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Results: 1-23 |
Results: 23
ETHANOL INGESTION INCREASES GELATINASE ACTIVITY AND ACUTE EDEMATOUS INJURY IN LUNGS ISOLATED FROM ENDOTOXIN-TREATED RATS
Authors: LOIS M ROMAN J MOSS IM GUIDOT DM
Citation: M. Lois et al., ETHANOL INGESTION INCREASES GELATINASE ACTIVITY AND ACUTE EDEMATOUS INJURY IN LUNGS ISOLATED FROM ENDOTOXIN-TREATED RATS, Journal of investigative medicine, 46(1), 1998, pp. 16-16
ETHANOL INGESTION DECREASES ALVEOLAR TYPE-II CELL MITOCHONDRIAL GLUTATHIONE LEVELS AND IMPAIRS SURFACTANT SECRETION AND VIABILITY IN-VITRO - A PROPOSED MECHANISM BY WHICH CHRONIC-ALCOHOLISM PREDISPOSES TO ACUTE LUNG INJURY
Authors: HOLGUIN F MOSS IM BROWN LS GUIDOT DM
Citation: F. Holguin et al., ETHANOL INGESTION DECREASES ALVEOLAR TYPE-II CELL MITOCHONDRIAL GLUTATHIONE LEVELS AND IMPAIRS SURFACTANT SECRETION AND VIABILITY IN-VITRO - A PROPOSED MECHANISM BY WHICH CHRONIC-ALCOHOLISM PREDISPOSES TO ACUTE LUNG INJURY, Journal of investigative medicine, 46(1), 1998, pp. 17-17
DOES INCREASED MITOCHONDRIAL BIOGENESIS EXPLAIN THE PHENOMENON OF ACQUIRED TOLERANCE TO OXIDATIVE STRESS
Authors: GUIDOT DM
Citation: Dm. Guidot, DOES INCREASED MITOCHONDRIAL BIOGENESIS EXPLAIN THE PHENOMENON OF ACQUIRED TOLERANCE TO OXIDATIVE STRESS, Journal of investigative medicine, 46(1), 1998, pp. 17-17
CHRONIC ETHANOL INGESTION IMPAIRS ALVEOLAR TYPE-II CELL GLUTATHIONE HOMEOSTASIS AND FUNCTION AND PREDISPOSES TO ENDOTOXIN-MEDIATED ACUTE EDEMATOUS LUNG INJURY IN RATS
Authors: HOLGUIN F MOSS IM BROWN LAS GUIDOT DM
Citation: F. Holguin et al., CHRONIC ETHANOL INGESTION IMPAIRS ALVEOLAR TYPE-II CELL GLUTATHIONE HOMEOSTASIS AND FUNCTION AND PREDISPOSES TO ENDOTOXIN-MEDIATED ACUTE EDEMATOUS LUNG INJURY IN RATS, The Journal of clinical investigation, 101(4), 1998, pp. 761-768
ENDOTOXIN PRETREATMENT IN-VIVO INCREASES THE MITOCHONDRIAL RESPIRATORY CAPACITY IN RAT HEPATOCYTES
Authors: GUIDOT DM
Citation: Dm. Guidot, ENDOTOXIN PRETREATMENT IN-VIVO INCREASES THE MITOCHONDRIAL RESPIRATORY CAPACITY IN RAT HEPATOCYTES, Archives of biochemistry and biophysics, 354(1), 1998, pp. 9-17
MODULATING PHOSPHATIDIC-ACID METABOLISM DECREASES OXIDATIVE INJURY INRAT LUNGS
Authors: GUIDOT DM BURSTEN SL RICE GC CHANEY RB SINGER JW REPINE AJ HYBERTSON BM REPINE JE
Citation: Dm. Guidot et al., MODULATING PHOSPHATIDIC-ACID METABOLISM DECREASES OXIDATIVE INJURY INRAT LUNGS, American journal of physiology. Lung cellular and molecular physiology, 17(5), 1997, pp. 957-966
