Authors: Wan, XP Chen, SH Sadeghpour, A Wang, Q Kirsch, GE

Citation: Xp. Wan et al., Accelerated inactivation in a mutant Na+ channel associated with idiopathic ventricular fibrillation, AM J P-HEAR, 280(1), 2001, pp. H354-H360

Authors: Klemic, KG Kirsch, GE Jones, SW

Citation: Kg. Klemic et al., U-type inactivation of Kv3.1 and Shaker potassium channels, BIOPHYS J, 81(2), 2001, pp. 814-826

Authors: Wan, XP Wang, Q Kirsch, GE

Citation: Xp. Wan et al., Functional suppression of sodium channels by beta(1)-subunits as a molecular mechanism of idiopathic ventricular fibrillation, J MOL CEL C, 32(10), 2000, pp. 1873-1884

Authors: Brugada, R Brugada, J Antzelevitch, C Kirsch, GE Potenza, D Towbin, JA Brugada, P

Citation: R. Brugada et al., Sodium channel blockers identify risk for sudden death in patients with ST-Segment elevation and right bundle branch block but structurally normal hearts, CIRCULATION, 101(5), 2000, pp. 510-515

Authors: Kirsch, GE

Citation: Ge. Kirsch, Ion channel defects in primary electrical diseases of the heart, CHANNELOPATHIES - COMMON MECHANISMS IN AURA, ARRHYTHMIA AND ALKALOSIS, 2000, pp. 115-151

Authors: Kirsch, GE

Citation: Ge. Kirsch, Ion channel defects in cardiac arrhythmia, J MEMBR BIO, 170(3), 1999, pp. 181-190