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Results: 1-11 |
Results: 11
Evidence for neuroaxonal injury in patients with proteolipid gene mutations
Authors: Garbern, J Shy, M Krajewski, K Kamholz, J Hobson, G Cambi, F
Citation: J. Garbern et al., Evidence for neuroaxonal injury in patients with proteolipid gene mutations, NEUROLOGY, 57(10), 2001, pp. 1938-1939
Mutations in the cytoplasmic domain of P0 reveal a role for PKC-mediated phosphorylation in adhesion and myelination
Authors: Xu, WB Shy, M Kamholz, J Elferink, L Xu, G Lilien, J Balsamo, J
Citation: Wb. Xu et al., Mutations in the cytoplasmic domain of P0 reveal a role for PKC-mediated phosphorylation in adhesion and myelination, J CELL BIOL, 155(3), 2001, pp. 439-445
Cell cycle arrest induced by ectopic expression of p27 is not sufficient to promote oligodendrocyte differentiation
Authors: Tang, XM Beesley, JS Grinspan, JB Seth, P Kamholz, J Cambi, F
Citation: Xm. Tang et al., Cell cycle arrest induced by ectopic expression of p27 is not sufficient to promote oligodendrocyte differentiation, J CELL BIOC, 76(2), 2000, pp. 270-279
Gtx, an oligodendrocyte-specific homeodomain protein, has repressor activity
Authors: Awatramani, R Beesley, J Yang, HB Jiang, HY Cambi, F Grinspan, J Garbern, J Kamholz, J
Citation: R. Awatramani et al., Gtx, an oligodendrocyte-specific homeodomain protein, has repressor activity, J NEUROSC R, 61(4), 2000, pp. 376-387
GFAP-positive and myelin marker-positive glia in normal and pathologic environments
Authors: Dyer, CA Kendler, A Jean-Guillaume, D Awatramani, R Lee, A Mason, LM Kamholz, J
Citation: Ca. Dyer et al., GFAP-positive and myelin marker-positive glia in normal and pathologic environments, J NEUROSC R, 60(3), 2000, pp. 412-426
Proteolipid protein mRNA stability is regulated by axonal contact in the rodent peripheral nervous system
Authors: Jiang, HY Duchala, CS Awatramani, R Shumas, S Carlock, L Kamholz, J Garbern, J Scherer, SS Shy, ME Macklin, WB
Citation: Hy. Jiang et al., Proteolipid protein mRNA stability is regulated by axonal contact in the rodent peripheral nervous system, J NEUROBIOL, 44(1), 2000, pp. 7-19
Neurological dysfunction and axonal degeneration in Charcot-Marie-Tooth disease type 1A
Authors: Krajewski, KM Lewis, RA Fuerst, DR Turansky, C Hinderer, SR Garbern, J Kamholz, J Shy, ME
Citation: Km. Krajewski et al., Neurological dysfunction and axonal degeneration in Charcot-Marie-Tooth disease type 1A, BRAIN, 123, 2000, pp. 1516-1527
Charcot-Marie-Tooth disease type 1 - Molecular pathogenesis to gene therapy
Authors: Kamholz, J Menichella, D Jani, A Garbern, J Lewis, RA Krajewski, KM Lilien, J Scherer, SS Shy, ME
Citation: J. Kamholz et al., Charcot-Marie-Tooth disease type 1 - Molecular pathogenesis to gene therapy, BRAIN, 123, 2000, pp. 222-233
Additional copies of the proteolipid protein gene causing Pelizaeus-Merzbacher disease arise by separate integration into the X chromosome
Authors: Hodes, ME Woodward, K Spinner, NB Emanuel, BS Enrico-Simon, A Kamholz, J Stambolian, D Zackai, EH Pratt, VM Thomas, IT Crandall, K Dlouhy, SR Malcolm, S
Citation: Me. Hodes et al., Additional copies of the proteolipid protein gene causing Pelizaeus-Merzbacher disease arise by separate integration into the X chromosome, AM J HU GEN, 67(1), 2000, pp. 14-22
Modulation of cell-mediated immunity prolongs adenovirus-mediated transgene expression in sciatic nerve
Authors: Jani, A Menichella, D Jiang, HY Chbihi, T Acsadi, G Shy, ME Kamholz, J
Citation: A. Jani et al., Modulation of cell-mediated immunity prolongs adenovirus-mediated transgene expression in sciatic nerve, HUM GENE TH, 10(5), 1999, pp. 787-800
The molecular pathogenesis of Pelizaeus-Merzbacher disease
Authors: Garbern, J Cambi, F Shy, M Kamholz, J
Citation: J. Garbern et al., The molecular pathogenesis of Pelizaeus-Merzbacher disease, ARCH NEUROL, 56(10), 1999, pp. 1210-1214
Risultati: 1-11 |