ENDOTOXIN TREATMENT INCREASES LUNG MITOCHONDRIAL SCAVENGING OF EXTRAMITOCHONDRIAL SUPEROXIDE IN HYPEROXIA-EXPOSED RATS
Authors: GUIDOT DM
Citation: Dm. Guidot, ENDOTOXIN TREATMENT INCREASES LUNG MITOCHONDRIAL SCAVENGING OF EXTRAMITOCHONDRIAL SUPEROXIDE IN HYPEROXIA-EXPOSED RATS, Journal of investigative medicine, 44(1), 1996, pp. 47-47
INTERLEUKIN-1 (IL-1) PRETREATMENT PRESERVES LUNG BUT NOT KIDNEY MITOCHONDRIAL SCAVENGING OF SUPEROXIDE ANION (O-2(.-))
Authors: KITOWSKI AD GUIDOT DM REPINE JE
Citation: Ad. Kitowski et al., INTERLEUKIN-1 (IL-1) PRETREATMENT PRESERVES LUNG BUT NOT KIDNEY MITOCHONDRIAL SCAVENGING OF SUPEROXIDE ANION (O-2(.-)), Journal of investigative medicine, 44(1), 1996, pp. 117-117
INHALED NO PREVENTS IL-1-INDUCED NEUTROPHIL ACCUMULATION AND ASSOCIATED ACUTE EDEMA IN ISOLATED RAT LUNGS
Authors: GUIDOT DM HYBERTSON BM KITLOWSKI RP REPINE JE
Citation: Dm. Guidot et al., INHALED NO PREVENTS IL-1-INDUCED NEUTROPHIL ACCUMULATION AND ASSOCIATED ACUTE EDEMA IN ISOLATED RAT LUNGS, American journal of physiology. Lung cellular and molecular physiology, 15(2), 1996, pp. 225-229
ENDOTOXIN TREATMENT INCREASES LUNG MITOCHONDRIAL SCAVENGING OF EXTRAMITOCHONDRIAL SUPEROXIDE IN HYPEROXIA-EXPOSED RATS
Authors: GUIDOT DM
Citation: Dm. Guidot, ENDOTOXIN TREATMENT INCREASES LUNG MITOCHONDRIAL SCAVENGING OF EXTRAMITOCHONDRIAL SUPEROXIDE IN HYPEROXIA-EXPOSED RATS, Archives of biochemistry and biophysics, 326(2), 1996, pp. 266-270
IN-VITRO SELECTION OF RNA-BASED IRREVERSIBLE INHIBITORS OF HUMAN NEUTROPHIL ELASTASE
Authors: SMITH D KIRSCHENHEUTER GP CHARLTON J GUIDOT DM REPINE JE
Citation: D. Smith et al., IN-VITRO SELECTION OF RNA-BASED IRREVERSIBLE INHIBITORS OF HUMAN NEUTROPHIL ELASTASE, Chemistry & biology, 2(11), 1995, pp. 741-750
INHALED NITRIC-OXIDE PREVENTS NEUTROPHIL-MEDIATED, OXYGEN RADICAL-DEPENDENT LEAK IN ISOLATED RAT LUNGS
Authors: GUIDOT DM REPINE MJ HYBERTSON BM REPINE JE
Citation: Dm. Guidot et al., INHALED NITRIC-OXIDE PREVENTS NEUTROPHIL-MEDIATED, OXYGEN RADICAL-DEPENDENT LEAK IN ISOLATED RAT LUNGS, American journal of physiology. Lung cellular and molecular physiology, 13(1), 1995, pp. 2-5
PHOSPHATIDIC-ACID SIGNALING MEDIATES LUNG CYTOKINE EXPRESSION AND LUNG INFLAMMATORY INJURY AFTER HEMORRHAGE IN MICE
Authors: ABRAHAM E BURSTEN S SHENKAR R ALLBEE J TUDER R WOODSON P GUIDOT DM RICE G SINGER JW REPINE JE
