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Results: 1-15 |
Results: 15

Authors: Hodgetts, SI Grounds, MD
Citation: Si. Hodgetts et Md. Grounds, Complement and myoblast transfer therapy: Donor myoblast survival is enhanced following depletion of host complement C3 using cobra venom factor, butnot in the absence of C5, IMM CELL B, 79(3), 2001, pp. 231-239

Authors: White, JD Davies, M Grounds, MD
Citation: Jd. White et al., Leukaemia inhibitory factor increases myoblast replication and survival and affects extracellular matrix production: combined in vivo and in vitro studies in post-natal skeletal muscle, CELL TIS RE, 306(1), 2001, pp. 129-141

Authors: Smythe, GM Davies, MJ Paulin, D Grounds, MD
Citation: Gm. Smythe et al., Absence of desmin slightly prolongs myoblast proliferation and delays fusion in vivo in regenerating grafts of skeletal muscle, CELL TIS RE, 304(2), 2001, pp. 287-294

Authors: Huijbregts, J White, JD Grounds, MD
Citation: J. Huijbregts et al., The absence of MyoD in regenerating skeletal muscle affects the expressionpattern of basement membrane, interstitial matrix and integrin molecules that is consistent with delayed myotube formation, ACT HISTOCH, 103(4), 2001, pp. 379-396

Authors: Collins, RA Grounds, MD
Citation: Ra. Collins et Md. Grounds, The role of tumor necrosis factor-alpha (TNF-alpha) in skeletal muscle regeneration: Studies in TNF-alpha(-/-) and TNF-alpha(-/-)/LT-alpha(-/-) mice, J HIST CYTO, 49(8), 2001, pp. 989-1001

Authors: Smythe, GM Grounds, MD
Citation: Gm. Smythe et Md. Grounds, Absence of MyoD increases donor myoblast migration into host muscle, EXP CELL RE, 267(2), 2001, pp. 267-274

Authors: Smythe, GM Hodgetts, SI Grounds, MD
Citation: Gm. Smythe et al., Immunobiology and the future of myoblast transfer therapy, MOL THER, 1(4), 2000, pp. 304-313

Authors: Grounds, MD
Citation: Md. Grounds, Myoblast transfer therapy in the new millennium, CELL TRANSP, 9(4), 2000, pp. 485-487

Authors: Hodgetts, SI Beilharz, MW Scalzo, AA Grounds, MD
Citation: Si. Hodgetts et al., Why do cultured transplanted myoblasts die in vivo? DNA quantification shows enhanced survival of donor male myoblasts in host mice depleted of CD4(+) and CD8(+) cells or NK1.1(+) cells, CELL TRANSP, 9(4), 2000, pp. 489-502

Authors: Smythe, GM Grounds, MD
Citation: Gm. Smythe et Md. Grounds, Exposure to tissue culture conditions can adversely affect myoblast behavior in vivo in whole muscle grafts: Implications for myoblast transfer therapy, CELL TRANSP, 9(3), 2000, pp. 379-393

Authors: Smythe, GM Fan, Y Grounds, MD
Citation: Gm. Smythe et al., Enhanced migration and fusion of donor myoblasts in dystrophic and normal host muscle, MUSCLE NERV, 23(4), 2000, pp. 560-574

Authors: White, JD Scaffidi, A Davies, M McGeachie, J Rudnicki, MA Grounds, MD
Citation: Jd. White et al., Myotube formation is delayed but not prevented in MyoD-deficient skeletal muscle: Studies in regenerating whole muscle grafts of adult mice, J HIST CYTO, 48(11), 2000, pp. 1531-1543

Authors: Sorokin, LM Maley, MAL Moch, H von der Mark, H von der Mark, K Cadalbert, L Karosi, S Davies, MJ McGeachie, JK Grounds, MD
Citation: Lm. Sorokin et al., Laminin alpha 4 and integrin alpha 6 are upregulated in regenerating dy/dyskeletal muscle: Comparative expression of laminin and integrin isoforms in muscles regenerating after crush injury, EXP CELL RE, 256(2), 2000, pp. 500-514

Authors: Grounds, MD
Citation: Md. Grounds, Muscle regeneration: molecular aspects and therapeutic implications, CURR OP NEU, 12(5), 1999, pp. 535-543

Authors: McGeachie, JK Grounds, MD
Citation: Jk. Mcgeachie et Md. Grounds, The timing between skeletal muscle myoblast replication and fusion into myotubes, and the stability of regenerated dystrophic myofibres: an autoradiographic study in mdx mice, J ANAT, 194, 1999, pp. 287-295
Risultati: 1-15 |