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Results: 1-18 |
Results: 18
Role of co-repressors in transcriptional repression mediated by the t(8;21), t(16;21), t(12;21), and inv(16) fusion proteins
Authors: Hiebert, SW Lutterbach, B Amann, J
Citation: Sw. Hiebert et al., Role of co-repressors in transcriptional repression mediated by the t(8;21), t(16;21), t(12;21), and inv(16) fusion proteins, CURR OPIN H, 8(4), 2001, pp. 197-200
E2F-1 induces the stabilization of p53 but blocks p53-mediated transactivation
Authors: Nip, J Strom, DK Eischen, CM Cleveland, JL Zambetti, GP Hiebert, SW
Citation: J. Nip et al., E2F-1 induces the stabilization of p53 but blocks p53-mediated transactivation, ONCOGENE, 20(8), 2001, pp. 910-920
Bcl-2 is an apoptotic target suppressed by both c-Myc and E2F-1
Authors: Eischen, CM Packham, G Nip, J Fee, BE Hiebert, SW Zambetti, GP Cleveland, JL
Citation: Cm. Eischen et al., Bcl-2 is an apoptotic target suppressed by both c-Myc and E2F-1, ONCOGENE, 20(48), 2001, pp. 6983-6993
TEL contacts multiple co-repressors and specifically associates with histone deacetylase-3
Authors: Wang, LL Hiebert, SW
Citation: Ll. Wang et Sw. Hiebert, TEL contacts multiple co-repressors and specifically associates with histone deacetylase-3, ONCOGENE, 20(28), 2001, pp. 3716-3725
Mechanisms of transcriptional repression by the t(8;21)-, t(12;21)-, and inv(16)-encoded fusion proteins
Authors: Hiebert, SW Lutterbach, B Durst, K Wang, LL Linggi, B Wu, S Wood, L Amann, J King, D Hou, Y
Citation: Sw. Hiebert et al., Mechanisms of transcriptional repression by the t(8;21)-, t(12;21)-, and inv(16)-encoded fusion proteins, CANC CHEMOT, 48, 2001, pp. S31-S34
ETO, a target of t(8;21) in acute leukemia, makes distinct contacts with multiple histone deacetylases and binds mSin3A through its oligomerization domain
Authors: Amann, JM Nip, J Strom, DK Lutterbach, B Harada, H Lenny, N Downing, JR Meyers, S Hiebert, SW
Citation: Jm. Amann et al., ETO, a target of t(8;21) in acute leukemia, makes distinct contacts with multiple histone deacetylases and binds mSin3A through its oligomerization domain, MOL CELL B, 21(19), 2001, pp. 6470-6483
Alterations in subnuclear trafficking of nuclear regulatory factors in acute leukemia
Authors: Meyers, S Hiebert, SW
Citation: S. Meyers et Sw. Hiebert, Alterations in subnuclear trafficking of nuclear regulatory factors in acute leukemia, J CELL BIOC, 2000, pp. 93-98
Role of the transcription factor AML-1 in acute leukemia and hematopoieticdifferentiation
Authors: Lutterbach, B Hiebert, SW
Citation: B. Lutterbach et Sw. Hiebert, Role of the transcription factor AML-1 in acute leukemia and hematopoieticdifferentiation, GENE, 245(2), 2000, pp. 223-235
The ETO protein disrupted in t(8;21)-associated acute myeloid leukemia is a corepressor for the promyelocytic leukemia zinc finger protein
Authors: Melnick, AM Westendorf, JJ Polinger, A Carlile, GW Arai, S Ball, HJ Lutterbach, B Hiebert, SW Licht, JD
Citation: Am. Melnick et al., The ETO protein disrupted in t(8;21)-associated acute myeloid leukemia is a corepressor for the promyelocytic leukemia zinc finger protein, MOL CELL B, 20(6), 2000, pp. 2075-2086
TEL, a putative tumor suppressor, modulates cell growth and cell morphology of Ras-transformed cells while repressing the transcription of stromelysin-1
Authors: Fenrick, R Wang, LL Nip, J Amann, JM Rooney, RJ Walker-Daniels, J Crawford, HC Hulboy, DL Kinch, MS Matrisian, LM Hiebert, SW
Citation: R. Fenrick et al., TEL, a putative tumor suppressor, modulates cell growth and cell morphology of Ras-transformed cells while repressing the transcription of stromelysin-1, MOL CELL B, 20(16), 2000, pp. 5828-5839
Expression of the AML-1 oncogene shortens the G(1) phase of the cell cycle
Authors: Strom, DK Nipp, J Westendorf, JJ Linggi, B Lutterbach, B Downing, JR Lenny, N Hiebert, SW
Citation: Dk. Strom et al., Expression of the AML-1 oncogene shortens the G(1) phase of the cell cycle, J BIOL CHEM, 275(5), 2000, pp. 3438-3445
A mechanism of repression by acute myeloid leukemia-1, the target of multiple chromosomal translocations in acute leukemia
Authors: Lutterbach, B Westendorf, JJ Linggi, B Isaac, S Seto, E Hiebert, SW
Citation: B. Lutterbach et al., A mechanism of repression by acute myeloid leukemia-1, the target of multiple chromosomal translocations in acute leukemia, J BIOL CHEM, 275(1), 2000, pp. 651-656
AML-1/ETO fusion protein is a dominant negative inhibitor of transcriptional repression by the promyelocptic leukemia zinc finger protein
Authors: Melnick, A Carlile, GW McConnell, MJ Polinger, A Hiebert, SW Licht, JD
Citation: A. Melnick et al., AML-1/ETO fusion protein is a dominant negative inhibitor of transcriptional repression by the promyelocptic leukemia zinc finger protein, BLOOD, 96(12), 2000, pp. 3939-3947
Mammalian runt-domain proteins and their roles in hematopoiesis, osteogenesis, and leukemia
Authors: Westendorf, JJ Hiebert, SW
Citation: Jj. Westendorf et Sw. Hiebert, Mammalian runt-domain proteins and their roles in hematopoiesis, osteogenesis, and leukemia, J CELL BIOC, 1999, pp. 51-58
Identification and characterization of a protein containing formin homology (FH1/FH2) domains
Authors: Westendorf, JJ Mernaugh, R Hiebert, SW
Citation: Jj. Westendorf et al., Identification and characterization of a protein containing formin homology (FH1/FH2) domains, GENE, 232(2), 1999, pp. 173-182
Both TEL and AML-1 contribute repression domains to the t(12;21) fusion protein
Authors: Fenrick, R Amann, JM Lutterbach, B Wang, LL Westendorf, JJ Downing, JR Hiebert, SW
Citation: R. Fenrick et al., Both TEL and AML-1 contribute repression domains to the t(12;21) fusion protein, MOL CELL B, 19(10), 1999, pp. 6566-6574
The inv(16) encodes an acute myeloid leukemia 1 transcriptional corepressor
Authors: Lutterbach, B Hou, Y Durst, KL Hiebert, SW
Citation: B. Lutterbach et al., The inv(16) encodes an acute myeloid leukemia 1 transcriptional corepressor, P NAS US, 96(22), 1999, pp. 12822-12827
Role of histone deacetylases in acute leukemia
Authors: Fenrick, R Hiebert, SW
Citation: R. Fenrick et Sw. Hiebert, Role of histone deacetylases in acute leukemia, J CELL BIOC, 1998, pp. 194-202
Risultati: 1-18 |