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Citation: Ld. Vandekar et al., ICV INJECTION OF THE SEROTONIN 5-HT1B AGONIST CP-93,129 INCREASES THESECRETION OF ACTH, PROLACTIN, AND RENIN AND INCREASES BLOOD-PRESSURE BY NONSEROTONERGIC MECHANISMS, Pharmacology, biochemistry and behavior, 48(2), 1994, pp. 429-436
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Citation: Q. Li et al., ATTENUATION OF HORMONE RESPONSES TO THE 5-HT1A AGONIST IPSAPIRONE BY LONG-TERM TREATMENT WITH FLUOXETINE, BUT NOT DESIPRAMINE, IN MALE-RATS, Biological psychiatry, 36(5), 1994, pp. 300-308
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Citation: Ad. Levy et al., REPEATED EXPOSURE TO COCAINE PRODUCES LONG-LASTING DEFICITS IN THE SEROTONERGIC STIMULATION OF PROLACTIN AND RENIN, BUT NOT ADRENOCORTICOTROPIN SECRETION, European journal of pharmacology, 241(2-3), 1993, pp. 275-278
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Citation: Pa. Rittenhouse et al., NEURONS IN THE HYPOTHALAMIC PARAVENTRICULAR NUCLEUS MEDIATE THE SEROTONERGIC STIMULATION OF PROLACTIN SECRETION VIA 5-HT1C 2 RECEPTORS/, Endocrinology, 133(2), 1993, pp. 661-667
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Citation: Q. Li et al., LONG-TERM FLUOXETINE, BUT NOT DESIPRAMINE, INHIBITS THE ACTH AND OXYTOCIN RESPONSES TO THE 5-HT(1A) AGONIST, 8-OH-DPAT, IN MALE-RATS, Brain research, 630(1-2), 1993, pp. 148-156