Merrill et al., REVERSAL OF NEW BUT NOT OLD RHEUMATOID NODULES BY COLCHICINE - EVIDENCE FROM AN IN-VITRO MODEL AND CASE-REPORTS OF 14 PATIENTS, Arthritis and rheumatism, 39(9), 1996, pp. 1272-1272
MECHANISM OF METHOTREXATE-INDUCED RHEUMATOID NODULOSIS - ENHANCEMENT OF ADENOSINE RELEASE BY MONOCYTES IN THE PROCESS OF GRANULOMA-FORMATION
Authors: MERRILL JT SALMON J SCHREIBMAN D SHEN C ZHANG HW LAHITA RG CRONSTEIN BN
Citation: Jt. Merrill et al., MECHANISM OF METHOTREXATE-INDUCED RHEUMATOID NODULOSIS - ENHANCEMENT OF ADENOSINE RELEASE BY MONOCYTES IN THE PROCESS OF GRANULOMA-FORMATION, Arthritis and rheumatism, 39(9), 1996, pp. 1542-1542
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