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Citation: F. De Beer et al., Apolipoprotein E2 (Lys146 -> Gln) causes hypertriglyceridemia due to an apolipoprotein E variant-specific inhibition of lipolysis of very low densitylipoproteins-triglycerides, ART THROM V, 20(7), 2000, pp. 1800-1806
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Citation: Mpj. De Winther et al., Macrophage scavenger receptor class A - A multifunctional receptor in atherosclerosis, ART THROM V, 20(2), 2000, pp. 290-297
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Citation: M. Van Eck et al., Effect of human scavenger receptor class A overexpression in bone marrow-derived cells on cholesterol levels and atherosclerosis in ApoE-deficient mice, ART THROM V, 20(12), 2000, pp. 2600-2606
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Citation: M. Van Eck et al., Effect of macrophage-derived mouse apoE, human apoE3-Leiden, and human apoE2 (Arg158 -> Cys) on cholesterol levels and atherosclerosis in apoE-deficient mice, ART THROM V, 20(1), 2000, pp. 119-127
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Citation: M. Van Geel et al., Identification of a novel beta-tubulin subfamily with one member (TUBB4Q) located near the telomere of chromosome region 4q35, CYTOG C GEN, 88(3-4), 2000, pp. 316-321
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Citation: Pj. Tacken et al., Very-low-density lipoprotein binding to the apolipoprotein E receptor 2 isenhanced by lipoprotein lipase, and does not require apolipoprotein E, BIOCHEM J, 347, 2000, pp. 357-361
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Citation: Ar. Mensenkamp et al., Hepatic lipid accumulation, altered very low density lipoprotein formationand apolipoprotein E deposition in apolipoprotein E3-Leiden transgenic mice, J HEPATOL, 33(2), 2000, pp. 189-198
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Citation: N. Herijgers et al., Effect of human scavenger receptor class A overexpression in bone marrow-derived cells on lipoprotein metabolism and atherosclerosis in low density lipoprotein receptor knockout mice, J LIPID RES, 41(9), 2000, pp. 1402-1409
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van Dijk, KW
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Citation: F. De Beer et al., Binding of beta-VLDL to heparan sulfate proteoglycans requires lipoproteinlipase, whereas ApoE only modulates binding affinity, ART THROM V, 19(3), 1999, pp. 633-637
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Citation: Kw. Van Dijk et al., Hyperlipidemia of ApoE2(Arg(158)-Cys) and ApoE3-Leiden transgenic mice is modulated predominantly by LDL receptor expression, ART THROM V, 19(12), 1999, pp. 2945-2951
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Citation: Mc. Jong et al., Reversal of hyperlipidaemia in apolipoprotein C1 transgenic mice by adenovirus-mediated gene delivery of the low-density-lipoprotein receptor, but not by the very-low-density-lipoprotein receptor, BIOCHEM J, 338, 1999, pp. 281-287
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Citation: Kw. Van Dijk et al., In LDL receptor-deficient mice, catabolism of remnant lipoproteins requires a high level of apoE but is inhibited by excess apoE, J LIPID RES, 40(2), 1999, pp. 336-344
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Hofker, MH
van Dijk, KW
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Citation: Ar. Mensenkamp et al., Apolipoprotein E participates in the regulation of very low density lipoprotein-triglyceride secretion by the liver, J BIOL CHEM, 274(50), 1999, pp. 35711-35718
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van Dijk, KW
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Havekes, LM
Hofker, MH
Citation: Mpj. De Winther et al., Macrophage specific overexpression of the human macrophage scavenger receptor in transgenic mice, using a 180-kb yeast artificial chromosome, leads to enhanced foam cell formation of isolated peritoneal macrophages, ATHEROSCLER, 147(2), 1999, pp. 339-347
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van Dijk, KW
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Citation: Mpj. De Winther et al., Scavenger receptor deficiency leads to more complex atherosclerotic lesions in APOE3Leiden transgenic mice, ATHEROSCLER, 144(2), 1999, pp. 315-321
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van der Cammen, M
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Emeis, JJ
Havekes, LM
Hofker, MH
Kraal, G
Citation: Mjj. Gijbels et al., Progression and regression of atherosclerosis in APOE3-Leiden transgenic mice: an immunohistochemical study, ATHEROSCLER, 143(1), 1999, pp. 15-25