Authors:
ROBERTSON GP
FURNARI FB
MIELE ME
GLENDENING MJ
WELCH DR
FOUNTAIN JW
LUGO TG
HUANG HJS
CAVENEE WK
Citation: Gp. Robertson et al., IN-VITRO LOSS OF HETEROZYGOSITY TARGETS THE PTEN MMAC1 GENE IN MELANOMA/, Proceedings of the National Academy of Sciences of the United Statesof America, 95(16), 1998, pp. 9418-9423
Authors:
NAGANE M
LEVITZKI A
GAZIT A
CAVENEE WK
HUANG HJS
Citation: M. Nagane et al., DRUG-RESISTANCE OF HUMAN GLIOBLASTOMA CELLS CONFERRED BY A TUMOR-SPECIFIC MUTANT EPIDERMAL GROWTH-FACTOR RECEPTOR THROUGH MODULATION OF BCL-X-L AND CASPASE-3-LIKE PROTEASES, Proceedings of the National Academy of Sciences of the United Statesof America, 95(10), 1998, pp. 5724-5729
Authors:
CAVENEE W
NAGANE M
KLINGBEIL C
LIN H
NISHIKAWA R
JI XD
HUANG CM
GILL GN
WILEY HS
HUANG HJS
Citation: W. Cavenee et al., TUMORIGENIC ENHANCEMENT BY MUTANT EGFR MEDIATED BY THRESHOLD LEVELS OF CONSTITUTIVE TYROSINE PHOSPHORYLATION AND UNATTENUATED SIGNALING, Journal of pathology, 186, 1998, pp. 15-15
Authors:
CARETHERS JM
FUNARI FB
ZIGMAN AF
LAVINE JE
JONES MC
GRAHAM GE
TEEBI AS
HUANG HJS
CHAUHAN DP
CHANG CL
CAVENEE WK
BOLAND CR
Citation: Jm. Carethers et al., LACK OF MUTATIONS OF THE PTEN TUMOR-SUPPRESSOR GENE IN 3 BANNAYAN-RILEY-RUVALCABBA-SYNDROME PATIENTS, Gastroenterology, 114(4), 1998, pp. 2349-2349
Citation: Fb. Furnari et al., THE PHOSPHOINOSITOL PHOSPHATASE-ACTIVITY OF PTEN MEDIATES A SERUM-SENSITIVE G(1) GROWTH ARREST IN GLIOMA-CELLS, Cancer research, 58(22), 1998, pp. 5002-5008
Authors:
CARETHERS JM
FURNARI FB
ZIGMAN AF
LAVINE JE
JONES MC
GRAHAM GE
TEEBI AS
HUANG HJS
HA HT
CHAUHAN DP
CHANG CL
CAVENEE WK
BOLAND CR
Citation: Jm. Carethers et al., ABSENCE OF PTEN MMAC1 GERM-LINE MUTATIONS IN SPORADIC BANNAYAN-RILEY-RUVALCABA-SYNDROME/, Cancer research, 58(13), 1998, pp. 2724-2726
Authors:
ARAP W
KNUDSEN E
SEWELL DA
SIDRANSKY D
WANG JYJ
HUANG HJS
CAVENEE WK
Citation: W. Arap et al., FUNCTIONAL-ANALYSIS OF WILD-TYPE AND MALIGNANT GLIOMA DERIVED CDKN2A-BETA ALLELES - EVIDENCE FOR AN RB-INDEPENDENT GROWTH SUPPRESSIVE PATHWAY, Oncogene, 15(17), 1997, pp. 2013-2020
Authors:
ARAP W
KNUDSEN ES
WANG JYJ
CAVENEE WK
HUANG HJS
Citation: W. Arap et al., POINT MUTATIONS CAN INACTIVATE IN-VITRO AND IN-VIVO ACTIVITIES OF P16(INK4A) CDKN2A IN HUMAN GLIOMA/, Oncogene, 14(5), 1997, pp. 603-609
Authors:
CAVENEE W
NAGANE M
KLINGBEIL C
LIN H
NISHIKAWA R
JI XD
HUANG CM
GILL GN
WILEY HS
HUANG HJS
Citation: W. Cavenee et al., TUMORIGENIC ENHANCEMENT BY MUTANT EGFR MEDIATED BY THRESHOLD LEVELS OF CONSTITUTIVE TYROSINE PHOSPHORYLATION AND UNATTENUATED SIGNALING, The FASEB journal, 11(9), 1997, pp. 3458-3458
Citation: Fb. Furnari et al., GROWTH SUPPRESSION OF GLIOMA-CELLS BY PTEN REQUIRES A FUNCTIONAL PHOSPHATASE CATALYTIC DOMAIN, Proceedings of the National Academy of Sciences of the United Statesof America, 94(23), 1997, pp. 12479-12484
Citation: Sy. Cheng et al., INTRACEREBRAL TUMOR-ASSOCIATED HEMORRHAGE CAUSED BY OVEREXPRESSION OFTHE VASCULAR ENDOTHELIAL GROWTH-FACTOR ISOFORMS VEGF(121) AND VEGF(165) BUT NOT VEGF(189), Proceedings of the National Academy of Sciences of the United Statesof America, 94(22), 1997, pp. 12081-12087
