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Results: 1-3 |
Results: 3
The NH2-terminal coiled-coil domain and tyrosine 177 play important roles in induction of a myeloproliferative disease in mice by Bcr-Abl
Authors: Zhang, ZW Subrahmanyam, R Wong, R Gross, AW Ren, RB
Citation: Zw. Zhang et al., The NH2-terminal coiled-coil domain and tyrosine 177 play important roles in induction of a myeloproliferative disease in mice by Bcr-Abl, MOL CELL B, 21(3), 2001, pp. 840-853
Bcr-Abl has a greater intrinsic capacity than v-Abl to induce the neoplastic expansion of myeloid cells
Authors: Gross, AW Ren, RB
Citation: Aw. Gross et Rb. Ren, Bcr-Abl has a greater intrinsic capacity than v-Abl to induce the neoplastic expansion of myeloid cells, ONCOGENE, 19(54), 2000, pp. 6286-6296
Bcr-Abl with an SH3 deletion retains the ability to induce a myeloproliferative disease in mice, yet c-Abl activated by an SH3 deletion induces only lymphoid malignancy
Authors: Gross, AW Zhang, XW Ren, RB
Citation: Aw. Gross et al., Bcr-Abl with an SH3 deletion retains the ability to induce a myeloproliferative disease in mice, yet c-Abl activated by an SH3 deletion induces only lymphoid malignancy, MOL CELL B, 19(10), 1999, pp. 6918-6928
Risultati: 1-3 |