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Results: 1-21 |
Results: 21
BETA-AMYLOID PREFERENTIALLY KILLS NEURONS, BUT DOES NOT DETECTABLY AFFECT TAU PHOSPHORYLATION IN CULTURED BRAIN-CELLS
Authors: REN K GLABE CG BLOOM GS
Citation: K. Ren et al., BETA-AMYLOID PREFERENTIALLY KILLS NEURONS, BUT DOES NOT DETECTABLY AFFECT TAU PHOSPHORYLATION IN CULTURED BRAIN-CELLS, Molecular biology of the cell, 9, 1998, pp. 2288-2288
A SOLID-PHASE, PRION-LIKE PATHWAY FOR AMYLOID A3 PRODUCTION AND ACCUMULATION IN LYSOSOMES
Authors: GLABE CG MARGOL L YANG AJ
Citation: Cg. Glabe et al., A SOLID-PHASE, PRION-LIKE PATHWAY FOR AMYLOID A3 PRODUCTION AND ACCUMULATION IN LYSOSOMES, Molecular biology of the cell, 9, 1998, pp. 2730-2730
LOSS OF ENDOSOMAL LYSOSOMAL MEMBRANE IMPERMEABILITY IS AN EARLY EVENTIN AMYLOID A-BETA-1-42 PATHOGENESIS/
Authors: YANG AJ CHANDSWANGBHUVANA D MARGOL L GLABE CG
Citation: Aj. Yang et al., LOSS OF ENDOSOMAL LYSOSOMAL MEMBRANE IMPERMEABILITY IS AN EARLY EVENTIN AMYLOID A-BETA-1-42 PATHOGENESIS/, Journal of neuroscience research, 52(6), 1998, pp. 691-698
AMYLOID-BETA PROTEIN IS INTERNALIZED SELECTIVELY BY HIPPOCAMPAL FIELDCA1 AND CAUSES NEURONS TO ACCUMULATE AMYLOIDOGENIC CARBOXYTERMINAL FRAGMENTS OF THE AMYLOID PRECURSOR PROTEIN
Authors: BAHR BA HOFFMAN KB YANG AJ HESS US GLABE CG LYNCH G
Citation: Ba. Bahr et al., AMYLOID-BETA PROTEIN IS INTERNALIZED SELECTIVELY BY HIPPOCAMPAL FIELDCA1 AND CAUSES NEURONS TO ACCUMULATE AMYLOIDOGENIC CARBOXYTERMINAL FRAGMENTS OF THE AMYLOID PRECURSOR PROTEIN, Journal of comparative neurology, 397(1), 1998, pp. 139-147
GAMMA-SECRETASE CLEAVAGE IS DISTINCT FROM ENDOPLASMIC-RETICULUM DEGRADATION OF THE TRANSMEMBRANE DOMAIN OF THE AMYLOID PRECURSOR PROTEIN
Authors: BUNNELL WL PHAM HV GLABE CG
Citation: Wl. Bunnell et al., GAMMA-SECRETASE CLEAVAGE IS DISTINCT FROM ENDOPLASMIC-RETICULUM DEGRADATION OF THE TRANSMEMBRANE DOMAIN OF THE AMYLOID PRECURSOR PROTEIN, The Journal of biological chemistry, 273(48), 1998, pp. 31947-31955
MEMBRANE-FUSION IS INDUCED BY A DISTINCT PEPTIDE SEQUENCE OF THE SEA-URCHIN FERTILIZATION PROTEIN BINDIN
Authors: ULRICH AS OTTER M GLABE CG HOEKSTRA D
Citation: As. Ulrich et al., MEMBRANE-FUSION IS INDUCED BY A DISTINCT PEPTIDE SEQUENCE OF THE SEA-URCHIN FERTILIZATION PROTEIN BINDIN, The Journal of biological chemistry, 273(27), 1998, pp. 16748-16755
COMPLEMENT ACTIVATION BY CROSS-LINKED TRUNCATED AND CHIMERIC FULL-LENGTH BETA-AMYLOID
Authors: CRIBBS DH VELAZQUEZ P SOREGHAN B GLABE CG TENNER AJ
Citation: Dh. Cribbs et al., COMPLEMENT ACTIVATION BY CROSS-LINKED TRUNCATED AND CHIMERIC FULL-LENGTH BETA-AMYLOID, NeuroReport, 8(16), 1997, pp. 3457-3462
