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Results: 1-25 | 26-31
Results: 1-25/31
IN-VITRO LOSS OF HETEROZYGOSITY TARGETS THE PTEN MMAC1 GENE IN MELANOMA/
Authors: ROBERTSON GP FURNARI FB MIELE ME GLENDENING MJ WELCH DR FOUNTAIN JW LUGO TG HUANG HJS CAVENEE WK
Citation: Gp. Robertson et al., IN-VITRO LOSS OF HETEROZYGOSITY TARGETS THE PTEN MMAC1 GENE IN MELANOMA/, Proceedings of the National Academy of Sciences of the United Statesof America, 95(16), 1998, pp. 9418-9423
DRUG-RESISTANCE OF HUMAN GLIOBLASTOMA CELLS CONFERRED BY A TUMOR-SPECIFIC MUTANT EPIDERMAL GROWTH-FACTOR RECEPTOR THROUGH MODULATION OF BCL-X-L AND CASPASE-3-LIKE PROTEASES
Authors: NAGANE M LEVITZKI A GAZIT A CAVENEE WK HUANG HJS
Citation: M. Nagane et al., DRUG-RESISTANCE OF HUMAN GLIOBLASTOMA CELLS CONFERRED BY A TUMOR-SPECIFIC MUTANT EPIDERMAL GROWTH-FACTOR RECEPTOR THROUGH MODULATION OF BCL-X-L AND CASPASE-3-LIKE PROTEASES, Proceedings of the National Academy of Sciences of the United Statesof America, 95(10), 1998, pp. 5724-5729
TUMORIGENIC ENHANCEMENT BY MUTANT EGFR MEDIATED BY THRESHOLD LEVELS OF CONSTITUTIVE TYROSINE PHOSPHORYLATION AND UNATTENUATED SIGNALING
Authors: CAVENEE W NAGANE M KLINGBEIL C LIN H NISHIKAWA R JI XD HUANG CM GILL GN WILEY HS HUANG HJS
Citation: W. Cavenee et al., TUMORIGENIC ENHANCEMENT BY MUTANT EGFR MEDIATED BY THRESHOLD LEVELS OF CONSTITUTIVE TYROSINE PHOSPHORYLATION AND UNATTENUATED SIGNALING, Journal of pathology, 186, 1998, pp. 15-15
LACK OF MUTATIONS OF THE PTEN TUMOR-SUPPRESSOR GENE IN 3 BANNAYAN-RILEY-RUVALCABBA-SYNDROME PATIENTS
Authors: CARETHERS JM FUNARI FB ZIGMAN AF LAVINE JE JONES MC GRAHAM GE TEEBI AS HUANG HJS CHAUHAN DP CHANG CL CAVENEE WK BOLAND CR
Citation: Jm. Carethers et al., LACK OF MUTATIONS OF THE PTEN TUMOR-SUPPRESSOR GENE IN 3 BANNAYAN-RILEY-RUVALCABBA-SYNDROME PATIENTS, Gastroenterology, 114(4), 1998, pp. 2349-2349
THE PHOSPHOINOSITOL PHOSPHATASE-ACTIVITY OF PTEN MEDIATES A SERUM-SENSITIVE G(1) GROWTH ARREST IN GLIOMA-CELLS
Authors: FURNARI FB HUANG HJS CAVENEE WK
Citation: Fb. Furnari et al., THE PHOSPHOINOSITOL PHOSPHATASE-ACTIVITY OF PTEN MEDIATES A SERUM-SENSITIVE G(1) GROWTH ARREST IN GLIOMA-CELLS, Cancer research, 58(22), 1998, pp. 5002-5008
ABSENCE OF PTEN MMAC1 GERM-LINE MUTATIONS IN SPORADIC BANNAYAN-RILEY-RUVALCABA-SYNDROME/
Authors: CARETHERS JM FURNARI FB ZIGMAN AF LAVINE JE JONES MC GRAHAM GE TEEBI AS HUANG HJS HA HT CHAUHAN DP CHANG CL CAVENEE WK BOLAND CR
