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Results: 1-10 |
Results: 10
Autonomic regulation of neuroimmunological responses: Implications for multiple sclerosis
Authors: Frohman, EM Monson, NL Lovett-Racke, AE Racke, MK
Citation: Em. Frohman et al., Autonomic regulation of neuroimmunological responses: Implications for multiple sclerosis, J CLIN IMM, 21(2), 2001, pp. 61-73
Role of nerve growth factor in experimental autoimmune encephalomyelitis
Authors: Arredondo, LR Deng, CS Ratts, RB Lovett-Racke, AE Holtzman, DM Racke, MK
Citation: Lr. Arredondo et al., Role of nerve growth factor in experimental autoimmune encephalomyelitis, EUR J IMMUN, 31(2), 2001, pp. 625-633
Developmentally regulated gene expression of Th2 cytokines in the brain
Authors: Lovett-Racke, AE Smith, ME Arredondo, LR Bittner, PS Ratts, RB Shive, CL Forsthuber, TG Racke, MK
Citation: Ae. Lovett-racke et al., Developmentally regulated gene expression of Th2 cytokines in the brain, BRAIN RES, 870(1-2), 2000, pp. 27-35
The role of CTLA-4 in tolerance induction and T cell differentiation in experimental autoimmune encephalomyelitis: i.p. antigen administration
Authors: Ratts, RB Arredondo, LR Bittner, P Perrin, PJ Lovett-Racke, AE Racke, MK
Citation: Rb. Ratts et al., The role of CTLA-4 in tolerance induction and T cell differentiation in experimental autoimmune encephalomyelitis: i.p. antigen administration, INT IMMUNOL, 11(12), 1999, pp. 1881-1888
The role of CTLA-4 in tolerance induction and T cell differentiation in experimental autoimmune encephalomyelitis: i.v. antigen administration
Authors: Ratts, RB Arredondo, LR Bittner, P Perrin, PJ Lovett-Racke, AE Racke, MK
Citation: Rb. Ratts et al., The role of CTLA-4 in tolerance induction and T cell differentiation in experimental autoimmune encephalomyelitis: i.v. antigen administration, INT IMMUNOL, 11(12), 1999, pp. 1889-1895
Bystander suppression in experimental autoimmune encephalomyelitis: where and how does it occur?
Authors: Racke, MK Lovett-Racke, AE
Citation: Mk. Racke et Ae. Lovett-racke, Bystander suppression in experimental autoimmune encephalomyelitis: where and how does it occur?, RES IMMUNOL, 149(9), 1998, pp. 820-827
Antigen-driven regulation of experimental autoimmune encephalomyelitis - Discussion
Authors: Begolka, WS Miller, SD Segal, BM Racke, MK Lovett-Racke, AE
Citation: Ws. Begolka et al., Antigen-driven regulation of experimental autoimmune encephalomyelitis - Discussion, RES IMMUNOL, 149(9), 1998, pp. 842-843
Cytokines as intrinsic and exogenous regulators of pathogenesis in experimental encephalomyelitis - Discussion
Authors: Willenborg, DO Racke, MK Lovett-Racke, AE Hoffman, LM Karpus, WJ Owens, T
Citation: Do. Willenborg et al., Cytokines as intrinsic and exogenous regulators of pathogenesis in experimental encephalomyelitis - Discussion, RES IMMUNOL, 149(9), 1998, pp. 843-844
Immune cell entry to the CNS - a focus for immunoregulation of EAE - Discussion
Authors: Segal, BM Willenborg, DO Racke, MK Lovett-Racke, AE Hoffman, LM Karpus, WJ Begolka, WS Miller, SD
Citation: Bm. Segal et al., Immune cell entry to the CNS - a focus for immunoregulation of EAE - Discussion, RES IMMUNOL, 149(9), 1998, pp. 844-846
Tumour necrosis factor-alpha and lymphotoxin-alpha in the pathology of experimental autoimmune encephalomyelitis: is either one responsible or is there another ligand-mediating disease? Discussion
Authors: Owens, T Segal, BM Racke, MK Lovett-Racke, AE Begolka, WS Miller, SD
Citation: T. Owens et al., Tumour necrosis factor-alpha and lymphotoxin-alpha in the pathology of experimental autoimmune encephalomyelitis: is either one responsible or is there another ligand-mediating disease? Discussion, RES IMMUNOL, 149(9), 1998, pp. 849-850
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