ACCELERATION OF AMYLOID FIBRIL FORMATION BY SPECIFIC BINDING OF A-BETA-(1-40) PEPTIDE TO GANGLIOSIDE-CONTAINING MEMBRANE-VESICLES
Authors: CHOOSMITH LP GARZONRODRIGUEZ W GLABE CG SUREWICZ WK
Citation: Lp. Choosmith et al., ACCELERATION OF AMYLOID FIBRIL FORMATION BY SPECIFIC BINDING OF A-BETA-(1-40) PEPTIDE TO GANGLIOSIDE-CONTAINING MEMBRANE-VESICLES, The Journal of biological chemistry, 272(37), 1997, pp. 22987-22990
SOLUBLE AMYLOID A-BETA-(1-40) EXISTS AS A STABLE DIMER AT LOW CONCENTRATIONS
Authors: GARZONRODRIGUEZ W SEPULVEDABECERRA M MILTON S GLABE CG
Citation: W. Garzonrodriguez et al., SOLUBLE AMYLOID A-BETA-(1-40) EXISTS AS A STABLE DIMER AT LOW CONCENTRATIONS, The Journal of biological chemistry, 272(34), 1997, pp. 21037-21044
PRESENILIN-1 IMMUNOREACTIVITY IS LOCALIZED INTRACELLULARLY IN ALZHEIMERS-DISEASE BRAIN, BUT NOT DETECTED IN AMYLOID PLAQUES
Authors: WEBER LL LEISSRING MA YANG AJ GLABE CG CRIBBS DH LAFERLA FM
Citation: Ll. Weber et al., PRESENILIN-1 IMMUNOREACTIVITY IS LOCALIZED INTRACELLULARLY IN ALZHEIMERS-DISEASE BRAIN, BUT NOT DETECTED IN AMYLOID PLAQUES, Experimental neurology, 143(1), 1997, pp. 37-44
IDENTIFICATION OF A 97-KDA HEAT-SHOCK-PROTEIN FROM S-FRANCISCANUS OVARIES WITH 94-PERCENT AMINO-ACID IDENTITY TO THE STRONGYLOCENTROTUS-PURPURATUS EGG SURFACE-RECEPTOR FOR SPERM
Authors: MAUK R JAWORSKI D KAMEI N GLABE CG
Citation: R. Mauk et al., IDENTIFICATION OF A 97-KDA HEAT-SHOCK-PROTEIN FROM S-FRANCISCANUS OVARIES WITH 94-PERCENT AMINO-ACID IDENTITY TO THE STRONGYLOCENTROTUS-PURPURATUS EGG SURFACE-RECEPTOR FOR SPERM, Developmental biology, 184(1), 1997, pp. 31-37
PREFERENTIAL ADSORPTION, INTERNALIZATION AND RESISTANCE TO DEGRADATION OF THE MAJOR ISOFORM OF THE ALZHEIMERS AMYLOID PEPTIDE, A-BETA-1-42,IN DIFFERENTIATED PC12 CELLS
Authors: BURDICK D KOSMOSKI J KNAUER MF GLABE CG
Citation: D. Burdick et al., PREFERENTIAL ADSORPTION, INTERNALIZATION AND RESISTANCE TO DEGRADATION OF THE MAJOR ISOFORM OF THE ALZHEIMERS AMYLOID PEPTIDE, A-BETA-1-42,IN DIFFERENTIATED PC12 CELLS, Brain research, 746(1-2), 1997, pp. 275-284
LYSOSOMAL MEMBRANE DAMAGE IS ONE OF EARLIEST MORPHOLOGICAL EVENTS IN BETA-AMYLOID MEDIATED NEUROTOXICITY
Authors: YANG AJ CHANDSWANGBHUVANA D SHU T MARGOL L GLABE CG
Citation: Aj. Yang et al., LYSOSOMAL MEMBRANE DAMAGE IS ONE OF EARLIEST MORPHOLOGICAL EVENTS IN BETA-AMYLOID MEDIATED NEUROTOXICITY, Molecular biology of the cell, 7, 1996, pp. 1909-1909
DEGRADATION AND PROTEOLYTIC PROCESSING OF THE TRANSMEMBRANE DOMAIN OFTHE AMYLOID PRECURSOR PROTEIN
Authors: BUNNELL WL GLABE CG
Citation: Wl. Bunnell et Cg. Glabe, DEGRADATION AND PROTEOLYTIC PROCESSING OF THE TRANSMEMBRANE DOMAIN OFTHE AMYLOID PRECURSOR PROTEIN, Molecular biology of the cell, 7, 1996, pp. 2502-2502
