AAAAAA

   
Results: 1-14 |
Results: 14
THE AMINO-ACID SUBSTITUTIONS SER288GLY AND VAL320ALA CONVERT THE 11-BETA-HYDROXYLASE (CYP11B1) INTO AN ALDOSTERONE (CYP11B2) PRODUCING ENZYME
Authors: MULATERO P CURNOW KM CORVOL P PASCOE L
Citation: P. Mulatero et al., THE AMINO-ACID SUBSTITUTIONS SER288GLY AND VAL320ALA CONVERT THE 11-BETA-HYDROXYLASE (CYP11B1) INTO AN ALDOSTERONE (CYP11B2) PRODUCING ENZYME, Journal of hypertension, 16, 1998, pp. 11-11
RECOMBINANT CYP11B GENES ENCODE ENZYMES THAT CAN CATALYZE CONVERSION OF 11-DEOXYCORTISOL TO CORTISOL, 18-HYDROXYCORTISOL, AND 18-OXOCORTISOL
Authors: MULATERO P CURNOW KM AUPETITFAISANT B FOEKLING M GOMEZSANCHEZ C VEGLIO F JEUNEMAITRE X CORVOL P PASCOE L
Citation: P. Mulatero et al., RECOMBINANT CYP11B GENES ENCODE ENZYMES THAT CAN CATALYZE CONVERSION OF 11-DEOXYCORTISOL TO CORTISOL, 18-HYDROXYCORTISOL, AND 18-OXOCORTISOL, The Journal of clinical endocrinology and metabolism, 83(11), 1998, pp. 3996-4001
ISOLATED ALDOSTERONE SYNTHASE DEFICIENCY CAUSED BY SIMULTANEOUS E198DAND V386A MUTATIONS IN THE CYP11B2 GENE
Authors: PORTRATDOYEN S TOURNIAIRE J RICHARD O MULATERO P AUPETITFAISANT B CURNOW KM PASCOE L MOREL Y
Citation: S. Portratdoyen et al., ISOLATED ALDOSTERONE SYNTHASE DEFICIENCY CAUSED BY SIMULTANEOUS E198DAND V386A MUTATIONS IN THE CYP11B2 GENE, The Journal of clinical endocrinology and metabolism, 83(11), 1998, pp. 4156-4161
THE AMINO-ACID SUBSTITUTIONS SER288GLY AND VAL320ALA CONVERT THE CORTISOL PRODUCING ENZYME, CYP11B1, INTO AN ALDOSTERONE-PRODUCING ENZYME
Authors: CURNOW KM MULATERO P EMERICBLANCHOUIN N AUPETITFAISANT B CORVOL P PASCOE L
Citation: Km. Curnow et al., THE AMINO-ACID SUBSTITUTIONS SER288GLY AND VAL320ALA CONVERT THE CORTISOL PRODUCING ENZYME, CYP11B1, INTO AN ALDOSTERONE-PRODUCING ENZYME, Nature structural biology, 4(1), 1997, pp. 32-35
ALDOSTERONE SECRETION - A MOLECULAR PERSPECTIVE
Authors: BOON WC COGHLAN JP CURNOW KM MCDOUGALL JG
Citation: Wc. Boon et al., ALDOSTERONE SECRETION - A MOLECULAR PERSPECTIVE, Trends in endocrinology and metabolism, 8(9), 1997, pp. 346-354
ANGIOTENSIN-II RECEPTORS - PROTEIN AND GENE STRUCTURES, EXPRESSION AND POTENTIAL PATHOLOGICAL INVOLVEMENTS
Authors: CLAUSER E CURNOW KM DAVIES E CONCHON S TEUTSCH B VIANELLO B MONNOT C CORVOL P
Citation: E. Clauser et al., ANGIOTENSIN-II RECEPTORS - PROTEIN AND GENE STRUCTURES, EXPRESSION AND POTENTIAL PATHOLOGICAL INVOLVEMENTS, European journal of endocrinology, 134(4), 1996, pp. 403-411
HUMAN TYPE-1 ANGIOTENSIN-II (AT(1)) RECEPTOR GENE STRUCTURE AND FUNCTION
Authors: CURNOW KM
Citation: Km. Curnow, HUMAN TYPE-1 ANGIOTENSIN-II (AT(1)) RECEPTOR GENE STRUCTURE AND FUNCTION, Clinical and experimental pharmacology and physiology, 23, 1996, pp. 67-73