Authors:
HUANG HJS
NAGANE M
KLINGBEIL CK
LIN H
NISHIKAWA R
JI XD
HUANG CM
GILL GN
WILEY HS
CAVENEE WK
Citation: Hjs. Huang et al., THE ENHANCED TUMORIGENIC ACTIVITY OF A MUTANT EPIDERMAL GROWTH-FACTORRECEPTOR COMMON IN HUMAN CANCERS IS MEDIATED BY THRESHOLD LEVELS OF CONSTITUTIVE TYROSINE PHOSPHORYLATION AND UNATTENUATED SIGNALING, The Journal of biological chemistry, 272(5), 1997, pp. 2927-2935
Authors:
COSTELLO JF
PLASS C
ARAP W
CHAPMAN VM
HELD WA
BERGER MS
HUANG HJS
CAVENEE WK
Citation: Jf. Costello et al., CYCLIN-DEPENDENT KINASE-6 (CDK6) AMPLIFICATION IN HUMAN GLIOMAS IDENTIFIED USING 2-DIMENSIONAL SEPARATION OF GENOMIC DNA, Cancer research, 57(7), 1997, pp. 1250-1254
Authors:
PIETSCH T
VALTER MM
WOLF HK
VONDEIMLING A
HUANG HJS
CAVENEE WK
WIESTLER OD
Citation: T. Pietsch et al., EXPRESSION AND DISTRIBUTION OF VASCULAR ENDOTHELIAL GROWTH-FACTOR PROTEIN IN HUMAN BRAIN-TUMORS, Acta Neuropathologica, 93(2), 1997, pp. 109-117
Authors:
CHENG SY
HUANG HJS
NAGANE M
JI XD
WANG DG
SHIH CCY
ARAP W
HUANG CM
CAVENEE WK
Citation: Sy. Cheng et al., SUPPRESSION OF GLIOBLASTOMA ANGIOGENICITY AND TUMORIGENICITY BY INHIBITION OF ENDOGENOUS EXPRESSION OF VASCULAR ENDOTHELIAL GROWTH-FACTOR, Proceedings of the National Academy of Sciences of the United Statesof America, 93(16), 1996, pp. 8502-8507
Authors:
PRIGENT SA
NAGANE M
LIN H
HUVAR I
BOSS GR
FERAMISCO JR
CAVENEE WK
HUANG HJS
Citation: Sa. Prigent et al., ENHANCED TUMORIGENIC BEHAVIOR OF GLIOBLASTOMA CELLS EXPRESSING A TRUNCATED EPIDERMAL GROWTH-FACTOR RECEPTOR IS MEDIATED THROUGH THE RAS-SHC-GRB2 PATHWAY, The Journal of biological chemistry, 271(41), 1996, pp. 25639-25645
Authors:
WASSERMAN LM
NEWSHAM I
HUANG HJS
CAVENEE WK
Citation: Lm. Wasserman et al., CAMP EFFECTS ON MYOGENIC GENE-EXPRESSION IN RHABDOMYOSARCOMA CELLS, Experimental cell research, 227(1), 1996, pp. 55-62
Authors:
NAGANE M
COUFAL F
LIN H
BOGLER O
CAVENEE WK
HUANG HJS
Citation: M. Nagane et al., A COMMON MUTANT EPIDERMAL GROWTH-FACTOR RECEPTOR CONFERS ENHANCED TUMORIGENICITY ON HUMAN GLIOBLASTOMA CELLS BY INCREASING PROLIFERATION AND REDUCING APOPTOSIS, Cancer research, 56(21), 1996, pp. 5079-5086
Authors:
HAN YC
CADAY CG
NANDA A
CAVENEE WK
HUANG HJS
Citation: Yc. Han et al., TYRPHOSTIN AG-1478 PREFERENTIALLY INHIBITS HUMAN GLIOMA-CELLS EXPRESSING TRUNCATED RATHER THAN WILD-TYPE EPIDERMAL GROWTH-FACTOR RECEPTORS, Cancer research, 56(17), 1996, pp. 3859-3861
Authors:
COSTELLO JF
BERGER MS
HUANG HJS
CAVENEE WK
Citation: Jf. Costello et al., SILENCING OF P16 CDKN2 EXPRESSION IN HUMAN GLIOMAS BY METHYLATION ANDCHROMATIN CONDENSATION/, Cancer research, 56(10), 1996, pp. 2405-2410
Authors:
VANMEIR EG
ROEMER K
DISERENS AC
KIKUCHI T
REMPEL SA
HAAS M
HUANG HJS
FRIEDMANN T
DETRIBOLET N
CAVENEE WK
Citation: Eg. Vanmeir et al., SINGLE-CELL MONITORING OF GROWTH ARREST AND MORPHOLOGICAL-CHANGES INDUCED BY TRANSFER OF WILD-TYPE P53 ALLELES TO GLIOBLASTOMA CELLS, Proceedings of the National Academy of Sciences of the United Statesof America, 92(4), 1995, pp. 1008-1012