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Results: 1-10 |
Results: 10
REDUCTION IN ENDOTOXIN-INDUCED ORGAN DYSFUNCTION AND CYTOKINE SECRETION BY A CYCLIC NITRONE ANTIOXIDANT
Authors: DOWNS TR DAGE RC FRENCH JF
Citation: Tr. Downs et al., REDUCTION IN ENDOTOXIN-INDUCED ORGAN DYSFUNCTION AND CYTOKINE SECRETION BY A CYCLIC NITRONE ANTIOXIDANT, International journal of immunopharmacology, 17(7), 1995, pp. 571-580
DUAL INHIBITION OF ANGIOTENSIN-CONVERTING ENZYME AND NEUTRAL ENDOPEPTIDASE IN RATS WITH HYPERTENSION
Authors: FRENCH JF ANDERSON BA DOWNS TR DAGE RC
Citation: Jf. French et al., DUAL INHIBITION OF ANGIOTENSIN-CONVERTING ENZYME AND NEUTRAL ENDOPEPTIDASE IN RATS WITH HYPERTENSION, Journal of cardiovascular pharmacology, 26(1), 1995, pp. 107-113
GROWTH HORMONE-RELEASING HORMONE IMMUNOREACTIVITY IN MOUSE PLACENTA, MATERNAL BLOOD, AND AMNIOTIC-FLUID - MOLECULAR CHARACTERIZATION AND SECRETION FROM PRIMARY-CELL CULTURES IN-VITRO
Authors: MIZOBUCHI M DOWNS TR FROHMAN LA
Citation: M. Mizobuchi et al., GROWTH HORMONE-RELEASING HORMONE IMMUNOREACTIVITY IN MOUSE PLACENTA, MATERNAL BLOOD, AND AMNIOTIC-FLUID - MOLECULAR CHARACTERIZATION AND SECRETION FROM PRIMARY-CELL CULTURES IN-VITRO, Endocrinology, 136(4), 1995, pp. 1731-1736
DEVELOPMENT OF PITUITARY CELL-TYPES IN THE SPONTANEOUS DWARF (DW) RAT- EVIDENCE FOR AN ISOLATED DEFECT IN SOMATOTROPH DIFFERENTIATION
Authors: ZEITLER P DOWNS TR FROHMAN LA
Citation: P. Zeitler et al., DEVELOPMENT OF PITUITARY CELL-TYPES IN THE SPONTANEOUS DWARF (DW) RAT- EVIDENCE FOR AN ISOLATED DEFECT IN SOMATOTROPH DIFFERENTIATION, Endocrine, 2(8), 1994, pp. 729-733
PROTECTIVE EFFECTS OF A CYCLIC NITRONE ANTIOXIDANT IN ANIMAL-MODELS OF ENDOTOXIC-SHOCK AND CHRONIC BACTEREMIA
Authors: FRENCH JF THOMAS CE DOWNS TR OHLWEILER DF CARR AA DAGE RC
Citation: Jf. French et al., PROTECTIVE EFFECTS OF A CYCLIC NITRONE ANTIOXIDANT IN ANIMAL-MODELS OF ENDOTOXIC-SHOCK AND CHRONIC BACTEREMIA, Circulatory shock, 43(3), 1994, pp. 130-136
DEVELOPMENT OF NEUROEPITHELIAL TUMORS OF THE ADRENAL-MEDULLA IN TRANSGENIC MICE EXPRESSING A MOUSE HYPOTHALAMIC GROWTH HORMONE-RELEASING HORMONE PROMOTER-SIMIAN VIRUS-40 T-ANTIGEN FUSION GENE
Authors: GIRALDI FP MIZOBUCHI M HOROWITZ ZD DOWNS TR ALEPPO G KIER A WAGNER T YUN JS KOPCHICK JJ FROHMAN LA
Citation: Fp. Giraldi et al., DEVELOPMENT OF NEUROEPITHELIAL TUMORS OF THE ADRENAL-MEDULLA IN TRANSGENIC MICE EXPRESSING A MOUSE HYPOTHALAMIC GROWTH HORMONE-RELEASING HORMONE PROMOTER-SIMIAN VIRUS-40 T-ANTIGEN FUSION GENE, Endocrinology, 134(3), 1994, pp. 1219-1224
MOUSE HYPOTHALAMIC GROWTH HORMONE-RELEASING HORMONE AND SOMATOSTATIN RESPONSES TO PROBES OF SIGNAL-TRANSDUCTION SYSTEMS
Authors: SATO M DOWNS TR FROHMAN LA
Citation: M. Sato et al., MOUSE HYPOTHALAMIC GROWTH HORMONE-RELEASING HORMONE AND SOMATOSTATIN RESPONSES TO PROBES OF SIGNAL-TRANSDUCTION SYSTEMS, Peptides, 14(4), 1993, pp. 671-677
OBESITY-ASSOCIATED DECREASE IN GROWTH HORMONE-RELEASING HORMONE GENE-EXPRESSION - A MECHANISM FOR REDUCED GROWTH-HORMONE MESSENGER-RNA LEVELS IN GENETICALLY-OBESE ZUCKER RATS
Authors: AHMAD I FINKELSTEIN JA DOWNS TR FROHMAN LA
Citation: I. Ahmad et al., OBESITY-ASSOCIATED DECREASE IN GROWTH HORMONE-RELEASING HORMONE GENE-EXPRESSION - A MECHANISM FOR REDUCED GROWTH-HORMONE MESSENGER-RNA LEVELS IN GENETICALLY-OBESE ZUCKER RATS, Neuroendocrinology, 58(3), 1993, pp. 332-337
CONGENITAL GIGANTISM DUE TO GROWTH HORMONE-RELEASING HORMONE EXCESS AND PITUITARY HYPERPLASIA WITH ADENOMATOUS TRANSFORMATION
Authors: ZIMMERMAN D YOUNG WF EBERSOLD MJ SCHEITHAUER BW KOVACS K HORVATH E WHITAKER MD EBERHARDT NL DOWNS TR FROHMAN LA
Citation: D. Zimmerman et al., CONGENITAL GIGANTISM DUE TO GROWTH HORMONE-RELEASING HORMONE EXCESS AND PITUITARY HYPERPLASIA WITH ADENOMATOUS TRANSFORMATION, The Journal of clinical endocrinology and metabolism, 76(1), 1993, pp. 216-222
IMPAIRED GROWTH HORMONE-RELEASING HORMONE SIGNAL-TRANSDUCTION IN THE DWARF (DW) RAT IS INDEPENDENT OF A GENERALIZED DEFECT IN THE STIMULATORY G-PROTEIN, G(S)ALPHA
Authors: ZEITLER P DOWNS TR FROHMAN LA
Citation: P. Zeitler et al., IMPAIRED GROWTH HORMONE-RELEASING HORMONE SIGNAL-TRANSDUCTION IN THE DWARF (DW) RAT IS INDEPENDENT OF A GENERALIZED DEFECT IN THE STIMULATORY G-PROTEIN, G(S)ALPHA, Endocrinology, 133(6), 1993, pp. 2782-2784
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