AAAAAA

   
Results: 1-12 |
Results: 12

Authors: Cremesti, A Paris, F Grassme, H Holler, N Tschopp, J Fuks, Z Gulbins, E Kolesnick, R
Citation: A. Cremesti et al., Ceramide enables Fas to cap and kill, J BIOL CHEM, 276(26), 2001, pp. 23954-23961

Authors: Kataoka, T Holler, N Micheau, O Martinon, F Tinel, A Hofmann, K Tschopp, J
Citation: T. Kataoka et al., Bcl-rambo, a novel Bcl-2 homologue that induces apoptosis via its unique C-terminal extension, J BIOL CHEM, 276(22), 2001, pp. 19548-19554

Authors: Werner-Favre, C Bovia, F Schneider, P Holler, N Barnet, M Kindler, V Tschopp, J Zubler, RH
Citation: C. Werner-favre et al., IgG subclass switch capacity is low in switched and in IgM-only, but high in IgD(+)IgM(+), post-germinal center (CD27(+)) human B cells, EUR J IMMUN, 31(1), 2001, pp. 243-249

Authors: Holler, N Zaru, R Micheau, O Thome, M Attinger, A Valitutti, S Bodmer, JL Schneider, P Seed, B Tschopp, J
Citation: N. Holler et al., Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule, NAT IMMUNOL, 1(6), 2000, pp. 489-495

Authors: Bodmer, JL Holler, N Reynard, S Vinciguerra, P Schneider, P Juo, P Blenis, J Tschopp, J
Citation: Jl. Bodmer et al., TRAIL receptor-2 signals apoptosis through FADD and caspase-8, NAT CELL BI, 2(4), 2000, pp. 241-243

Authors: Kataoka, T Budd, RC Holler, N Thome, M Martinon, F Irmler, M Burns, K Hahne, M Kennedy, N Kovacsovics, M Tschopp, J
Citation: T. Kataoka et al., The caspase-8 inhibitor FLIP promotes activation of NF-kappa B and Erk signaling pathways, CURR BIOL, 10(11), 2000, pp. 640-648

Authors: Villunger, A Huang, DCS Holler, N Tschopp, J Strasser, A
Citation: A. Villunger et al., Fas ligand-induced c-Jun kinase activation in lymphoid cells requires extensive receptor aggregation but is independent of DAXX, and Fas-mediated cell death does not involve DAXX, RIP, or RAIDD, J IMMUNOL, 165(3), 2000, pp. 1337-1343

Authors: Holler, N Kataoka, T Bodmer, JL Romero, P Romero, J Deperthes, D Engel, J Tschopp, J Schneider, P
Citation: N. Holler et al., Development of improved soluble inhibitors of FasL and CD40L based on oligomerized receptors, J IMMUNOL M, 237(1-2), 2000, pp. 159-173

Authors: Villunger, A O'Reilly, LA Holler, N Adams, J Strasser, A
Citation: A. Villunger et al., Fas ligand, Bcl-2, granulocyte colony-stimulating factor, and p38 mitogen-activated protein kinase: Regulators of distinct cell death and survival pathways in granulocytes, J EXP MED, 192(5), 2000, pp. 647-657

Authors: Rennert, P Schneider, P Cachero, TG Thompson, J Trabach, L Hertig, S Holler, N Qian, F Mullen, C Strauch, K Browning, JL Ambrose, C Tschopp, J
Citation: P. Rennert et al., A soluble form of B cell maturation antigen, a receptor for the tumor necrosis factor family member APRIL, inhibits tumor cell growth, J EXP MED, 192(11), 2000, pp. 1677-1683

Authors: Martinon, F Holler, N Richard, C Tschopp, J
Citation: F. Martinon et al., Activation of a pro-apoptotic amplification loop through inhibition of NF-kappa B-dependent survival signals by caspase-mediated inactivation of RIP, FEBS LETTER, 468(2-3), 2000, pp. 134-136

Authors: Schneider, P MacKay, F Steiner, V Hofmann, K Bodmer, JL Holler, N Ambrose, C Lawton, P Bixler, S Acha-Orbea, H Valmori, D Romero, P Werner-Favre, C Zubler, RH Browning, JL Tschopp, J
Citation: P. Schneider et al., BAFF, a novel ligand of the tumor necrosis factor family, stimulates B cell growth, J EXP MED, 189(11), 1999, pp. 1747-1756
Risultati: 1-12 |