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Results: 1-8 |
Results: 8
Members of the Kv1 and Kv2 voltage-dependent K+ channel families regulate insulin secretion
Authors: MacDonald, PE Ha, XF Wang, J Smukler, SR Sun, AM Gaisano, HY Salapatek, AMF Backx, PH Wheeler, MB
Citation: Pe. Macdonald et al., Members of the Kv1 and Kv2 voltage-dependent K+ channel families regulate insulin secretion, MOL ENDOCR, 15(8), 2001, pp. 1423-1435
Cholecystokinin-regulated exocytosis in rat pancreatic acinar cells is inhibited by a C-terminus truncated mutant of SNAP-23
Authors: Huang, XH Sheu, L Tamori, Y Trimble, WS Gaisano, HY
Citation: Xh. Huang et al., Cholecystokinin-regulated exocytosis in rat pancreatic acinar cells is inhibited by a C-terminus truncated mutant of SNAP-23, PANCREAS, 23(2), 2001, pp. 125-133
Ca2+ influx and cAMP elevation overcame botulinum toxin A but not tetanus toxin inhibition of insulin exocytosis
Authors: Huang, XH Kang, YH Pasyk, EA Sheu, L Wheeler, MB Trimble, WS Salapatek, A Gaisano, HY
Citation: Xh. Huang et al., Ca2+ influx and cAMP elevation overcame botulinum toxin A but not tetanus toxin inhibition of insulin exocytosis, AM J P-CELL, 281(3), 2001, pp. C740-C750
Supramaximal cholecystokinin displaces Munc18c from the pancreatic acinar basal surface, redirecting apical exocytosis to the basal membrane
Authors: Gaisano, HY Lutz, MP Leser, J Sheu, L Lynch, G Tang, L Tamori, Y Trimble, WS Salapatek, AMF
Citation: Hy. Gaisano et al., Supramaximal cholecystokinin displaces Munc18c from the pancreatic acinar basal surface, redirecting apical exocytosis to the basal membrane, J CLIN INV, 108(11), 2001, pp. 1597-1611
A hypothesis: SNARE-ing the mechanisms of regulated exocytosis and pathologic membrane fusions in the pancreatic acinar cell
Authors: Gaisano, HY
Citation: Hy. Gaisano, A hypothesis: SNARE-ing the mechanisms of regulated exocytosis and pathologic membrane fusions in the pancreatic acinar cell, PANCREAS, 20(3), 2000, pp. 217-226
Mutations to the third cytoplasmic domain of the glucagon-like peptide 1 (GLP-1) receptor can functionally uncouple GLP-1-stimulated insulin secretion in HIT-T15 cells
Authors: Salapatek, AMF MacDonald, PE Gaisano, HY Wheeler, MB
Citation: Amf. Salapatek et al., Mutations to the third cytoplasmic domain of the glucagon-like peptide 1 (GLP-1) receptor can functionally uncouple GLP-1-stimulated insulin secretion in HIT-T15 cells, MOL ENDOCR, 13(8), 1999, pp. 1305-1317
beta-cell hypertrophy in falfa rats is associated with basal glucose hypersensitivity and reduced SNARE protein expression
Authors: Chan, CB MacPhail, RM Sheu, L Wheeler, MB Gaisano, HY
Citation: Cb. Chan et al., beta-cell hypertrophy in falfa rats is associated with basal glucose hypersensitivity and reduced SNARE protein expression, DIABETES, 48(5), 1999, pp. 997-1005
Snare protein expression and adenoviral transfection of amphicrine AR42J
Authors: Gaisano, HY Huang, XH Sheu, L Ghai, M Newgard, CB Trinh, KY Trimble, WS
Citation: Hy. Gaisano et al., Snare protein expression and adenoviral transfection of amphicrine AR42J, BIOC BIOP R, 260(3), 1999, pp. 781-784
Risultati: 1-8 |