Authors:
Vorwerk, CK
Naskar, R
Schuettauf, F
Zurakowski, D
McDermott, LM
Quinto, KM
Dreyer, EB
Citation: Ck. Vorwerk et al., Excitotoxicity can be mediated through an interaction within the optic nerve; activation of cell body NMDA receptors is not required, VET OPHTHAL, 4(3), 2001, pp. 201-204
Citation: Ks. Shindler et al., Caspase inhibitors block zinc-chelator induced death of retinal ganglion cells, NEUROREPORT, 11(10), 2000, pp. 2299-2302
Authors:
Schuettauf, F
Naskar, R
Vorwerk, CK
Zurakowski, D
Dreyer, EB
Citation: F. Schuettauf et al., Ganglion cell loss after optic nerve crush mediated through AMPA-kainate and NMDA receptors, INV OPHTH V, 41(13), 2000, pp. 4313-4316
Authors:
Vorwerk, CK
Naskar, R
Schuettauf, F
Quinto, K
Zurakowski, D
Gochenauer, G
Robinson, MB
Mackler, SA
Dreyer, EB
Citation: Ck. Vorwerk et al., Depression of retinal glutamate transporter function leads to elevated intravitreal glutamate levels and ganglion cell death, INV OPHTH V, 41(11), 2000, pp. 3615-3621
Authors:
Vorwerk, CK
Kreutz, MR
Bockers, TM
Brosz, M
Dreyer, EB
Sabel, BA
Citation: Ck. Vorwerk et al., Susceptibility of retinal ganglion cells to excitotoxicity depends on somasize and retinal eccentricity, CURR EYE R, 19(1), 1999, pp. 59-65
Authors:
Dreyer, EB
Zurakowski, D
Gorla, M
Vorwerk, CK
Lipton, SA
Citation: Eb. Dreyer et al., The contribution of various NOS gene products to HIV-1 coat protein (gp120)-mediated retinal ganglion cell injury, INV OPHTH V, 40(5), 1999, pp. 983-989
Citation: Ck. Vorwerk et al., An experimental basis for implicating excitotoxicity in glaucomatous opticneuropathy, SURV OPHTHA, 43, 1999, pp. S142-S150