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Results: 1-21 |
Results: 21
MITOCHONDRIAL RETENTION OF CYTOCHROME-C IN FAS-MEDIATED APOPTOSIS
Authors: ADACHI S GOTTLIEB RA BABIOR BM
Citation: S. Adachi et al., MITOCHONDRIAL RETENTION OF CYTOCHROME-C IN FAS-MEDIATED APOPTOSIS, Journal of leukocyte biology, 1998, pp. 9-9
LACK OF RELEASE OF CYTOCHROME-C FROM MITOCHONDRIA INTO CYTOSOL EARLY IN THE COURSE OF FAS-MEDIATED APOPTOSIS OF JURKAT CELLS
Authors: ADACHI S GOTTLIEB RA BABIOR BM
Citation: S. Adachi et al., LACK OF RELEASE OF CYTOCHROME-C FROM MITOCHONDRIA INTO CYTOSOL EARLY IN THE COURSE OF FAS-MEDIATED APOPTOSIS OF JURKAT CELLS, The Journal of biological chemistry, 273(31), 1998, pp. 19892-19894
EFFECT OF VACUOLAR PROTON ATPASE ON PH(I), CA2+, AND APOPTOSIS IN NEONATAL CARDIOMYOCYTES DURING METABOLIC INHIBITION RECOVERY/
Authors: KARWATOWSKAPROKOPCZUK E NORDBERG JA LI HL ENGLER RL GOTTLIEB RA
Citation: E. Karwatowskaprokopczuk et al., EFFECT OF VACUOLAR PROTON ATPASE ON PH(I), CA2+, AND APOPTOSIS IN NEONATAL CARDIOMYOCYTES DURING METABOLIC INHIBITION RECOVERY/, Circulation research, 82(11), 1998, pp. 1139-1144
BCL-2 AND THE OUTER MITOCHONDRIAL-MEMBRANE IN THE INACTIVATION OF CYTOCHROME-C DURING FAS-MEDIATED APOPTOSIS
Authors: ADACHI S CROSS AR BABIOR BM GOTTLIEB RA
Citation: S. Adachi et al., BCL-2 AND THE OUTER MITOCHONDRIAL-MEMBRANE IN THE INACTIVATION OF CYTOCHROME-C DURING FAS-MEDIATED APOPTOSIS, The Journal of biological chemistry, 272(35), 1997, pp. 21878-21882
GRANULOCYTE-COLONY-STIMULATING FACTOR UP-REGULATES THE VACUOLAR PROTON ATPASE IN HUMAN NEUTROPHILS
Authors: NIESSEN H MEISENHOLDER GW LI HL GLUCK SL LEE BS BOWMAN B ENGLER RL BABIOR BM GOTTLIEB RA
Citation: H. Niessen et al., GRANULOCYTE-COLONY-STIMULATING FACTOR UP-REGULATES THE VACUOLAR PROTON ATPASE IN HUMAN NEUTROPHILS, Blood, 90(11), 1997, pp. 4598-4601
TRANSGLUTAMINATION OF A SINGLE MAJOR 43 KDA PROTEIN IN THE CYTOSKELETON OF APOPTOTIC CELLS FROM THE HUMAN T-LYMPHOBLASTOID LINE, JURKAT
Authors: CALVO KR BABIOR BM GOTTLIEB RA
Citation: Kr. Calvo et al., TRANSGLUTAMINATION OF A SINGLE MAJOR 43 KDA PROTEIN IN THE CYTOSKELETON OF APOPTOTIC CELLS FROM THE HUMAN T-LYMPHOBLASTOID LINE, JURKAT, Journal of investigative medicine, 44(1), 1996, pp. 109-109
ANTI-FAS APO-1 MONOCLONAL-ANTIBODY CH-11 DEPLETES GLUTATHIONE AND KILLS MULTIDRUG-RESISTANT HUMAN LEUKEMIC-CELLS - COMMENT/
Authors: GOTTLIEB RA
