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Results: 1-9 |
Results: 9
QUINIDINE NORMALIZES THE OPEN DURATION OF SLOW-CHANNEL MUTANTS OF THEACETYLCHOLINE-RECEPTOR
Authors: FUKUDOME T OHNO K BRENGMAN JM ENGEL AG
Citation: T. Fukudome et al., QUINIDINE NORMALIZES THE OPEN DURATION OF SLOW-CHANNEL MUTANTS OF THEACETYLCHOLINE-RECEPTOR, NeuroReport, 9(8), 1998, pp. 1907-1911
MODE SWITCHING KINETICS PRODUCED BY A NATURALLY-OCCURRING MUTATION INTHE CYTOPLASMIC LOOP OF THE HUMAN ACETYLCHOLINE-RECEPTOR EPSILON-SUBUNIT
Authors: MILONE M WANG HL OHNO K PRINCE R FUKUDOME T SHEN XM BRENGMAN JM GRIGGS RC SINE SM ENGEL AG
Citation: M. Milone et al., MODE SWITCHING KINETICS PRODUCED BY A NATURALLY-OCCURRING MUTATION INTHE CYTOPLASMIC LOOP OF THE HUMAN ACETYLCHOLINE-RECEPTOR EPSILON-SUBUNIT, Neuron, 20(3), 1998, pp. 575-588
MYOPATHY, MYASTHENIA, AND EPIDERMOLYSIS-BULLOSA SIMPLEX DUE TO PLECTIN DEFICIENCY
Authors: BANWELL BL FUKUDOME T SHEN XM STILLING G RUSSELL J ENGEL AG
Citation: Bl. Banwell et al., MYOPATHY, MYASTHENIA, AND EPIDERMOLYSIS-BULLOSA SIMPLEX DUE TO PLECTIN DEFICIENCY, Annals of neurology, 44(3), 1998, pp. 83-83
SLOW-CHANNEL MYASTHENIC SYNDROME CAUSED BY ENHANCED ACTIVATION, DESENSITIZATION, AND AGONIST BINDING-AFFINITY ATTRIBUTABLE TO MUTATION IN THE M2 DOMAIN OF THE ACETYLCHOLINE-RECEPTOR ALPHA-SUBUNIT
Authors: MILONE M WANG HL OHNO K FUKUDOME T PRUITT JN BREN N SINE SM ENGEL AG
Citation: M. Milone et al., SLOW-CHANNEL MYASTHENIC SYNDROME CAUSED BY ENHANCED ACTIVATION, DESENSITIZATION, AND AGONIST BINDING-AFFINITY ATTRIBUTABLE TO MUTATION IN THE M2 DOMAIN OF THE ACETYLCHOLINE-RECEPTOR ALPHA-SUBUNIT, The Journal of neuroscience, 17(15), 1997, pp. 5651-5665
CONGENITAL MYASTHENIC SYNDROME DUE TO A NULL MUTATION AND AN INFRAME DUPLICATION OF THE ACETYLCHOLINE-RECEPTOR (ACHR) EPSILON-SUBUNIT GENE
Authors: MILONE M OHNO K FUKUDOME T SHEN XM BRENGMAN JM GRIGGS RC ENGEL AG
Citation: M. Milone et al., CONGENITAL MYASTHENIC SYNDROME DUE TO A NULL MUTATION AND AN INFRAME DUPLICATION OF THE ACETYLCHOLINE-RECEPTOR (ACHR) EPSILON-SUBUNIT GENE, American journal of human genetics, 61(4), 1997, pp. 1994-1994
TRANSIENT ELEVATION OF NERVE GROWTH-FACTOR CONTENT IN THE RAT HIPPOCAMPUS AND FRONTAL-CORTEX BY CHRONIC ETHANOL TREATMENT
Authors: NAKANO T FUJIMOTO T SHIMOOKI S FUKUDOME T UCHIDA T TSUJI T MITSUYAMA Y AKIMOTO H FURUKAWA S
Citation: T. Nakano et al., TRANSIENT ELEVATION OF NERVE GROWTH-FACTOR CONTENT IN THE RAT HIPPOCAMPUS AND FRONTAL-CORTEX BY CHRONIC ETHANOL TREATMENT, PSYCHIATRY AND CLINICAL NEUROSCIENCES, 50(3), 1996, pp. 157-160
ADVANCED FLUORITE REGENERATION TECHNOLOGY TO RECOVER SPENT FLUORIDE CHEMICALS DRAINED FROM SEMICONDUCTOR MANUFACTURING PROCESS
Authors: MIKI N MAENO M FUKUDOME T OHMI T
Citation: N. Miki et al., ADVANCED FLUORITE REGENERATION TECHNOLOGY TO RECOVER SPENT FLUORIDE CHEMICALS DRAINED FROM SEMICONDUCTOR MANUFACTURING PROCESS, IEICE transactions on electronics, E79C(3), 1996, pp. 363-374
NOVEL SLOW-CHANNEL SYNDROME DUE TO MUTATION IN THE ACETYLCHOLINE-RECEPTOR (ACHR) ALPHA-SUBUNIT WITH INCREASED CONDUCTANCE, NANOMOLAR AFFINITY FOR ACETYLCHOLINE, AND PROLONGED OPEN DURATIONS OF THE ACHR CHANNEL
Authors: MILONE M OHNO K WANG HL FUKUDOME T PRUITT JN SINE SM ENGEL AG
Citation: M. Milone et al., NOVEL SLOW-CHANNEL SYNDROME DUE TO MUTATION IN THE ACETYLCHOLINE-RECEPTOR (ACHR) ALPHA-SUBUNIT WITH INCREASED CONDUCTANCE, NANOMOLAR AFFINITY FOR ACETYLCHOLINE, AND PROLONGED OPEN DURATIONS OF THE ACHR CHANNEL, Annals of neurology, 40(6), 1996, pp. 9-9
SPLIT TOLERANCE INDUCTION BY INTRATHYMIC INJECTION OF ACETYLCHOLINE-RECEPTOR IN A RAT MODEL OF AUTOIMMUNE MYASTHENIA-GRAVIS - IMPLICATIONS FOR THE DESIGN OF SPECIFIC IMMUNOTHERAPIES
Authors: OHTSURU I MATSUO H FUKUDOME T SUENAGA A TSUJIHATA M NAGATAKI S
Citation: I. Ohtsuru et al., SPLIT TOLERANCE INDUCTION BY INTRATHYMIC INJECTION OF ACETYLCHOLINE-RECEPTOR IN A RAT MODEL OF AUTOIMMUNE MYASTHENIA-GRAVIS - IMPLICATIONS FOR THE DESIGN OF SPECIFIC IMMUNOTHERAPIES, Clinical and experimental immunology, 102(3), 1995, pp. 462-467
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