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Results: 1-13 |
Results: 13
NATURAL-KILLER-CELLS FROM HUMAN-IMMUNODEFICIENCY-VIRUS (HIV)-INFECTEDINDIVIDUALS ARE AN IMPORTANT SOURCE OF CC-CHEMOKINES AND SUPPRESS HIV-1 ENTRY AND REPLICATION IN-VITRO
Authors: OLIVA A KINTER AL VACCAREZZA M RUBBERT A CATANZARO A MOIR S MONACO J EHLER L MIZELL S JACKSON R LI YX ROMANO JW FAUCI AS
Citation: A. Oliva et al., NATURAL-KILLER-CELLS FROM HUMAN-IMMUNODEFICIENCY-VIRUS (HIV)-INFECTEDINDIVIDUALS ARE AN IMPORTANT SOURCE OF CC-CHEMOKINES AND SUPPRESS HIV-1 ENTRY AND REPLICATION IN-VITRO, The Journal of clinical investigation, 102(1), 1998, pp. 223-231
HIV REPLICATION IN CD4(-CELLS OF HIV-INFECTED INDIVIDUALS IS REGULATED BY A BALANCE BETWEEN THE VIRAL SUPPRESSIVE EFFECTS OF ENDOGENOUS BETA-CHEMOKINES AND THE VIRAL INDUCTIVE EFFECTS OF OTHER ENDOGENOUS CYTOKINES() T)
Authors: KINTER AL OSTROWSKI M GOLETTI D OLIVA A WEISSMAN D GANTT K HARDY E JACKSON R EHLER L FAUCI AS
Citation: Al. Kinter et al., HIV REPLICATION IN CD4(-CELLS OF HIV-INFECTED INDIVIDUALS IS REGULATED BY A BALANCE BETWEEN THE VIRAL SUPPRESSIVE EFFECTS OF ENDOGENOUS BETA-CHEMOKINES AND THE VIRAL INDUCTIVE EFFECTS OF OTHER ENDOGENOUS CYTOKINES() T), Proceedings of the National Academy of Sciences of the United Statesof America, 93(24), 1996, pp. 14076-14081
MODULATION OF ENDOGENOUS IL-1-BETA AND IL-1 RECEPTOR ANTAGONIST RESULTS IN OPPOSING EFFECTS ON HIV EXPRESSION IN CHRONICALLY INFECTED MONOCYTIC CELLS
Authors: GOLETTI D KINTER AL HARDY EC POLI G FAUCI AS
Citation: D. Goletti et al., MODULATION OF ENDOGENOUS IL-1-BETA AND IL-1 RECEPTOR ANTAGONIST RESULTS IN OPPOSING EFFECTS ON HIV EXPRESSION IN CHRONICALLY INFECTED MONOCYTIC CELLS, The Journal of immunology, 156(9), 1996, pp. 3501-3508
INTERLEUKIN-2 INDUCES CD8(-CELL-MEDIATED SUPPRESSION OF HUMAN-IMMUNODEFICIENCY-VIRUS REPLICATION IN CD4(+) T-CELLS AND THIS EFFECT OVERRIDES ITS ABILITY TO STIMULATE VIRUS EXPRESSION() T)
Authors: KINTER AL BENDE SM HARDY EC JACKSON R FAUCI AS
Citation: Al. Kinter et al., INTERLEUKIN-2 INDUCES CD8(-CELL-MEDIATED SUPPRESSION OF HUMAN-IMMUNODEFICIENCY-VIRUS REPLICATION IN CD4(+) T-CELLS AND THIS EFFECT OVERRIDES ITS ABILITY TO STIMULATE VIRUS EXPRESSION() T), Proceedings of the National Academy of Sciences of the United Statesof America, 92(24), 1995, pp. 10985-10989
EFFECT OF CELLULAR-DIFFERENTIATION ON CYTOKINE-INDUCED EXPRESSION OF HUMAN-IMMUNODEFICIENCY-VIRUS IN CHRONICALLY INFECTED PROMONOCYTIC CELLS - DISSOCIATION OF CELLULAR-DIFFERENTIATION AND VIRAL EXPRESSION
Authors: GOLETTI D KINTER AL BISWAS P BENDE SM POLI G FAUCI AS
Citation: D. Goletti et al., EFFECT OF CELLULAR-DIFFERENTIATION ON CYTOKINE-INDUCED EXPRESSION OF HUMAN-IMMUNODEFICIENCY-VIRUS IN CHRONICALLY INFECTED PROMONOCYTIC CELLS - DISSOCIATION OF CELLULAR-DIFFERENTIATION AND VIRAL EXPRESSION, Journal of virology, 69(4), 1995, pp. 2540-2546
HIV REPLICATION IN IL-2-STIMULATED PERIPHERAL-BLOOD MONONUCLEAR-CELLSIS DRIVEN IN AN AUTOCRINE PARACRINE MANNER BY ENDOGENOUS CYTOKINES
Authors: KINTER AL POLI G FOX L HARDY E FAUCI AS