Citation: Jm. Carethers et al., ABSENCE OF PTEN MMAC1 GERM-LINE MUTATIONS IN SPORADIC BANNAYAN-RILEY-RUVALCABA-SYNDROME/, Cancer research, 58(13), 1998, pp. 2724-2726
EXPRESSION OF VASCULAR ENDOTHELIAL GROWTH-FACTOR IN HUMAN BRAIN-TUMORS
Authors: NISHIKAWA R CHENG SY NAGASHIMA R HUANG HJS CAVENEE WK MATSUTANI M
Citation: R. Nishikawa et al., EXPRESSION OF VASCULAR ENDOTHELIAL GROWTH-FACTOR IN HUMAN BRAIN-TUMORS, Acta Neuropathologica, 96(5), 1998, pp. 453-462
FUNCTIONAL-ANALYSIS OF WILD-TYPE AND MALIGNANT GLIOMA DERIVED CDKN2A-BETA ALLELES - EVIDENCE FOR AN RB-INDEPENDENT GROWTH SUPPRESSIVE PATHWAY
Authors: ARAP W KNUDSEN E SEWELL DA SIDRANSKY D WANG JYJ HUANG HJS CAVENEE WK
Citation: W. Arap et al., FUNCTIONAL-ANALYSIS OF WILD-TYPE AND MALIGNANT GLIOMA DERIVED CDKN2A-BETA ALLELES - EVIDENCE FOR AN RB-INDEPENDENT GROWTH SUPPRESSIVE PATHWAY, Oncogene, 15(17), 1997, pp. 2013-2020
POINT MUTATIONS CAN INACTIVATE IN-VITRO AND IN-VIVO ACTIVITIES OF P16(INK4A) CDKN2A IN HUMAN GLIOMA/
Authors: ARAP W KNUDSEN ES WANG JYJ CAVENEE WK HUANG HJS
Citation: W. Arap et al., POINT MUTATIONS CAN INACTIVATE IN-VITRO AND IN-VIVO ACTIVITIES OF P16(INK4A) CDKN2A IN HUMAN GLIOMA/, Oncogene, 14(5), 1997, pp. 603-609
TUMORIGENIC ENHANCEMENT BY MUTANT EGFR MEDIATED BY THRESHOLD LEVELS OF CONSTITUTIVE TYROSINE PHOSPHORYLATION AND UNATTENUATED SIGNALING
Authors: CAVENEE W NAGANE M KLINGBEIL C LIN H NISHIKAWA R JI XD HUANG CM GILL GN WILEY HS HUANG HJS
Citation: W. Cavenee et al., TUMORIGENIC ENHANCEMENT BY MUTANT EGFR MEDIATED BY THRESHOLD LEVELS OF CONSTITUTIVE TYROSINE PHOSPHORYLATION AND UNATTENUATED SIGNALING, The FASEB journal, 11(9), 1997, pp. 3458-3458
GROWTH SUPPRESSION OF GLIOMA-CELLS BY PTEN REQUIRES A FUNCTIONAL PHOSPHATASE CATALYTIC DOMAIN
Authors: FURNARI FB LIN H HUANG HJS CAVENEE WK
Citation: Fb. Furnari et al., GROWTH SUPPRESSION OF GLIOMA-CELLS BY PTEN REQUIRES A FUNCTIONAL PHOSPHATASE CATALYTIC DOMAIN, Proceedings of the National Academy of Sciences of the United Statesof America, 94(23), 1997, pp. 12479-12484
INTRACEREBRAL TUMOR-ASSOCIATED HEMORRHAGE CAUSED BY OVEREXPRESSION OFTHE VASCULAR ENDOTHELIAL GROWTH-FACTOR ISOFORMS VEGF(121) AND VEGF(165) BUT NOT VEGF(189)
Authors: CHENG SY NAGANE M HUANG HJS CAVENEE WK
Citation: Sy. Cheng et al., INTRACEREBRAL TUMOR-ASSOCIATED HEMORRHAGE CAUSED BY OVEREXPRESSION OFTHE VASCULAR ENDOTHELIAL GROWTH-FACTOR ISOFORMS VEGF(121) AND VEGF(165) BUT NOT VEGF(189), Proceedings of the National Academy of Sciences of the United Statesof America, 94(22), 1997, pp. 12081-12087
