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Results: 1-14 |
Results: 14

Authors: Mohiddin, S Fananapazir, L
Citation: S. Mohiddin et L. Fananapazir, Advances in understanding hypertrophic cardiomyopathy, HOSP PRACT, 36(5), 2001, pp. 23

Authors: Fananapazir, L Mohiddin, SA Shih, J
Citation: L. Fananapazir et al., Myocardial bridging of the left anterior descending coronary artery in children with hypertrophic cardiomyopathy - Reply, J AM COL C, 38(3), 2001, pp. 922-922

Authors: Begley, D Mohiddin, S Fananapazir, L
Citation: D. Begley et al., Dual chamber pacemaker therapy for mid-cavity obstructive hypertrophic cardiomyopathy, PACE, 24(11), 2001, pp. 1639-1644

Authors: Gollob, MH Green, MS Tang, ASL Gollob, T Karibe, A Roberts, R Ahmad, F Lozado, R Shah, G Fananapazir, L Bachinski, LL Roberts, R Tapscott, T Gonzales, O Begley, D Mohiddin, S
Citation: Mh. Gollob et al., Identification of a gene responsible for familial Wolff-Parkinson-White syndrome, N ENG J MED, 344(24), 2001, pp. 1823-1831

Authors: Karibe, A Tobacman, LS Strand, J Butters, C Back, N Bachinski, LL Arai, AE Ortiz, A Roberts, R Homsher, E Fananapazir, L
Citation: A. Karibe et al., Hypertrophic cardiomyopathy caused by a novel alpha-tropomyosin mutation (V95A) is associated with mild cardiac phenotype, abnormal calcium binding to troponin, abnormal myosin cycling, and poor prognosis, CIRCULATION, 103(1), 2001, pp. 65-71

Authors: Mohiddin, SA Begley, D Shih, J Fananapazir, L
Citation: Sa. Mohiddin et al., Myocardial bridging does not predict sudden death in children with hypertrophic cardiomyopathy but is associated with more severe cardiac disease, J AM COL C, 36(7), 2000, pp. 2270-2278

Authors: Li, ST Tack, CJ Fananapazir, L Goldstein, DS
Citation: St. Li et al., Myocardial perfusion and sympathetic innervation in patients with hypertrophic cardiomyopathy, J AM COL C, 35(7), 2000, pp. 1867-1873

Authors: Palmiter, KA Tyska, MJ Haeberle, JR Alpert, NR Fananapazir, L Warshaw, DM
Citation: Ka. Palmiter et al., R403Q and L908V mutant beta-cardiac myosin from patients with familial hypertrophic cardiomyopathy exhibit enhanced mechanical performance at the single molecule level, J MUSCLE R, 21(7), 2000, pp. 609-620

Authors: Olson, TM Doan, TP Kishimoto, NY Whitby, FG Ackerman, MJ Fananapazir, L
Citation: Tm. Olson et al., Inherited and de novo mutations in the cardiac actin gene cause hypertrophic cardiomyopathy, J MOL CEL C, 32(9), 2000, pp. 1687-1694

Authors: Atiga, WL Fananapazir, L McAreavey, D Calkins, H Berger, RD
Citation: Wl. Atiga et al., Temporal repolarization lability in hypertrophic cardiomyopathy caused by beta-myosin heavy-chain gene mutations, CIRCULATION, 101(11), 2000, pp. 1237-1242

Authors: Fananapazir, L
Citation: L. Fananapazir, Advances in molecular genetics and management of hypertrophic cardiomyopathy (vol 281, pg 1746, 1999), J AM MED A, 282(24), 1999, pp. 2303-2303

Authors: Fananapazir, L
Citation: L. Fananapazir, Hypertrophic cardiomyopathy - Reply, J AM MED A, 282(24), 1999, pp. 2303-2303

Authors: Fananapazir, L
Citation: L. Fananapazir, Advances in molecular genetics and management of hypertrophic cardiomyopathy, J AM MED A, 281(18), 1999, pp. 1746-1752

Authors: Mohiddin, SA Begley, D Fananapazir, L
Citation: Sa. Mohiddin et al., Myocardial bridging in children with hypertrophic cardiomyopathy, N ENG J MED, 341(4), 1999, pp. 288-289
Risultati: 1-14 |