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Results: 1-11 |
Results: 11
A PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR-ALPHA (PPAR-ALPHA) CDNA CLONED FROM GUINEA-PIG LIVER ENCODES A PROTEIN WITH SIMILAR PROPERTIESTO THE MOUSE PPAR-ALPHA - IMPLICATIONS FOR SPECIES-DIFFERENCES IN RESPONSES TO PEROXISOME PROLIFERATORS
Authors: TUGWOOD JD HOLDEN PR JAMES NH PRINCE RA ROBERTS RA
Citation: Jd. Tugwood et al., A PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR-ALPHA (PPAR-ALPHA) CDNA CLONED FROM GUINEA-PIG LIVER ENCODES A PROTEIN WITH SIMILAR PROPERTIESTO THE MOUSE PPAR-ALPHA - IMPLICATIONS FOR SPECIES-DIFFERENCES IN RESPONSES TO PEROXISOME PROLIFERATORS, Archives of toxicology, 72(3), 1998, pp. 169-177
THE NONGENOTOXIC HEPATOCARCINOGEN NAFENOPIN SUPPRESSES RODENT HEPATOCYTE APOPTOSIS INDUCED BY TGF-BETA(1), DNA-DAMAGE AND FAS
Authors: GILL JH JAMES NH ROBERTS RA DIVE C
Citation: Jh. Gill et al., THE NONGENOTOXIC HEPATOCARCINOGEN NAFENOPIN SUPPRESSES RODENT HEPATOCYTE APOPTOSIS INDUCED BY TGF-BETA(1), DNA-DAMAGE AND FAS, Carcinogenesis, 19(2), 1998, pp. 299-304
EVIDENCE FOR THE SUPPRESSION OF APOPTOSIS BY THE PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR-ALPHA (PPAR-ALPHA)
Authors: ROBERTS RA JAMES NH WOODYATT NJ MACDONALD N TUGWOOD JD
Citation: Ra. Roberts et al., EVIDENCE FOR THE SUPPRESSION OF APOPTOSIS BY THE PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR-ALPHA (PPAR-ALPHA), Carcinogenesis, 19(1), 1998, pp. 43-48
TUMOR-NECROSIS-FACTOR-ALPHA (TNF-ALPHA) SUPPRESSES APOPTOSIS AND INDUCES DNA-SYNTHESIS IN RODENT HEPATOCYTES - A MEDIATOR OF THE HEPATOCARCINOGENICITY OF PEROXISOME PROLIFERATORS
Authors: ROLFE M JAMES NH ROBERTS RA
Citation: M. Rolfe et al., TUMOR-NECROSIS-FACTOR-ALPHA (TNF-ALPHA) SUPPRESSES APOPTOSIS AND INDUCES DNA-SYNTHESIS IN RODENT HEPATOCYTES - A MEDIATOR OF THE HEPATOCARCINOGENICITY OF PEROXISOME PROLIFERATORS, Carcinogenesis, 18(11), 1997, pp. 2277-2280
SPECIES-DIFFERENCES IN RESPONSE TO PEROXISOME PROLIFERATORS CORRELATEIN-VITRO WITH INDUCTION OF DNA-SYNTHESIS RATHER THAN SUPPRESSION OF APOPTOSIS
Authors: JAMES NH ROBERTS RA
Citation: Nh. James et Ra. Roberts, SPECIES-DIFFERENCES IN RESPONSE TO PEROXISOME PROLIFERATORS CORRELATEIN-VITRO WITH INDUCTION OF DNA-SYNTHESIS RATHER THAN SUPPRESSION OF APOPTOSIS, Carcinogenesis, 17(8), 1996, pp. 1623-1632
ENHANCED HEPATOCYTE COLONY GROWTH IN SOFT AGAR AFTER IN-VIVO TREATMENT WITH A GENOTOXIC CARCINOGEN - A POTENTIAL ASSAY FOR HEPATOCARCINOGENS
