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Results: 1-13 |
Results: 13
Hypogammaglobulinemia and reduced numbers of B-cells in children with myelodysplastic syndrome
Authors: Srivannaboon, K Conley, ME Coustan-Smith, E Wang, WC
Citation: K. Srivannaboon et al., Hypogammaglobulinemia and reduced numbers of B-cells in children with myelodysplastic syndrome, J PED H ONC, 23(2), 2001, pp. 122-125
Detection of minimal residual disease in acute lymphoblastic leukemia: theSt Jude experience
Authors: Campana, D Neale, GAM Coustan-Smith, E Pui, CH
Citation: D. Campana et al., Detection of minimal residual disease in acute lymphoblastic leukemia: theSt Jude experience, LEUKEMIA, 15(2), 2001, pp. 278-279
Identification of novel markers for monitoring minimal residual disease inacute lymphoblastic leukemia
Authors: Chen, JS Coustan-Smith, E Suzuki, T Neale, GA Mihara, K Pui, CH Campana, D
Citation: Js. Chen et al., Identification of novel markers for monitoring minimal residual disease inacute lymphoblastic leukemia, BLOOD, 97(7), 2001, pp. 2115-2120
Simple methods for the rapid exchange of flow cytometric data between remote centers
Authors: Lorenzana, R Coustan-Smith, E Antillon, F Ribeiro, RC Campana, D
Citation: R. Lorenzana et al., Simple methods for the rapid exchange of flow cytometric data between remote centers, LEUKEMIA, 14(2), 2000, pp. 336-337
Clinical importance of minimal residual disease in childhood acute lymphoblastic leukemia
Authors: Coustan-Smith, E Sancho, J Hancock, ML Boyett, JM Behm, FG Raimondi, SC Sandlund, JT Rivera, GK Rubnitz, JE Ribeiro, RC Pui, CH Campana, D
Citation: E. Coustan-smith et al., Clinical importance of minimal residual disease in childhood acute lymphoblastic leukemia, BLOOD, 96(8), 2000, pp. 2591-2596
Clinical importance of minimal residual disease in childhood acute lymphoblastic leukemia
Authors: Coustan-Smith, E Sancho, J Hancock, ML Boyett, JM Behm, FG Raimondi, SC Sandlund, JT Rivera, GK Rubnitz, JE Ribeiro, RC Pui, CH Campana, D
Citation: E. Coustan-smith et al., Clinical importance of minimal residual disease in childhood acute lymphoblastic leukemia, BLOOD, 96(8), 2000, pp. 2691-2696
CD38 ligation inhibits normal and leukemic myelopoiesis
Authors: Todisco, E Suzuki, T Srivannaboon, K Coustan-Smith, E Raimondi, SC Behm, FG Kitanaka, A Campana, D
Citation: E. Todisco et al., CD38 ligation inhibits normal and leukemic myelopoiesis, BLOOD, 95(2), 2000, pp. 535-542
Tandem application of flow cytometry and polymerase chain reaction for comprehensive detection of minimal residual disease in childhood acute lymphoblastic leukemia
Authors: Neale, GAM Coustan-Smith, E Pan, Q Chen, X Gruhn, B Stow, P Behm, FG Pui, CH Campana, D
Citation: Gam. Neale et al., Tandem application of flow cytometry and polymerase chain reaction for comprehensive detection of minimal residual disease in childhood acute lymphoblastic leukemia, LEUKEMIA, 13(8), 1999, pp. 1221-1226
Detection of minimal residual disease in acute leukemia by flow cytometry
Authors: Campana, D Coustan-Smith, E
Citation: D. Campana et E. Coustan-smith, Detection of minimal residual disease in acute leukemia by flow cytometry, CYTOMETRY, 38(4), 1999, pp. 139-152
An essential role for BLNK in human B cell development
Authors: Minegishi, Y Rohrer, J Coustan-Smith, E Lederman, HM Pappu, R Campana, D Chan, AC Conley, ME
Citation: Y. Minegishi et al., An essential role for BLNK in human B cell development, SCIENCE, 286(5446), 1999, pp. 1954-1957
CD38-mediated signaling events in murine pro-B cells expressing human CD38with or without its cytoplasmic domain
Authors: Kitanaka, A Suzuki, T Ito, C Nishigaki, H Coustan-Smith, E Tanaka, T Kubota, Y Campana, D
Citation: A. Kitanaka et al., CD38-mediated signaling events in murine pro-B cells expressing human CD38with or without its cytoplasmic domain, J IMMUNOL, 162(4), 1999, pp. 1952-1958
Mutations in Ig alpha (CD79a) result in in B-cell development
Authors: Minegishi, Y Coustan-Smith, E Rapalus, L Ersoy, F Campana, D Conley, ME
Citation: Y. Minegishi et al., Mutations in Ig alpha (CD79a) result in in B-cell development, J CLIN INV, 104(8), 1999, pp. 1115-1121
Hyperdiploid acute lymphoblastic leukemia with 51 to 65 chromosomes: A distinct biological entity with a marked propensity to undergo apoptosis
Authors: Ito, C Kumagai, M Manabe, A Coustan-Smith, E Raimondi, SC Behm, FG Murti, KG Rubnitz, JE Pui, CH Campana, D
Citation: C. Ito et al., Hyperdiploid acute lymphoblastic leukemia with 51 to 65 chromosomes: A distinct biological entity with a marked propensity to undergo apoptosis, BLOOD, 93(1), 1999, pp. 315-320
Risultati: 1-13 |