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Results: 1-10 |
Results: 10
FAMILIAL STREPTOMYCIN OTOTOXICITY IN A SOUTH-AFRICAN FAMILY - A MITOCHONDRIAL DISORDER
Authors: GARDNER JC GOLIATH R VILJOEN D SELLARS S CORTOPASSI G HUTCHIN T GREENBERG J BEIGHTON P
Citation: Jc. Gardner et al., FAMILIAL STREPTOMYCIN OTOTOXICITY IN A SOUTH-AFRICAN FAMILY - A MITOCHONDRIAL DISORDER, Journal of Medical Genetics, 34(11), 1997, pp. 904-906
DEGENERATION OF HUMAN ONCOGENES AND MITOCHONDRIAL GENES OCCURS IN CELLS THAT EXHIBIT AGE-RELATED PATHOLOGY
Authors: CORTOPASSI G LIU YF HUTCHIN T
Citation: G. Cortopassi et al., DEGENERATION OF HUMAN ONCOGENES AND MITOCHONDRIAL GENES OCCURS IN CELLS THAT EXHIBIT AGE-RELATED PATHOLOGY, Experimental gerontology, 31(1-2), 1996, pp. 253-265
TOPICAL GENTAMICIN-INDUCED HEARING-LOSS - A MITOCHONDRIAL RIBOSOMAL-RNA STUDY OF GENETIC SUSCEPTIBILITY
Authors: CHEN JM WILLIAMSON PA HUTCHIN T NEDZELSKI JM CORTOPASSI GA
Citation: Jm. Chen et al., TOPICAL GENTAMICIN-INDUCED HEARING-LOSS - A MITOCHONDRIAL RIBOSOMAL-RNA STUDY OF GENETIC SUSCEPTIBILITY, The American journal of otology, 17(6), 1996, pp. 850-852
MITOCHONDRIAL-DNA HAPLOTYPE PREDICTS DEAFNESS RISK
Authors: HUTCHIN T CORTOPASSI G
Citation: T. Hutchin et G. Cortopassi, MITOCHONDRIAL-DNA HAPLOTYPE PREDICTS DEAFNESS RISK, American journal of medical genetics, 60(6), 1995, pp. 592-592
A MITOCHONDRIAL-DNA CLONE IS ASSOCIATED WITH INCREASED RISK FOR ALZHEIMER-DISEASE
Authors: HUTCHIN T CORTOPASSI G
Citation: T. Hutchin et G. Cortopassi, A MITOCHONDRIAL-DNA CLONE IS ASSOCIATED WITH INCREASED RISK FOR ALZHEIMER-DISEASE, Proceedings of the National Academy of Sciences of the United Statesof America, 92(15), 1995, pp. 6892-6895
MITOCHONDRIAL AND APOE GENOTYPING AND RISK FOR ALZHEIMERS-DISEASE
Authors: HUTCHIN T PERICAKVANCE M CORTOPASSI G
Citation: T. Hutchin et al., MITOCHONDRIAL AND APOE GENOTYPING AND RISK FOR ALZHEIMERS-DISEASE, American journal of human genetics, 57(4), 1995, pp. 1401-1401
A MOLECULAR AND CELLULAR HYPOTHESIS FOR AMINOGLYCOSIDE-INDUCED DEAFNESS
Authors: CORTOPASSI G HUTCHIN T
Citation: G. Cortopassi et T. Hutchin, A MOLECULAR AND CELLULAR HYPOTHESIS FOR AMINOGLYCOSIDE-INDUCED DEAFNESS, Hearing research, 78(1), 1994, pp. 27-30
PROPOSED MOLECULAR AND CELLULAR MECHANISM FOR AMINOGLYCOSIDE OTOTOXICITY
Authors: HUTCHIN T CORTOPASSI G
Citation: T. Hutchin et G. Cortopassi, PROPOSED MOLECULAR AND CELLULAR MECHANISM FOR AMINOGLYCOSIDE OTOTOXICITY, Antimicrobial agents and chemotherapy, 38(11), 1994, pp. 2517-2520
PROPOSED MOLECULAR AND CELLULAR MECHANISM FOR AMINOGLYCOSIDE OTOTOXICITY
Authors: HUTCHIN T CORTOPASSI G
Citation: T. Hutchin et G. Cortopassi, PROPOSED MOLECULAR AND CELLULAR MECHANISM FOR AMINOGLYCOSIDE OTOTOXICITY, Antimicrobial agents and chemotherapy, 38(11), 1994, pp. 2517-2520
A MOLECULAR-BASIS FOR HUMAN HYPERSENSITIVITY TO AMINOGLYCOSIDE ANTIBIOTICS
Authors: HUTCHIN T HAWORTH I HIGASHI K FISCHEGHODSIAN N STONEKING M SAHA N ARNOS C CORTOPASSI G
Citation: T. Hutchin et al., A MOLECULAR-BASIS FOR HUMAN HYPERSENSITIVITY TO AMINOGLYCOSIDE ANTIBIOTICS, Nucleic acids research, 21(18), 1993, pp. 4174-4179
Risultati: 1-10 |