Citation: Al. Kinter et al., HIV REPLICATION IN IL-2-STIMULATED PERIPHERAL-BLOOD MONONUCLEAR-CELLSIS DRIVEN IN AN AUTOCRINE PARACRINE MANNER BY ENDOGENOUS CYTOKINES, The Journal of immunology, 154(5), 1995, pp. 2448-2459
CYTOKINE REGULATION OF ACUTE AND CHRONIC HIV-INFECTION IN-VITRO - FROM CELL-LINES TO PRIMARY MONONUCLEAR-CELLS
Authors: POLI G KINTER AL VICENZI E FAUCI AS
Citation: G. Poli et al., CYTOKINE REGULATION OF ACUTE AND CHRONIC HIV-INFECTION IN-VITRO - FROM CELL-LINES TO PRIMARY MONONUCLEAR-CELLS, Research in immunology, 145(8-9), 1994, pp. 578-582
IL-10 AND THE TH-2 CYTOKINE IL-4 REGULATE HIV REPLICATION IN HUMAN MACROPHAGES IN CONCERT WITH PROINFLAMMATORY CYTOKINES
Authors: POLI G WEISSMAN D KINTER AL FAUCI AS
Citation: G. Poli et al., IL-10 AND THE TH-2 CYTOKINE IL-4 REGULATE HIV REPLICATION IN HUMAN MACROPHAGES IN CONCERT WITH PROINFLAMMATORY CYTOKINES, AIDS research and human retroviruses, 10, 1994, pp. 190000071-190000071
ENDOGENOUS CYTOKINES DRIVE HIV-1 REPLICATION IN PRIMARY HUMAN PERIPHERAL-BLOOD MONONUCLEAR-CELLS (PBMC) STIMULATED WITH IL-2 IN THE ABSENCEOF MITOGENS
Authors: KINTER AL POLI G FAUCI AS
Citation: Al. Kinter et al., ENDOGENOUS CYTOKINES DRIVE HIV-1 REPLICATION IN PRIMARY HUMAN PERIPHERAL-BLOOD MONONUCLEAR-CELLS (PBMC) STIMULATED WITH IL-2 IN THE ABSENCEOF MITOGENS, AIDS research and human retroviruses, 10, 1994, pp. 190000076-190000076
INTERLEUKIN-1 INDUCES EXPRESSION OF THE HUMAN-IMMUNODEFICIENCY-VIRUS ALONE AND IN SYNERGY WITH INTERLEUKIN-6 IN CHRONICALLY INFECTED U1 CELLS - INHIBITION OF INDUCTIVE EFFECTS BY THE INTERLEUKIN-1 RECEPTOR ANTAGONIST
Authors: POLI G KINTER AL FAUCI AS
Citation: G. Poli et al., INTERLEUKIN-1 INDUCES EXPRESSION OF THE HUMAN-IMMUNODEFICIENCY-VIRUS ALONE AND IN SYNERGY WITH INTERLEUKIN-6 IN CHRONICALLY INFECTED U1 CELLS - INHIBITION OF INDUCTIVE EFFECTS BY THE INTERLEUKIN-1 RECEPTOR ANTAGONIST, Proceedings of the National Academy of Sciences of the United Statesof America, 91(1), 1994, pp. 108-112
CYTOKINE-DEPENDENT AND INDEPENDENT CONTROL OF HIV TRANSCRIPTION IN PERSISTENTLY INFECTED CELL-LINES
Authors: POLI G KINTER AL BISWAS P BRESSLER P CHIANG L STANLEY SK RABSON AB FAUCI AS
Citation: G. Poli et al., CYTOKINE-DEPENDENT AND INDEPENDENT CONTROL OF HIV TRANSCRIPTION IN PERSISTENTLY INFECTED CELL-LINES, Journal of acquired immune deficiency syndromes, 6(6), 1993, pp. 694-694
N-ACETYL-CYSTEINE (NAC) IS A POTENT SUPPRESSOR OF HIV TRANSCRIPTION IN PERSISTENTLY INFECTED MONOCYTIC CELLS
Authors: KINTER AL POLI G FAUCI AS
Citation: Al. Kinter et al., N-ACETYL-CYSTEINE (NAC) IS A POTENT SUPPRESSOR OF HIV TRANSCRIPTION IN PERSISTENTLY INFECTED MONOCYTIC CELLS, Journal of acquired immune deficiency syndromes, 6(6), 1993, pp. 707-707
RETINOIC ACID MIMICS TRANSFORMING GROWTH-FACTOR-BETA IN THE REGULATION OF HUMAN-IMMUNODEFICIENCY-VIRUS EXPRESSION IN MONOCYTIC CELLS
Authors: POLI G KINTER AL JUSTEMENT JS BRESSLER P KEHRL JH FAUCI AS
Citation: G. Poli et al., RETINOIC ACID MIMICS TRANSFORMING GROWTH-FACTOR-BETA IN THE REGULATION OF HUMAN-IMMUNODEFICIENCY-VIRUS EXPRESSION IN MONOCYTIC CELLS, Proceedings of the National Academy of Sciences of the United Statesof America, 89(7), 1992, pp. 2689-2693
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