Citation: Ra. Gottlieb, ANTI-FAS APO-1 MONOCLONAL-ANTIBODY CH-11 DEPLETES GLUTATHIONE AND KILLS MULTIDRUG-RESISTANT HUMAN LEUKEMIC-CELLS - COMMENT/, Blood cells, molecules, & diseases, 22(1), 1996, pp. 3-4
MUTANT CYSTIC-FIBROSIS TRANSMEMBRANE CONDUCTANCE REGULATOR INHIBITS ACIDIFICATION AND APOPTOSIS IN C127 CELLS - POSSIBLE RELEVANCE TO CYSTIC-FIBROSIS
Authors: GOTTLIEB RA DOSANJH A
Citation: Ra. Gottlieb et A. Dosanjh, MUTANT CYSTIC-FIBROSIS TRANSMEMBRANE CONDUCTANCE REGULATOR INHIBITS ACIDIFICATION AND APOPTOSIS IN C127 CELLS - POSSIBLE RELEVANCE TO CYSTIC-FIBROSIS, Proceedings of the National Academy of Sciences of the United Statesof America, 93(8), 1996, pp. 3587-3591
APOPTOSIS INDUCED IN JURKAT CELLS BY SEVERAL AGENTS IS PRECEDED BY INTRACELLULAR ACIDIFICATION
Authors: GOTTLIEB RA NORDBERG J SKOWRONSKI E BABIOR BM
Citation: Ra. Gottlieb et al., APOPTOSIS INDUCED IN JURKAT CELLS BY SEVERAL AGENTS IS PRECEDED BY INTRACELLULAR ACIDIFICATION, Proceedings of the National Academy of Sciences of the United Statesof America, 93(2), 1996, pp. 654-658
PRECONDITIONING IN RABBIT CARDIOMYOCYTES - ROLE OF PH, VACUOLAR PROTON ATPASE, AND APOPTOSIS
Authors: GOTTLIEB RA GRUOL DL ZHU JY ENGLER RL
Citation: Ra. Gottlieb et al., PRECONDITIONING IN RABBIT CARDIOMYOCYTES - ROLE OF PH, VACUOLAR PROTON ATPASE, AND APOPTOSIS, The Journal of clinical investigation, 97(10), 1996, pp. 2391-2398
LOSS OF FUNCTION OF CYTOCHROME-C IN JURKAT CELLS UNDERGOING FAS-MEDIATED APOPTOSIS
Authors: KRIPPNER A MATSUNOYAGI A GOTTLIEB RA BABIOR BM
Citation: A. Krippner et al., LOSS OF FUNCTION OF CYTOCHROME-C IN JURKAT CELLS UNDERGOING FAS-MEDIATED APOPTOSIS, The Journal of biological chemistry, 271(35), 1996, pp. 21629-21636
EVENTS IN APOPTOSIS - ACIDIFICATION IS DOWNSTREAM OF PROTEASE ACTIVATION AND BCL-2 PROTECTION
Authors: MEISENHOLDER GW MARTIN SJ GREEN DR NORDBERG J BABIOR BM GOTTLIEB RA
Citation: Gw. Meisenholder et al., EVENTS IN APOPTOSIS - ACIDIFICATION IS DOWNSTREAM OF PROTEASE ACTIVATION AND BCL-2 PROTECTION, The Journal of biological chemistry, 271(27), 1996, pp. 16260-16262
G-CSF CAUSES STABILIZATION AND TRANSLOCATION OF THE VACUOLAR PROTON ATPASE IN HUMAN NEUTROPHILS
Authors: NIESSEN H GLUCK SL LEE BS FORGAC M BOWMAN B ENGLER RL BABIOR BM GOTTLIEB RA
Citation: H. Niessen et al., G-CSF CAUSES STABILIZATION AND TRANSLOCATION OF THE VACUOLAR PROTON ATPASE IN HUMAN NEUTROPHILS, Blood, 88(10), 1996, pp. 2601-2601
