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Citation: J. Frantsve et al., Socs-1 inhibits TEL-JAK2-mediated transformation of hematopoietic cells through inhibition of JAK2 kinase activity and induction of proteasome-mediated degradation, MOL CELL B, 21(10), 2001, pp. 3547-3557
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Citation: P. Andreasson et al., The expression of ETV6/CBFA2 (TEL/AML1) is not sufficient for the transformation of hematopoietic cell lines in vitro or the induction of hematologicdisease in vivo, CANC GENET, 130(2), 2001, pp. 93-104
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Sternberg, DW
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Citation: Dw. Sternberg et al., The TEL/PDGF beta R fusion in chronic myelomonocytic leukemia signals through STAT5-dependent and STAT5-independent pathways, BLOOD, 98(12), 2001, pp. 3390-3397
Authors:
Tomasson, MH
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Citation: Mh. Tomasson et al., Induction of myeloproliferative disease in mice by tyrosine kinase fusion oncogenes does not require granulocyte-macrophage colony-stimulating factoror interleukin-3, BLOOD, 97(5), 2001, pp. 1435-1441
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Li, SG
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Citation: Sg. Li et al., Interleukin 3 and granulocyte-macrophage colony-stimulating factor are notrequired for induction of chronic myeloid leukemia-like myeloproliferativedisease in mice by BCR/ABL, BLOOD, 97(5), 2001, pp. 1442-1450
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Gilliland, DG
Citation: J. Schwaller et al., H4(D10S170), a gene frequently rearranged in papillary thyroid carcinoma, is fused to the platelet-derived growth factor receptor beta gene in atypical chronic myeloid leukemia with t(5;10)(q33;q22), BLOOD, 97(12), 2001, pp. 3910-3918
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Schwaller, J
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Williams, IR
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Citation: J. Schwaller et al., Stat5 is essential for the myelo- and lymphoproliferative disease induced by TEL/JAK2, MOL CELL, 6(3), 2000, pp. 693-704
Citation: De. Levy et Dg. Gilliland, Divergent roles of STAT1 and STAT5 in malignancy as revealed by gene disruptions in mice, ONCOGENE, 19(21), 2000, pp. 2505-2510
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Wilbanks, AM
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Citation: Am. Wilbanks et al., TEL/PDGF beta R fusion protein activates STAT1 and STAT5: A common mechanism for transformation by tyrosine kinase fusion proteins, EXP HEMATOL, 28(5), 2000, pp. 584-593
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Citation: Q. Liu et al., Signal transduction and transforming properties of the TEL-TRKC fusions associated with t(12;15)(p13;q25) in congenital fibrosarcoma and acute myelogenous leukemia, EMBO J, 19(8), 2000, pp. 1827-1838
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Dalton, V
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Gilliland, DG
Citation: Ml. Loh et al., Translocation (2;8)(p12;q24) associated with a cryptic t(12;21)(p13;q22) TEL/AML1 gene rearrangement in a child with acute lymphoblastic leukemia, CANC GENET, 122(2), 2000, pp. 79-82
Authors:
Tomasson, MH
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Gilliland, DG
Citation: Mh. Tomasson et al., Fatal myeloproliferation, induced in mice by TEL/PDGF beta R expression, depends on PDGF beta R tyrosines 579/581, J CLIN INV, 105(4), 2000, pp. 423-432
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Gilliland, DG
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Nelson, RA
Rose, EH
Dugan, MH
Citation: Ja. Thompson et al., Effect of recombinant human erythropoietin combined with granulocyte/macrophage colony-stimulating factor in the treatment of patients with myelodysplastic syndrome, BLOOD, 95(4), 2000, pp. 1175-1179
Authors:
Song, WJ
Sullivan, MG
Legare, RD
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Tan, XL
Kufrin, D
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Resende, IC
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Roy, DC
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Kurnit, D
Willman, C
Gewirtz, AM
Speck, NA
Bushweller, JH
Li, FP
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Gilliland, DG
Citation: Wj. Song et al., Haploinsufficiency of CBFA2 causes familial thrombocytopenia with propensity to develop acute myelogenous leukaemia, NAT GENET, 23(2), 1999, pp. 166-175
Citation: Ts. Ross et Dg. Gilliland, Transforming properties of the Huntingtin interacting protein 1/platelet-derived growth factor beta receptor fusion protein, J BIOL CHEM, 274(32), 1999, pp. 22328-22336
Authors:
Tomasson, MH
Williams, IR
Hasserjian, R
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McGrath, SM
Schwaller, J
Druker, B
Gilliland, DG
Citation: Mh. Tomasson et al., TEL/PDGF beta R induces hematologic malignancies in mice that respond to aspecific tyrosine kinase inhibitor, BLOOD, 93(5), 1999, pp. 1707-1714