THE ENHANCED TUMORIGENIC ACTIVITY OF A MUTANT EPIDERMAL GROWTH-FACTORRECEPTOR COMMON IN HUMAN CANCERS IS MEDIATED BY THRESHOLD LEVELS OF CONSTITUTIVE TYROSINE PHOSPHORYLATION AND UNATTENUATED SIGNALING
Authors: HUANG HJS NAGANE M KLINGBEIL CK LIN H NISHIKAWA R JI XD HUANG CM GILL GN WILEY HS CAVENEE WK
Citation: Hjs. Huang et al., THE ENHANCED TUMORIGENIC ACTIVITY OF A MUTANT EPIDERMAL GROWTH-FACTORRECEPTOR COMMON IN HUMAN CANCERS IS MEDIATED BY THRESHOLD LEVELS OF CONSTITUTIVE TYROSINE PHOSPHORYLATION AND UNATTENUATED SIGNALING, The Journal of biological chemistry, 272(5), 1997, pp. 2927-2935
CYCLIN-DEPENDENT KINASE-6 (CDK6) AMPLIFICATION IN HUMAN GLIOMAS IDENTIFIED USING 2-DIMENSIONAL SEPARATION OF GENOMIC DNA
Authors: COSTELLO JF PLASS C ARAP W CHAPMAN VM HELD WA BERGER MS HUANG HJS CAVENEE WK
Citation: Jf. Costello et al., CYCLIN-DEPENDENT KINASE-6 (CDK6) AMPLIFICATION IN HUMAN GLIOMAS IDENTIFIED USING 2-DIMENSIONAL SEPARATION OF GENOMIC DNA, Cancer research, 57(7), 1997, pp. 1250-1254
EXPRESSION AND DISTRIBUTION OF VASCULAR ENDOTHELIAL GROWTH-FACTOR PROTEIN IN HUMAN BRAIN-TUMORS
Authors: PIETSCH T VALTER MM WOLF HK VONDEIMLING A HUANG HJS CAVENEE WK WIESTLER OD
Citation: T. Pietsch et al., EXPRESSION AND DISTRIBUTION OF VASCULAR ENDOTHELIAL GROWTH-FACTOR PROTEIN IN HUMAN BRAIN-TUMORS, Acta Neuropathologica, 93(2), 1997, pp. 109-117
MOLECULAR-BIOLOGY OF MALIGNANT DEGENERATION OF ASTROCYTOMA
Authors: FURNARI FB HUANG HJS CAVENEE WK
Citation: Fb. Furnari et al., MOLECULAR-BIOLOGY OF MALIGNANT DEGENERATION OF ASTROCYTOMA, Pediatric neurosurgery, 24(1), 1996, pp. 41-49
SUPPRESSION OF GLIOBLASTOMA ANGIOGENICITY AND TUMORIGENICITY BY INHIBITION OF ENDOGENOUS EXPRESSION OF VASCULAR ENDOTHELIAL GROWTH-FACTOR
Authors: CHENG SY HUANG HJS NAGANE M JI XD WANG DG SHIH CCY ARAP W HUANG CM CAVENEE WK
Citation: Sy. Cheng et al., SUPPRESSION OF GLIOBLASTOMA ANGIOGENICITY AND TUMORIGENICITY BY INHIBITION OF ENDOGENOUS EXPRESSION OF VASCULAR ENDOTHELIAL GROWTH-FACTOR, Proceedings of the National Academy of Sciences of the United Statesof America, 93(16), 1996, pp. 8502-8507
ENHANCED TUMORIGENIC BEHAVIOR OF GLIOBLASTOMA CELLS EXPRESSING A TRUNCATED EPIDERMAL GROWTH-FACTOR RECEPTOR IS MEDIATED THROUGH THE RAS-SHC-GRB2 PATHWAY
Authors: PRIGENT SA NAGANE M LIN H HUVAR I BOSS GR FERAMISCO JR CAVENEE WK HUANG HJS
Citation: Sa. Prigent et al., ENHANCED TUMORIGENIC BEHAVIOR OF GLIOBLASTOMA CELLS EXPRESSING A TRUNCATED EPIDERMAL GROWTH-FACTOR RECEPTOR IS MEDIATED THROUGH THE RAS-SHC-GRB2 PATHWAY, The Journal of biological chemistry, 271(41), 1996, pp. 25639-25645
