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Authors: CUDA G FANANAPAZIR L ZHU WS SELLERS JR EPSTEIN ND
Citation: G. Cuda et al., SKELETAL-MUSCLE EXPRESSION AND ABNORMAL FUNCTION OF BETA-MYOSIN IN HYPERTROPHIC CARDIOMYOPATHY, The Journal of clinical investigation, 91(6), 1993, pp. 2861-2865

Authors: FANANAPAZIR L CUDA G SELLERS J EPSTEIN ND
Citation: L. Fananapazir et al., DIFFERENCES IN MOTILITY ACTIVITY OF BETA-MYOSIN FROM SKELETAL-MUSCLE IN HYPERTROPHIC CARDIOMYOPATHY KINDREDS WITH INDEPENDENT BETA-MYOSIN HEAVY-CHAIN GENE-MUTATIONS RESULTING IN THE IDENTICAL 741 GLY-]ARG AMINO-ACID SUBSTITUTION, Circulation, 88(4), 1993, pp. 343-343

Authors: CUDA G SELLERS J EPSTEIN ND FANANAPAZIR L
Citation: G. Cuda et al., IN-VITRO MOTILITY ACTIVITY OF BETA-CARDIAC MYOSIN DEPENDS ON THE NATURE OF THE BETA-MYOSIN HEAVY-CHAIN GENE MUTATION IN HYPERTROPHIC CARDIOMYOPATHY, Circulation, 88(4), 1993, pp. 343-343

Authors: SWEENEY HL LANKFORD E DANTZIG J EPSTEIN ND FANANAPAZIR L
Citation: Hl. Sweeney et al., ABNORMAL CONTRACTILE PROPERTIES OF MUSCLE-FIBERS EXPRESSING MUTATIONSIN BETA-MYOSIN THAT CAUSE HYPERTROPHIC CARDIOMYOPATHY, Circulation, 88(4), 1993, pp. 525-525

Authors: KENNEY RT MALECH HL EPSTEIN ND ROBERTS RL LETO TL
Citation: Rt. Kenney et al., CHARACTERIZATION OF THE P67(PHOX) GENE - GENOMIC ORGANIZATION AND RESTRICTION-FRAGMENT-LENGTH-POLYMORPHISM ANALYSIS FOR PRENATAL-DIAGNOSIS IN CHRONIC GRANULOMATOUS-DISEASE, Blood, 82(12), 1993, pp. 3739-3744
Risultati: 1-25 | 26-30 |