Citation: E. Abraham et al., PHOSPHATIDIC-ACID SIGNALING MEDIATES LUNG CYTOKINE EXPRESSION AND LUNG INFLAMMATORY INJURY AFTER HEMORRHAGE IN MICE, The Journal of experimental medicine, 181(2), 1995, pp. 569-575
MITOCHONDRIAL RESPIRATION SCAVENGES EXTRAMITOCHONDRIAL SUPEROXIDE ANION VIA A NONENZYMATIC MECHANISM
Authors: GUIDOT DM REPINE JE KITLOWSKI AD FLORES SC NELSON SK WRIGHT RM MCCORD JM
Citation: Dm. Guidot et al., MITOCHONDRIAL RESPIRATION SCAVENGES EXTRAMITOCHONDRIAL SUPEROXIDE ANION VIA A NONENZYMATIC MECHANISM, The Journal of clinical investigation, 96(2), 1995, pp. 1131-1136
INTERLEUKIN-1 TREATMENT INCREASES NEUTROPHILS BUT NOT ANTIOXIDANT ENZYME-ACTIVITY OR RESISTANCE TO ISCHEMIA-REPERFUSION INJURY IN RAT KIDNEYS
Authors: GUIDOT DM LINAS SL REPINE MJ SHANLEY PF FISHER HS REPINE JE
Citation: Dm. Guidot et al., INTERLEUKIN-1 TREATMENT INCREASES NEUTROPHILS BUT NOT ANTIOXIDANT ENZYME-ACTIVITY OR RESISTANCE TO ISCHEMIA-REPERFUSION INJURY IN RAT KIDNEYS, Inflammation, 18(5), 1994, pp. 537-545
INTRINSIC 5-LIPOXYGENASE ACTIVITY IS REQUIRED FOR NEUTROPHIL RESPONSIVITY
Authors: GUIDOT DM REPINE MJ WESTCOTT JY REPINE JE
Citation: Dm. Guidot et al., INTRINSIC 5-LIPOXYGENASE ACTIVITY IS REQUIRED FOR NEUTROPHIL RESPONSIVITY, Proceedings of the National Academy of Sciences of the United Statesof America, 91(17), 1994, pp. 8156-8159
INTRATRACHEAL BUT NOT INTRAVASCULAR INTERLEUKIN-1 CAUSES ACUTE EDEMATOUS INJURY IN ISOLATED NEUTROPHIL-PERFUSED RAT LUNGS THROUGH AN OXYGENRADICAL-MEDIATED MECHANISM
Authors: GUIDOT DM STEVENS EE REPINE MJ LUCCABROCO AE REPINE JE
Citation: Dm. Guidot et al., INTRATRACHEAL BUT NOT INTRAVASCULAR INTERLEUKIN-1 CAUSES ACUTE EDEMATOUS INJURY IN ISOLATED NEUTROPHIL-PERFUSED RAT LUNGS THROUGH AN OXYGENRADICAL-MEDIATED MECHANISM, The Journal of laboratory and clinical medicine, 123(4), 1994, pp. 605-609
A DUAL ROLE FOR ELECTRON-TRANSPORT IN CELLULAR OXIDANT-ANTIOXIDANT BALANCE - MITOCHONDRIAL RESPIRATION PRODUCES SUPEROXIDE IN HYPEROXIA BUTSCAVENGES EXTRAMITOCHONDRIAL SUPEROXIDE IN NORMOXIA
Authors: GUIDOT DM MCCORD JM REPINE JE
Citation: Dm. Guidot et al., A DUAL ROLE FOR ELECTRON-TRANSPORT IN CELLULAR OXIDANT-ANTIOXIDANT BALANCE - MITOCHONDRIAL RESPIRATION PRODUCES SUPEROXIDE IN HYPEROXIA BUTSCAVENGES EXTRAMITOCHONDRIAL SUPEROXIDE IN NORMOXIA, Clinical research, 42(2), 1994, pp. 10000145-10000145
INTRINSIC NEUTROPHIL NADPH OXIDASE AND 5-LIPOXYGENASE ACTIVITY ARE BOTH REQUIRED FOR INTERLEUKIN-8-MEDIATED INJURY IN ISOLATED-PERFUSED RATLUNGS
Authors: GUIDOT DM REPINE MJ WESTCOTT JY REPINE JE