CAMP EFFECTS ON MYOGENIC GENE-EXPRESSION IN RHABDOMYOSARCOMA CELLS
Authors: WASSERMAN LM NEWSHAM I HUANG HJS CAVENEE WK
Citation: Lm. Wasserman et al., CAMP EFFECTS ON MYOGENIC GENE-EXPRESSION IN RHABDOMYOSARCOMA CELLS, Experimental cell research, 227(1), 1996, pp. 55-62
A COMMON MUTANT EPIDERMAL GROWTH-FACTOR RECEPTOR CONFERS ENHANCED TUMORIGENICITY ON HUMAN GLIOBLASTOMA CELLS BY INCREASING PROLIFERATION AND REDUCING APOPTOSIS
Authors: NAGANE M COUFAL F LIN H BOGLER O CAVENEE WK HUANG HJS
Citation: M. Nagane et al., A COMMON MUTANT EPIDERMAL GROWTH-FACTOR RECEPTOR CONFERS ENHANCED TUMORIGENICITY ON HUMAN GLIOBLASTOMA CELLS BY INCREASING PROLIFERATION AND REDUCING APOPTOSIS, Cancer research, 56(21), 1996, pp. 5079-5086
TYRPHOSTIN AG-1478 PREFERENTIALLY INHIBITS HUMAN GLIOMA-CELLS EXPRESSING TRUNCATED RATHER THAN WILD-TYPE EPIDERMAL GROWTH-FACTOR RECEPTORS
Authors: HAN YC CADAY CG NANDA A CAVENEE WK HUANG HJS
Citation: Yc. Han et al., TYRPHOSTIN AG-1478 PREFERENTIALLY INHIBITS HUMAN GLIOMA-CELLS EXPRESSING TRUNCATED RATHER THAN WILD-TYPE EPIDERMAL GROWTH-FACTOR RECEPTORS, Cancer research, 56(17), 1996, pp. 3859-3861
SILENCING OF P16 CDKN2 EXPRESSION IN HUMAN GLIOMAS BY METHYLATION ANDCHROMATIN CONDENSATION/
Authors: COSTELLO JF BERGER MS HUANG HJS CAVENEE WK
Citation: Jf. Costello et al., SILENCING OF P16 CDKN2 EXPRESSION IN HUMAN GLIOMAS BY METHYLATION ANDCHROMATIN CONDENSATION/, Cancer research, 56(10), 1996, pp. 2405-2410
THE P53 GENE AND ITS ROLE IN HUMAN BRAIN-TUMORS
Authors: BOGLER O HUANG HJS KLEIHUES P CAVENEE WK
Citation: O. Bogler et al., THE P53 GENE AND ITS ROLE IN HUMAN BRAIN-TUMORS, Glia, 15(3), 1995, pp. 308-327
GENETICS AND MALIGNANT PROGRESSION OF HUMAN BRAIN-TUMORS
Authors: FURNARI FB HUANG HJS CAVENEE WK
Citation: Fb. Furnari et al., GENETICS AND MALIGNANT PROGRESSION OF HUMAN BRAIN-TUMORS, Cancer surveys, 25, 1995, pp. 233-275
SINGLE-CELL MONITORING OF GROWTH ARREST AND MORPHOLOGICAL-CHANGES INDUCED BY TRANSFER OF WILD-TYPE P53 ALLELES TO GLIOBLASTOMA CELLS
Authors: VANMEIR EG ROEMER K DISERENS AC KIKUCHI T REMPEL SA HAAS M HUANG HJS FRIEDMANN T DETRIBOLET N CAVENEE WK
Citation: Eg. Vanmeir et al., SINGLE-CELL MONITORING OF GROWTH ARREST AND MORPHOLOGICAL-CHANGES INDUCED BY TRANSFER OF WILD-TYPE P53 ALLELES TO GLIOBLASTOMA CELLS, Proceedings of the National Academy of Sciences of the United Statesof America, 92(4), 1995, pp. 1008-1012
Risultati: 1-25 | 26-31