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Results: 1-25 | 26-38
Results: 1-25/38
SYNGENEIC BONE-MARROW TRANSPLANTATION IN A PATIENT WITH PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA
Authors: PRATT G DAVIES FE RICHARDS S HILLMEN P MORGAN GJ SMITH GM
Citation: G. Pratt et al., SYNGENEIC BONE-MARROW TRANSPLANTATION IN A PATIENT WITH PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA, Bone marrow transplantation, 21, 1998, pp. 160-160
SOMATIC MUTATION AND CLONAL SELECTION IN THE PATHOGENESIS AND IN THE CONTROL OF PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA
Authors: BESSLER M HILLMEN P
Citation: M. Bessler et P. Hillmen, SOMATIC MUTATION AND CLONAL SELECTION IN THE PATHOGENESIS AND IN THE CONTROL OF PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA, Seminars in hematology, 35(2), 1998, pp. 149-167
CAMPATH1H THERAPY FOR PATIENTS WITH REFRACTORY CHRONIC LYMPHOCYTIC-LEUKEMIA (CLL)
Authors: RAWSTRON AC DAVIES FE EVANS PA JAMIL A GALVIN MC HAYNES AP HALE G MORGAN GJ HILLMEN P
Citation: Ac. Rawstron et al., CAMPATH1H THERAPY FOR PATIENTS WITH REFRACTORY CHRONIC LYMPHOCYTIC-LEUKEMIA (CLL), British Journal of Haematology, 101, 1998, pp. 97-97
SYNGENEIC NONE MARROW TRANSPLANTATION IN PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA (PNH) - ERADICATION OF THE PNH CLONE AS DOCUMENTED BY FLOW-CYTOMETRY
Authors: PRATT G DAVIES FE RICHARDS S HILLMEN P MORGAN GJ SMITH GM
Citation: G. Pratt et al., SYNGENEIC NONE MARROW TRANSPLANTATION IN PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA (PNH) - ERADICATION OF THE PNH CLONE AS DOCUMENTED BY FLOW-CYTOMETRY, British Journal of Haematology, 101, 1998, pp. 303-303
GPI-DEFICIENT CELLS (PNH PHENOTYPE) ARE PRESENT IN MOST INDIVIDUALS
Authors: RAWSTRON AC ROLLINSON SJ WRIGHT D HALE G SHORT MA RICHARDS S MORGAN GJ HILLMEN P
Citation: Ac. Rawstron et al., GPI-DEFICIENT CELLS (PNH PHENOTYPE) ARE PRESENT IN MOST INDIVIDUALS, British Journal of Haematology, 101, 1998, pp. 320-320
LYMPHOCYTE SUBSET ANALYSIS AND GLYCOSYLPHOSPHATIDYLINOSITOL PHENOTYPEIN PATIENTS WITH PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA
Authors: RICHARDS SJ NORFOLK DR SWIRSKY DM HILLMEN P
Citation: Sj. Richards et al., LYMPHOCYTE SUBSET ANALYSIS AND GLYCOSYLPHOSPHATIDYLINOSITOL PHENOTYPEIN PATIENTS WITH PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA, Blood, 92(5), 1998, pp. 1799-1806
CIRCULATING PRIMITIVE STEM-CELLS IN PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA (PNH) ARE PREDOMINANTLY NORMAL IN PHENOTYPE BUT GRANULOCYTE-COLONY-STIMULATING FACTOR TREATMENT MOBILIZES MAINLY PNH STEM-CELLS
Authors: JOHNSON RJ RAWSTRON AC RICHARDS S MORGAN GJ NORFOLK DR OCONNOR S HILLMEN P
Citation: Rj. Johnson et al., CIRCULATING PRIMITIVE STEM-CELLS IN PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA (PNH) ARE PREDOMINANTLY NORMAL IN PHENOTYPE BUT GRANULOCYTE-COLONY-STIMULATING FACTOR TREATMENT MOBILIZES MAINLY PNH STEM-CELLS, Blood, 91(12), 1998, pp. 4504-4508
ABSENCE OF GPI-ANCHORED PROTEINS FROM PNH LYMPHOCYTES HINDERS LYMPHOCYTE-ACTIVATION AND MEMORY CELL-FORMATION IN-VIVO
Authors: RICHARDS SJ NORFOLK DR HILLMEN P
Citation: Sj. Richards et al., ABSENCE OF GPI-ANCHORED PROTEINS FROM PNH LYMPHOCYTES HINDERS LYMPHOCYTE-ACTIVATION AND MEMORY CELL-FORMATION IN-VIVO, Blood, 90(10), 1997, pp. 16-16
GPI-DEFICIENT CELLS (PNH PHENOTYPE) ARE PRESENT IN MOST NORMAL INDIVIDUALS