Citation: Dm. Guidot et al., INTRINSIC NEUTROPHIL NADPH OXIDASE AND 5-LIPOXYGENASE ACTIVITY ARE BOTH REQUIRED FOR INTERLEUKIN-8-MEDIATED INJURY IN ISOLATED-PERFUSED RATLUNGS, Clinical research, 42(2), 1994, pp. 10000145-10000145
INHIBITION OF LYSOPHOSPHATIDIC ACID ACYL TRANSFERASE (LPAAT) ACTIVITYBY CT1501R SUPPRESSES NEUTROPHIL ADHERENCE AND CHEMOTAXIS IN-VITRO AND DECREASES INTERLEUKIN-8 (IL-8)-INDUCED INJURY IN ISOLATED RAT LUNGS PERFUSED WITH HUMAN NEUTROPHILS
Authors: GUIDOT DM RICE GC SINGER JW BURSTEN S BIANCO JA ABRAHAM E REPINE JE
Citation: Dm. Guidot et al., INHIBITION OF LYSOPHOSPHATIDIC ACID ACYL TRANSFERASE (LPAAT) ACTIVITYBY CT1501R SUPPRESSES NEUTROPHIL ADHERENCE AND CHEMOTAXIS IN-VITRO AND DECREASES INTERLEUKIN-8 (IL-8)-INDUCED INJURY IN ISOLATED RAT LUNGS PERFUSED WITH HUMAN NEUTROPHILS, Clinical research, 42(2), 1994, pp. 10000145-10000145
NEUTROPHIL-DERIVED NADPH OXIDASE AND LIPOXYGENASE PRODUCTS MEDIATE DAMAGE IN ISOLATED NEUTROPHIL-PERFUSED RAT LUNGS GIVEN INTERLEUKIN-8 (IL-8) INTRATRACHEALLY
Authors: REPINE MJ GUIDOT DM REPINE JE
Citation: Mj. Repine et al., NEUTROPHIL-DERIVED NADPH OXIDASE AND LIPOXYGENASE PRODUCTS MEDIATE DAMAGE IN ISOLATED NEUTROPHIL-PERFUSED RAT LUNGS GIVEN INTERLEUKIN-8 (IL-8) INTRATRACHEALLY, Clinical research, 42(1), 1994, pp. 10000005-10000005
MITOCHONDRIAL RESPIRATION PRODUCES SUPEROXIDE IN HYPEROXIA BUT SCAVENGES EXTRAMITOCHONDRIAL SUPEROXIDE IN NORMOXIA - A DUAL ROLE FOR ELECTRON-TRANSPORT IN CELLULAR OXIDANT-ANTIOXIDANT BALANCE
Authors: GUIDOT DM REPINE JE MCCORD JM
Citation: Dm. Guidot et al., MITOCHONDRIAL RESPIRATION PRODUCES SUPEROXIDE IN HYPEROXIA BUT SCAVENGES EXTRAMITOCHONDRIAL SUPEROXIDE IN NORMOXIA - A DUAL ROLE FOR ELECTRON-TRANSPORT IN CELLULAR OXIDANT-ANTIOXIDANT BALANCE, Free radical biology & medicine, 15(5), 1993, pp. 540-540
ABSENCE OF ELECTRON-TRANSPORT (RHO(0) STATE) RESTORES GROWTH OF A MANGANESE-SUPEROXIDE DISMUTASE-DEFICIENT SACCHAROMYCES-CEREVISIAE IN HYPEROXIA - EVIDENCE FOR ELECTRON-TRANSPORT AS A MAJOR SOURCE OF SUPEROXIDE GENERATION IN-VIVO
Authors: GUIDOT DM MCCORD JM WRIGHT RM REPINE JE
Citation: Dm. Guidot et al., ABSENCE OF ELECTRON-TRANSPORT (RHO(0) STATE) RESTORES GROWTH OF A MANGANESE-SUPEROXIDE DISMUTASE-DEFICIENT SACCHAROMYCES-CEREVISIAE IN HYPEROXIA - EVIDENCE FOR ELECTRON-TRANSPORT AS A MAJOR SOURCE OF SUPEROXIDE GENERATION IN-VIVO, The Journal of biological chemistry, 268(35), 1993, pp. 26699-26703
Risultati: 1-23 |