CELL ACIDIFICATION IN APOPTOSIS - GRANULOCYTE-COLONY-STIMULATING FACTOR DELAYS PROGRAMMED CELL-DEATH IN NEUTROPHILS BY UP-REGULATING THE VACUOLAR H-ATPASE()
Authors: GOTTLIEB RA GIESING HA ZHU JY ENGLER RL BABIOR BM
Citation: Ra. Gottlieb et al., CELL ACIDIFICATION IN APOPTOSIS - GRANULOCYTE-COLONY-STIMULATING FACTOR DELAYS PROGRAMMED CELL-DEATH IN NEUTROPHILS BY UP-REGULATING THE VACUOLAR H-ATPASE(), Proceedings of the National Academy of Sciences of the United Statesof America, 92(13), 1995, pp. 5965-5968
PRECONDITIONING AND CELL-DEATH DURING MYOCYTE METABOLIC INHIBITION RECOVERY - ROLE OF ANION CHANNELS AND VACUOLAR ATPASE
Authors: GOTTLIEB RA ZHU J BABIOR B ENGLER R
Citation: Ra. Gottlieb et al., PRECONDITIONING AND CELL-DEATH DURING MYOCYTE METABOLIC INHIBITION RECOVERY - ROLE OF ANION CHANNELS AND VACUOLAR ATPASE, Circulation, 92(8), 1995, pp. 3711-3711
THE ACID DEOXYRIBONUCLEASE OF NEUTROPHILS - A POSSIBLE PARTICIPANT INAPOPTOSIS-ASSOCIATED GENOME DESTRUCTION
Authors: GOTTLIEB RA GIESING HA ENGLER RL BABIOR BM
Citation: Ra. Gottlieb et al., THE ACID DEOXYRIBONUCLEASE OF NEUTROPHILS - A POSSIBLE PARTICIPANT INAPOPTOSIS-ASSOCIATED GENOME DESTRUCTION, Blood, 86(6), 1995, pp. 2414-2418
ALTERATION OF NHE-1 ACTIVITY IS A FEATURE OF FAS-MEDIATED APOPTOSIS
Authors: GOTTLIEB RA NORDBERG J BABIOR BM
Citation: Ra. Gottlieb et al., ALTERATION OF NHE-1 ACTIVITY IS A FEATURE OF FAS-MEDIATED APOPTOSIS, Blood, 86(10), 1995, pp. 2905-2905
REPERFUSION INJURY INDUCES APOPTOSIS IN RABBIT CARDIOMYOCYTES
Authors: GOTTLIEB RA BURLESON KO KLONER RA BABIOR BM ENGLER RL
Citation: Ra. Gottlieb et al., REPERFUSION INJURY INDUCES APOPTOSIS IN RABBIT CARDIOMYOCYTES, The Journal of clinical investigation, 94(4), 1994, pp. 1621-1628
G-CSF MAY RETARD APOPTOSIS IN NEUTROPHILS BY DELAYING INTRACELLULAR ACIDIFICATION
Authors: GIESING H GOTTLIEB RA ZHU JY ENGLER RL BABIOR BM
Citation: H. Giesing et al., G-CSF MAY RETARD APOPTOSIS IN NEUTROPHILS BY DELAYING INTRACELLULAR ACIDIFICATION, Blood, 84(10), 1994, pp. 10000371-10000371
MYOCYTE CELL-DEATH BY APOPTOSIS DURING REPERFUSION
Authors: ENGLER RL GOTTLIEB RA BURLESON KO BABIOR BM
Citation: Rl. Engler et al., MYOCYTE CELL-DEATH BY APOPTOSIS DURING REPERFUSION, Circulation, 88(4), 1993, pp. 5-5
A DNASE ACTIVITY IS PRESENT IN NEUTROPHILS
Authors: GOTTLIEB RA GIESING H BABIOR BM
Citation: Ra. Gottlieb et al., A DNASE ACTIVITY IS PRESENT IN NEUTROPHILS, Blood, 82(10), 1993, pp. 10000509-10000509
Risultati: 1-21 |