Authors: JAMES NH ASHBY J ROBERTS RA
Citation: Nh. James et al., ENHANCED HEPATOCYTE COLONY GROWTH IN SOFT AGAR AFTER IN-VIVO TREATMENT WITH A GENOTOXIC CARCINOGEN - A POTENTIAL ASSAY FOR HEPATOCARCINOGENS, Cancer letters, 93(1), 1995, pp. 121-128
SPECIES-DIFFERENCES IN THE CLONAL EXPANSION OF HEPATOCYTES IN RESPONSE TO THE COACTION OF EPIDERMAL GROWTH-FACTOR AND NAFENOPIN, A RODENT HEPATOCARCINOGENIC PEROXISOME PROLIFERATOR
Authors: JAMES NH ROBERTS RA
Citation: Nh. James et Ra. Roberts, SPECIES-DIFFERENCES IN THE CLONAL EXPANSION OF HEPATOCYTES IN RESPONSE TO THE COACTION OF EPIDERMAL GROWTH-FACTOR AND NAFENOPIN, A RODENT HEPATOCARCINOGENIC PEROXISOME PROLIFERATOR, Fundamental and applied toxicology, 26(1), 1995, pp. 143-149
QUANTITATION OF APOPTOTIC BODIES IN RAT-LIVER BY IN-SITU END LABELING(ISEL) - CORRELATION WITH MORPHOLOGY
Authors: WHEELDON EB WILLIAMS SM SOAMES AR JAMES NH ROBERTS RA
Citation: Eb. Wheeldon et al., QUANTITATION OF APOPTOTIC BODIES IN RAT-LIVER BY IN-SITU END LABELING(ISEL) - CORRELATION WITH MORPHOLOGY, Toxicologic pathology, 23(3), 1995, pp. 410-415
NONGENOTOXIC HEPATOCARCINOGENS STIMULATE DNA-SYNTHESIS AND THEIR WITHDRAWAL INDUCES APOPTOSIS, BUT IN DIFFERENT HEPATOCYTE POPULATIONS
Authors: ROBERTS RA SOAMES AR GILL JH JAMES NH WHEELDON EB
Citation: Ra. Roberts et al., NONGENOTOXIC HEPATOCARCINOGENS STIMULATE DNA-SYNTHESIS AND THEIR WITHDRAWAL INDUCES APOPTOSIS, BUT IN DIFFERENT HEPATOCYTE POPULATIONS, Carcinogenesis, 16(8), 1995, pp. 1693-1698
DOSING-INDUCED STRESS CAUSES HEPATOCYTE APOPTOSIS IN RATS PRIMED BY THE RODENT NONGENOTOXIC HEPATOCARCINOGEN CYPROTERONE-ACETATE
Authors: ROBERTS RA SOAMES AR JAMES NH GILL JH WHEELDON EB
Citation: Ra. Roberts et al., DOSING-INDUCED STRESS CAUSES HEPATOCYTE APOPTOSIS IN RATS PRIMED BY THE RODENT NONGENOTOXIC HEPATOCARCINOGEN CYPROTERONE-ACETATE, Toxicology and applied pharmacology, 135(2), 1995, pp. 192-199
THE PEROXISOME PROLIFERATOR CLASS OF NONGENOTOXIC HEPATOCARCINOGENS SYNERGIZE WITH EPIDERMAL GROWTH-FACTOR TO PROMOTE CLONAL EXPANSION OF INITIATED RAT HEPATOCYTES
Authors: JAMES NH ROBERTS RA
Citation: Nh. James et Ra. Roberts, THE PEROXISOME PROLIFERATOR CLASS OF NONGENOTOXIC HEPATOCARCINOGENS SYNERGIZE WITH EPIDERMAL GROWTH-FACTOR TO PROMOTE CLONAL EXPANSION OF INITIATED RAT HEPATOCYTES, Carcinogenesis, 15(12), 1994, pp. 2687-2694
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