POLAR RESIDUES IN THE TRANSMEMBRANE DOMAINS OF THE TYPE-1 ANGIOTENSIN-II RECEPTOR ARE REQUIRED FOR BINDING AND COUPLING - RECONSTITUTION OFTHE BINDING-SITE BY COEXPRESSION OF 2 DEFICIENT MUTANTS
Authors: MONNOT C BIHOREAU C CONCHON S CURNOW KM CORVOL P CLAUSER E
Citation: C. Monnot et al., POLAR RESIDUES IN THE TRANSMEMBRANE DOMAINS OF THE TYPE-1 ANGIOTENSIN-II RECEPTOR ARE REQUIRED FOR BINDING AND COUPLING - RECONSTITUTION OFTHE BINDING-SITE BY COEXPRESSION OF 2 DEFICIENT MUTANTS, The Journal of biological chemistry, 271(3), 1996, pp. 1507-1513
ALTERNATIVELY SPLICED HUMAN TYPE-1 ANGIOTENSIN-II RECEPTOR MESSENGER-RNAS ARE TRANSLATED AT DIFFERENT EFFICIENCIES AND ENCODE 2 RECEPTOR ISOFORMS
Authors: CURNOW KM PASCOE L DAVIES E WHITE PC CORVOL P CLAUSER E
Citation: Km. Curnow et al., ALTERNATIVELY SPLICED HUMAN TYPE-1 ANGIOTENSIN-II RECEPTOR MESSENGER-RNAS ARE TRANSLATED AT DIFFERENT EFFICIENCIES AND ENCODE 2 RECEPTOR ISOFORMS, Molecular endocrinology, 9(9), 1995, pp. 1250-1262
EXPRESSION CLONING OF TYPE-2 ANGIOTENSIN-II RECEPTOR REVEALS A UNIQUECLASS OF 7-TRANSMEMBRANE RECEPTORS - COMMENT
Authors: CURNOW KM
Citation: Km. Curnow, EXPRESSION CLONING OF TYPE-2 ANGIOTENSIN-II RECEPTOR REVEALS A UNIQUECLASS OF 7-TRANSMEMBRANE RECEPTORS - COMMENT, Journal of endocrinological investigation, 18(7), 1995, pp. 566-570
GENETIC-RECOMBINATION AS A CAUSE OF INHERITED DISORDERS OF ALDOSTERONE AND CORTISOL BIOSYNTHESIS AND A CONTRIBUTOR TO GENETIC-VARIATION IN BLOOD-PRESSURE
Authors: PASCOE L CURNOW KM
Citation: L. Pascoe et Km. Curnow, GENETIC-RECOMBINATION AS A CAUSE OF INHERITED DISORDERS OF ALDOSTERONE AND CORTISOL BIOSYNTHESIS AND A CONTRIBUTOR TO GENETIC-VARIATION IN BLOOD-PRESSURE, Steroids, 60(1), 1995, pp. 22-27
GLUCOCORTICOID-SUPPRESSIBLE HYPERALDOSTERONISM AND ADRENAL-TUMORS OCCURRING IN A SINGLE FRENCH PEDIGREE
Authors: PASCOE L JEUNEMAITRE X LEBRETHON MC CURNOW KM GOMEZSANCHEZ CE GASC JM SAEZ JM CORVOL P
Citation: L. Pascoe et al., GLUCOCORTICOID-SUPPRESSIBLE HYPERALDOSTERONISM AND ADRENAL-TUMORS OCCURRING IN A SINGLE FRENCH PEDIGREE, The Journal of clinical investigation, 96(5), 1995, pp. 2236-2246
DISORDERS OF STEROID 11-BETA-HYDROXYLASE ISOZYMES
Authors: WHITE PC CURNOW KM PASCOE L
Citation: Pc. White et al., DISORDERS OF STEROID 11-BETA-HYDROXYLASE ISOZYMES, Endocrine reviews, 15(4), 1994, pp. 421-438
MUTATIONS IN THE CYP11B1 GENE CAUSING CONGENITAL ADRENAL-HYPERPLASIA AND HYPERTENSION CLUSTER IN EXON-6, EXON-7, AND EXON-8
Authors: CURNOW KM SLUTSKER L VITEK J COLE T SPEISER PW NEW MI WHITE PC PASCOE L
Citation: Km. Curnow et al., MUTATIONS IN THE CYP11B1 GENE CAUSING CONGENITAL ADRENAL-HYPERPLASIA AND HYPERTENSION CLUSTER IN EXON-6, EXON-7, AND EXON-8, Proceedings of the National Academy of Sciences of the United Statesof America, 90(10), 1993, pp. 4552-4556
Risultati: 1-14 |