THE SEA-URCHIN SPERM RECEPTOR FOR EGG JELLY IS A MODULAR PROTEIN WITHEXTENSIVE HOMOLOGY TO THE HUMAN POLYCYSTIC KIDNEY-DISEASE PROTEIN, PKD1
Authors: MOY GW MENDOZA LM SCHULZ JR SWANSON WJ GLABE CG VACQUIER VD
Citation: Gw. Moy et al., THE SEA-URCHIN SPERM RECEPTOR FOR EGG JELLY IS A MODULAR PROTEIN WITHEXTENSIVE HOMOLOGY TO THE HUMAN POLYCYSTIC KIDNEY-DISEASE PROTEIN, PKD1, The Journal of cell biology, 133(4), 1996, pp. 809-817
CELL-SURFACE APP751 FORMS COMPLEXES WITH PROTEASE NEXIN-2 LIGANDS ANDIS INTERNALIZED VIA THE LOW-DENSITY-LIPOPROTEIN RECEPTOR-RELATED PROTEIN (LRP)
Authors: KNAUER MF ORLANDO RA GLABE CG
Citation: Mf. Knauer et al., CELL-SURFACE APP751 FORMS COMPLEXES WITH PROTEASE NEXIN-2 LIGANDS ANDIS INTERNALIZED VIA THE LOW-DENSITY-LIPOPROTEIN RECEPTOR-RELATED PROTEIN (LRP), Brain research, 740(1-2), 1996, pp. 6-14
STRUCTURE-ACTIVITY ANALYSES OF BETA-AMYLOID PEPTIDES - CONTRIBUTIONS OF THE BETA-25-35 REGION TO AGGREGATION AND NEUROTOXICITY
Authors: PIKE CJ WALENCEWICZWASSERMAN AJ KOSMOSKI J CRIBBS DH GLABE CG COTMAN CW
Citation: Cj. Pike et al., STRUCTURE-ACTIVITY ANALYSES OF BETA-AMYLOID PEPTIDES - CONTRIBUTIONS OF THE BETA-25-35 REGION TO AGGREGATION AND NEUROTOXICITY, Journal of neurochemistry, 64(1), 1995, pp. 253-265
A SPARSE-MATRIX SCREEN TO ESTABLISH INITIAL CONDITIONS FOR PROTEIN RENATURATION
Authors: HOFMANN A TAI M WONG W GLABE CG
Citation: A. Hofmann et al., A SPARSE-MATRIX SCREEN TO ESTABLISH INITIAL CONDITIONS FOR PROTEIN RENATURATION, Analytical biochemistry, 230(1), 1995, pp. 8-15
BETA-AMYLOID ACTIVATES COMPLEMENT BY BINDING TO A SPECIFIC REGION OF THE COLLAGEN-LIKE DOMAIN OF THE C1Q A CHAIN
Authors: JIANG H BURDICK D GLABE CG COTMAN CW TENNER AJ
Citation: H. Jiang et al., BETA-AMYLOID ACTIVATES COMPLEMENT BY BINDING TO A SPECIFIC REGION OF THE COLLAGEN-LIKE DOMAIN OF THE C1Q A CHAIN, The FASEB journal, 8(5), 1994, pp. 10000768-10000768
BETA-AMYLOID ACTIVATES COMPLEMENT BY BINDING TO A SPECIFIC REGION OF THE COLLAGEN-LIKE DOMAIN OF THE C1Q A-CHAIN
Authors: JIANG HX BURDICK D GLABE CG COTMAN CW TENNER AJ
Citation: Hx. Jiang et al., BETA-AMYLOID ACTIVATES COMPLEMENT BY BINDING TO A SPECIFIC REGION OF THE COLLAGEN-LIKE DOMAIN OF THE C1Q A-CHAIN, The Journal of immunology, 152(10), 1994, pp. 5050-5059
IN-VITRO AND IN-VIVO BINDING OF HUMAN-IMMUNODEFICIENCY-VIRUS TYPE-1 TAT PROTEIN AND SP1 TRANSCRIPTION FACTOR
Authors: JEANG KT CHUN R LIN NH GATIGNOL A GLABE CG FAN H
Citation: Kt. Jeang et al., IN-VITRO AND IN-VIVO BINDING OF HUMAN-IMMUNODEFICIENCY-VIRUS TYPE-1 TAT PROTEIN AND SP1 TRANSCRIPTION FACTOR, Journal of virology, 67(10), 1993, pp. 6224-6233
Risultati: 1-21 |