Authors: RAWSTRON AC ROLLINSON SJ RICHARDS S SHORT MA MORGAN GJ WRIGHT D HALE G HILLMEN P
Citation: Ac. Rawstron et al., GPI-DEFICIENT CELLS (PNH PHENOTYPE) ARE PRESENT IN MOST NORMAL INDIVIDUALS, Blood, 90(10), 1997, pp. 1202-1202
MINIMAL RESIDUAL DISEASE DETECTION USING A FLUORESCENT CONSENSUS IGH PCR AT WEEK-20 OF THERAPY IS STRONGLY PREDICTIVE OF RELAPSE IN CHILDREN WITH PRECURSOR B-ALL
Authors: EVANS PAS FORSYTH PD SHORT MA OWEN RG SHIACH CR KINSEY S DAVIES FE JACK AS HILLMEN P MORGAN GJ
Citation: Pas. Evans et al., MINIMAL RESIDUAL DISEASE DETECTION USING A FLUORESCENT CONSENSUS IGH PCR AT WEEK-20 OF THERAPY IS STRONGLY PREDICTIVE OF RELAPSE IN CHILDREN WITH PRECURSOR B-ALL, Blood, 90(10), 1997, pp. 1876-1876
CD56 BUT NOT SYNDECAN-1 EXPRESSION CORRELATES WITH DEGREE OF MARROW INFILTRATION AND LEVEL OF CIRCULATING PLASMA-CELLS
Authors: RAWSTRON AC BARRANS SL BLYTHE D DAVIES FE ENGLISH A PRATT G CHILD JA MORGAN GJ JACK AS HILLMEN P
Citation: Ac. Rawstron et al., CD56 BUT NOT SYNDECAN-1 EXPRESSION CORRELATES WITH DEGREE OF MARROW INFILTRATION AND LEVEL OF CIRCULATING PLASMA-CELLS, Blood, 90(10), 1997, pp. 2354-2354
CAMPATH1H THERAPY FOR PATIENTS WITH REFRACTORY CHRONIC LYMPHOCYTIC-LEUKEMIA (CLL)
Authors: RAWSTRON AC DAVIES FE EVANS P MORGAN GJ JAMIL A GALVIN MC HAYNES AP HALE G HILLMEN P
Citation: Ac. Rawstron et al., CAMPATH1H THERAPY FOR PATIENTS WITH REFRACTORY CHRONIC LYMPHOCYTIC-LEUKEMIA (CLL), Blood, 90(10), 1997, pp. 2356-2356
BOTH PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA (PNH) TYPE-II CELLS AND PNH TYPE-III CELLS CAN ARISE FROM DIFFERENT POINT MUTATIONS INVOLVING THE SAME CODON OF THE PIG-A GENE
Authors: ROLLINSON S RICHARDS S NORFOLK D BIBI K MORGAN G HILLMEN P
Citation: S. Rollinson et al., BOTH PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA (PNH) TYPE-II CELLS AND PNH TYPE-III CELLS CAN ARISE FROM DIFFERENT POINT MUTATIONS INVOLVING THE SAME CODON OF THE PIG-A GENE, Blood, 89(8), 1997, pp. 3069-3071
FACTOR-V LEIDEN MUTATION INVESTIGATED BY AMPLIFICATION CREATED RESTRICTION ENZYME SITE (ACRES) IN PNH PATIENTS WITH AND WITHOUT THROMBOSIS
Authors: NAFA K BESSLER M MASON P VULLIAMY T HILLMEN P CASTROMALASPINA H LUZZATTO L
Citation: K. Nafa et al., FACTOR-V LEIDEN MUTATION INVESTIGATED BY AMPLIFICATION CREATED RESTRICTION ENZYME SITE (ACRES) IN PNH PATIENTS WITH AND WITHOUT THROMBOSIS, Haematologica, 81(6), 1996, pp. 540-542
THERAPY WITH RECOMBINANT ERYTHROPOIETIN IN PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA
Authors: MCMULLIN MF HILLMEN P ELDER GE LAPPIN TRJ LUZZATTO L
Citation: Mf. Mcmullin et al., THERAPY WITH RECOMBINANT ERYTHROPOIETIN IN PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA, British Journal of Haematology, 94(2), 1996, pp. 424-424
MOBILIZATION OF PBSCS FOR EX-VIVO MANIPULATION WITH A VIEW TO GENE-THERAPY FOR PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA (PNH)
Authors: JOHNSON RJ HILLMEN P OWEN RG NORFOLK DR SMITH GM OCONNOR SJM RAWSTRON AC MORGAN GJ
Citation: Rj. Johnson et al., MOBILIZATION OF PBSCS FOR EX-VIVO MANIPULATION WITH A VIEW TO GENE-THERAPY FOR PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA (PNH), British Journal of Haematology, 93, 1996, pp. 319-319
SERUM ERYTHROPOIETIN LEVELS IN PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA - IMPLICATIONS FOR THERAPY
Authors: MCMULLIN MF HILLMEN P ELDER GE LAPPIN TRJ LUZZATTO L
