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Results: 1-20 |
Results: 20
Hematologically important mutations: X-linked chronic granulomatous disease (Second update)
Authors: Heyworth, PG Curnutte, JT Rae, J Noack, D Roos, D van Koppen, E Cross, AR
Citation: Pg. Heyworth et al., Hematologically important mutations: X-linked chronic granulomatous disease (Second update), BL CELL M D, 27(1), 2001, pp. 16-26
Induction of gp91-phox, a component of the phagocyte NADPH oxidase, in microglial cells during central nervous system inflammation
Authors: Green, SP Cairns, B Rae, J Errett-Baroncini, C Hongo, JAS Erickson, RW Curnutte, JT
Citation: Sp. Green et al., Induction of gp91-phox, a component of the phagocyte NADPH oxidase, in microglial cells during central nervous system inflammation, J CEREBR B, 21(4), 2001, pp. 374-384
Transplantation of a fetus with paternal Thy-1(+)CD34(+) cells for chronicgranulomatous disease
Authors: Muench, MO Rae, J Barcena, A Leemhuis, T Farrell, J Humeau, L Maxwell-Wiggins, JR Capper, J Mychaliska, GB Albanese, CT Martin, T Tsukamoto, A Curnutte, JT Harrison, MR
Citation: Mo. Muench et al., Transplantation of a fetus with paternal Thy-1(+)CD34(+) cells for chronicgranulomatous disease, BONE MAR TR, 27(4), 2001, pp. 355-364
Role of C5a in multiorgan failure during sepsis
Authors: Huber-Lang, M Sarma, VJ Lu, KT McGuire, SR Padgaonkar, VA Guo, RF Younkin, EM Kunkel, RG Ding, JB Erickson, R Curnutte, JT Ward, PA
Citation: M. Huber-lang et al., Role of C5a in multiorgan failure during sepsis, J IMMUNOL, 166(2), 2001, pp. 1193-1199
Autosomal recessive chronic granulomatous disease caused by defects in NCF-1, the gene encoding the phagocyte p47-phox: mutations not arising in the NCF-1 pseudogenes
Authors: Noack, D Rae, J Cross, AR Ellis, BA Newburger, PE Curnutte, JT Heyworth, PG
Citation: D. Noack et al., Autosomal recessive chronic granulomatous disease caused by defects in NCF-1, the gene encoding the phagocyte p47-phox: mutations not arising in the NCF-1 pseudogenes, BLOOD, 97(1), 2001, pp. 305-311
Experimental histoplasmosis in mice treated with anti-murine interferon-gamma antibody and in interferon-gamma gene knockout mice
Authors: Clemons, KV Darbonne, WC Curnutte, JT Sobel, RA Stevens, DA
Citation: Kv. Clemons et al., Experimental histoplasmosis in mice treated with anti-murine interferon-gamma antibody and in interferon-gamma gene knockout mice, MICROBES IN, 2(9), 2000, pp. 997-1001
Hematologically important mutations: The autosomal recessive forms of chronic granulomatous disease (First update)
Authors: Cross, AR Noack, D Rae, J Curnutte, JT Heyworth, PG
Citation: Ar. Cross et al., Hematologically important mutations: The autosomal recessive forms of chronic granulomatous disease (First update), BL CELL M D, 26(5), 2000, pp. 561
Genomic structure of the human p47-phox (NCF1) Gene
Authors: Chanock, SJ Roesler, J Zhan, SX Hopkins, P Lee, P Barrett, DT Christensen, BL Curnutte, JT Gorlach, A
Citation: Sj. Chanock et al., Genomic structure of the human p47-phox (NCF1) Gene, BL CELL M D, 26(1), 2000, pp. 37-46
Molecular analysis of 9 new families with chronic granulomatous disease caused by mutations in CYBA, the gene encoding p22(phox)
Authors: Rae, J Noack, D Heyworth, PG Ellis, BA Curnutte, JT Cross, AR
Citation: J. Rae et al., Molecular analysis of 9 new families with chronic granulomatous disease caused by mutations in CYBA, the gene encoding p22(phox), BLOOD, 96(3), 2000, pp. 1106-1112
Recombination events between the p47-phox gene and its highly homologous pseudogenes are the main cause of autosomal recessive chronic granulomatous disease
Authors: Roesler, J Curnutte, JT Rae, J Barrett, D Patino, P Chanock, SJ Goerlach, A
Citation: J. Roesler et al., Recombination events between the p47-phox gene and its highly homologous pseudogenes are the main cause of autosomal recessive chronic granulomatous disease, BLOOD, 95(6), 2000, pp. 2150-2156
Molecular analysis of chronic granulomatous disease caused by defects in gp91-phox
Authors: Patino, PJ Perez, JE Lopez, JA Condino-Neto, A Grumach, AS Botero, JH Curnutte, JT de Olarte, DG