Citation: Mf. Mcmullin et al., SERUM ERYTHROPOIETIN LEVELS IN PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA - IMPLICATIONS FOR THERAPY, British Journal of Haematology, 92(4), 1996, pp. 815-817
THE EFFECTS OF G-CSF (FILGRASTIM) USED TO MOBILIZE PERIPHERAL-BLOOD STEM-CELLS (PBSCS) IN PATIENTS WITH PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA(PNH)
Authors: JOHNSON RJ MORGAN GJ OWEN RG SMITH GM OCONNOR S RAWSTRON AC NORFOLK DR PARAPIA LA HILLMEN P
Citation: Rj. Johnson et al., THE EFFECTS OF G-CSF (FILGRASTIM) USED TO MOBILIZE PERIPHERAL-BLOOD STEM-CELLS (PBSCS) IN PATIENTS WITH PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA(PNH), Blood, 88(10), 1996, pp. 3670-3670
BOTH PNH TYPE-II AND PNH TYPE-III CELLS CAN ARISE BY DIFFERENT POINT MUTATIONS AFFECTING THE SAME CODON IN THE PIG-A GENE
Authors: ROLLINSON SJ OCONNOR SJM NORFOLK DR MORGAN GH HILLMEN P
Citation: Sj. Rollinson et al., BOTH PNH TYPE-II AND PNH TYPE-III CELLS CAN ARISE BY DIFFERENT POINT MUTATIONS AFFECTING THE SAME CODON IN THE PIG-A GENE, Blood, 88(10), 1996, pp. 1204-1204
NATURAL-HISTORY OF PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA
Authors: HILLMEN P LEWIS SM BESSLER M LUZZATTO L DACIE JV
Citation: P. Hillmen et al., NATURAL-HISTORY OF PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA, The New England journal of medicine, 333(19), 1995, pp. 1253-1258
MUTATIONS IN THE PIG-A GENE CAUSING PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA ARE MAINLY OF THE FRAMESHIFT TYPE
Authors: NAFA K MASON PJ HILLMEN P LUZZATTO L BESSLER M
Citation: K. Nafa et al., MUTATIONS IN THE PIG-A GENE CAUSING PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA ARE MAINLY OF THE FRAMESHIFT TYPE, Blood, 86(12), 1995, pp. 4650-4655
HEPATIC VEIN-THROMBOSIS IN A PATIENT WITH THE INAB PHENOTYPE - IS DEFICIENCY OF DAF IMPORTANT IN THE DEVELOPMENT OF THROMBOSIS IN PNH
Authors: HILLMEN P HADZIC D OCONNOR SJM MOWAT A LAYTON M
Citation: P. Hillmen et al., HEPATIC VEIN-THROMBOSIS IN A PATIENT WITH THE INAB PHENOTYPE - IS DEFICIENCY OF DAF IMPORTANT IN THE DEVELOPMENT OF THROMBOSIS IN PNH, Blood, 86(10), 1995, pp. 514-514
FACTOR-V LEIDEN MUTATION INVESTIGATED BY A MODIFIED PCR TECHNIQUE IN PNH PATIENTS WITH THROMBOSIS
Authors: NAFA K BESSLER M HILLMEN P MASON PJ VULLIAMY T CASTROMALASPINA H LUZZATTO L
Citation: K. Nafa et al., FACTOR-V LEIDEN MUTATION INVESTIGATED BY A MODIFIED PCR TECHNIQUE IN PNH PATIENTS WITH THROMBOSIS, Blood, 86(10), 1995, pp. 807-807
GENOMIC ORGANIZATION OF THE X-LINKED GENE (PIG-A) THAT IS MUTATED IN PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA AND OF A RELATED AUTOSOMAL PSEUDOGENE MAPPED TO 12Q21
Authors: BESSLER M HILLMEN P LONGO L LUZZATTO L MASON PJ
Citation: M. Bessler et al., GENOMIC ORGANIZATION OF THE X-LINKED GENE (PIG-A) THAT IS MUTATED IN PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA AND OF A RELATED AUTOSOMAL PSEUDOGENE MAPPED TO 12Q21, Human molecular genetics, 3(5), 1994, pp. 751-757
TISSUE-PLASMINOGEN ACTIVATOR FOR HEPATIC VEIN-THROMBOSIS IN PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA
Authors: MCMULLIN MF HILLMEN P JACKSON J GANLY P LUZZATTO L
Citation: Mf. Mcmullin et al., TISSUE-PLASMINOGEN ACTIVATOR FOR HEPATIC VEIN-THROMBOSIS IN PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA, Journal of internal medicine, 235(1), 1994, pp. 85-89
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