Citation: Pj. Patino et al., Molecular analysis of chronic granulomatous disease caused by defects in gp91-phox, HUM MUTAT, 13(1), 1999, pp. 29-37
A novel mutation in the CYBB gene resulting in an unexpected pattern of exon skipping and chronic granulomatous disease
Authors: Noack, D Heyworth, PG Curnutte, JT Rae, J Cross, AR
Citation: D. Noack et al., A novel mutation in the CYBB gene resulting in an unexpected pattern of exon skipping and chronic granulomatous disease, BBA-MOL BAS, 1454(3), 1999, pp. 270-274
Autosomal recessive chronic granulomatous disease caused by novel mutations in NCF-2, the gene encoding the p67-phox component of phagocyte NADPH oxidase
Authors: Noack, D Rae, J Cross, AR Munoz, J Salmen, S Mendoza, JA Rossi, N Curnutte, JT Heyworth, PG
Citation: D. Noack et al., Autosomal recessive chronic granulomatous disease caused by novel mutations in NCF-2, the gene encoding the p67-phox component of phagocyte NADPH oxidase, HUM GENET, 105(5), 1999, pp. 460-467
The mechanism of activation of NADPH oxidase in the cell-free system: the activation process is primarily catalytic and not through the formation of a stoichiometric complex
Authors: Cross, AR Erickson, RW Curnutte, JT
Citation: Ar. Cross et al., The mechanism of activation of NADPH oxidase in the cell-free system: the activation process is primarily catalytic and not through the formation of a stoichiometric complex, BIOCHEM J, 341, 1999, pp. 251-255
Spontaneous activation of NADPH oxidase in a cell-free system: unexpected multiple effects of magnesium ion concentrations
Authors: Cross, AR Erickson, RW Ellis, BA Curnutte, JT
Citation: Ar. Cross et al., Spontaneous activation of NADPH oxidase in a cell-free system: unexpected multiple effects of magnesium ion concentrations, BIOCHEM J, 338, 1999, pp. 229-233
Activation of human neutrophil NADPH oxidase by phosphatidic acid or diacylglycerol in a cell-free system - Activity of diacylglycerol is dependent on its conversion to phosphatidic acid
Authors: Erickson, RW Langel-Peveri, P Traynor-Kaplan, AE Heyworth, PG Curnutte, JT
Citation: Rw. Erickson et al., Activation of human neutrophil NADPH oxidase by phosphatidic acid or diacylglycerol in a cell-free system - Activity of diacylglycerol is dependent on its conversion to phosphatidic acid, J BIOL CHEM, 274(32), 1999, pp. 22243-22250
Simultaneous presence of p47(phox) and flavocytochrome b(-245), are required for the activation of NADPH oxidase by anionic amphiphiles - Evidence for an intermediate state of oxidase activation
Authors: Cross, AR Erickson, RW Curnutte, JT
Citation: Ar. Cross et al., Simultaneous presence of p47(phox) and flavocytochrome b(-245), are required for the activation of NADPH oxidase by anionic amphiphiles - Evidence for an intermediate state of oxidase activation, J BIOL CHEM, 274(22), 1999, pp. 15519-15525
Molecular characterization of autosomal recessive chronic granulomatous disease caused by a defect of the nicotinamide adenine dinucleotide phosphate(reduced form) oxidase component p67-phox
Authors: Patino, PJ Rae, J Noack, D Erickson, R Ding, JB de Olarte, DG Curnutte, JT
Citation: Pj. Patino et al., Molecular characterization of autosomal recessive chronic granulomatous disease caused by a defect of the nicotinamide adenine dinucleotide phosphate(reduced form) oxidase component p67-phox, BLOOD, 94(7), 1999, pp. 2505-2514
Structure and regulation of NADPH oxidase of phagocytic leukocytes - Insights from chronic granulomatous disease
Authors: Heyworth, PG Curnutte, JT Badwey, JA
Citation: Pg. Heyworth et al., Structure and regulation of NADPH oxidase of phagocytic leukocytes - Insights from chronic granulomatous disease, CUR INF RES, 1999, pp. 165-191
Host defense molecule polymorphisms influence the risk for immune-mediatedcomplications in chronic granulomatous disease
Authors: Foster, CB Lehrnbecher, T Mol, F Steinberg, SM Venzon, DJ Walsh, TJ Noack, D Rae, J Winkelstein, JA Curnutte, JT Chanock, SJ
Citation: Cb. Foster et al., Host defense molecule polymorphisms influence the risk for immune-mediatedcomplications in chronic granulomatous disease, J CLIN INV, 102(12), 1998, pp. 2146-2155
Risultati: 1-20 |