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Results: 1-11 |
Results: 11
CHEMOTAXIS OF CHINESE-HAMSTER OVARY CELLS EXPRESSING THE HUMAN NEUTROPHIL FORMYL PEPTIDE RECEPTOR
Authors: MIETTINEN HM GRIPENTROG JM JESAITIS AJ
Citation: Hm. Miettinen et al., CHEMOTAXIS OF CHINESE-HAMSTER OVARY CELLS EXPRESSING THE HUMAN NEUTROPHIL FORMYL PEPTIDE RECEPTOR, Journal of leukocyte biology, 1998, pp. 137-137
CHEMOTAXIS OF CHINESE-HAMSTER OVARY CELLS EXPRESSING THE HUMAN NEUTROPHIL FORMYL PEPTIDE RECEPTOR - ROLE OF SIGNAL-TRANSDUCTION MOLECULES AND ALPHA(5)BETA(1) INTEGRIN
Authors: MIETTINEN HM GRIPENTROG JM JESAITIS AJ
Citation: Hm. Miettinen et al., CHEMOTAXIS OF CHINESE-HAMSTER OVARY CELLS EXPRESSING THE HUMAN NEUTROPHIL FORMYL PEPTIDE RECEPTOR - ROLE OF SIGNAL-TRANSDUCTION MOLECULES AND ALPHA(5)BETA(1) INTEGRIN, Journal of Cell Science, 111, 1998, pp. 1921-1928
IDENTIFICATION OF A LIGAND-BINDING SITE IN THE HUMAN NEUTROPHIL FORMYL PEPTIDE RECEPTOR USING A SITE-SPECIFIC FLUORESCENT PHOTOAFFINITY LABEL AND MASS-SPECTROMETRY
Authors: MILLS JS MIETTINEN HM BARNIDGE D VLASES MJ WIMERMACKIN S DRATZ EA SUNNER J JESAITIS AJ
Citation: Js. Mills et al., IDENTIFICATION OF A LIGAND-BINDING SITE IN THE HUMAN NEUTROPHIL FORMYL PEPTIDE RECEPTOR USING A SITE-SPECIFIC FLUORESCENT PHOTOAFFINITY LABEL AND MASS-SPECTROMETRY, The Journal of biological chemistry, 273(17), 1998, pp. 10428-10435
ENHANCED NADPH OXIDASE ACTIVITY IN NEUTROPHILS ISOLATED FROM CATTLE WITH BOVINE LEUKOCYTE ADHESION DEFICIENCY
Authors: SIPES KM EDENS H KEHRLI M CUTLER JE MIETTINEN HM JUTILA MA QUINN MT
Citation: Km. Sipes et al., ENHANCED NADPH OXIDASE ACTIVITY IN NEUTROPHILS ISOLATED FROM CATTLE WITH BOVINE LEUKOCYTE ADHESION DEFICIENCY, Journal of allergy and clinical immunology, 99(1), 1997, pp. 1861-1861
THE LIGAND-BINDING SITE OF THE FORMYL PEPTIDE RECEPTOR MAPS IN THE TRANSMEMBRANE REGION
Authors: MIETTINEN HM MILLS JS GRIPENTROG JM DRATZ EA GRANGER BL JESAITIS AJ
Citation: Hm. Miettinen et al., THE LIGAND-BINDING SITE OF THE FORMYL PEPTIDE RECEPTOR MAPS IN THE TRANSMEMBRANE REGION, The Journal of immunology, 159(8), 1997, pp. 4045-4054
MUTAGENESIS ANALYSIS OF THE INTERACTION BETWEEN FORMYL PEPTIDE RECEPTOR, N-FORMYLATED PEPTIDE, AND G-PROTEIN IN TRANSFECTED FIBROBLASTS
Authors: MIETTINEN HM GRIPENTROG JM JESAITIS AJ
Citation: Hm. Miettinen et al., MUTAGENESIS ANALYSIS OF THE INTERACTION BETWEEN FORMYL PEPTIDE RECEPTOR, N-FORMYLATED PEPTIDE, AND G-PROTEIN IN TRANSFECTED FIBROBLASTS, Molecular biology of the cell, 6, 1995, pp. 326-326
ANALYSIS OF TRANSPORT AND TARGETING OF SYNDECAN-1 - EFFECT OF CYTOPLASMIC TAIL DELETIONS
Authors: MIETTINEN HM EDWARDS SN JALKANEN M
Citation: Hm. Miettinen et al., ANALYSIS OF TRANSPORT AND TARGETING OF SYNDECAN-1 - EFFECT OF CYTOPLASMIC TAIL DELETIONS, Molecular biology of the cell, 5(12), 1994, pp. 1325-1339
EXPRESSION OF SYNDECAN-1 IS INDUCED BY DIFFERENTIATION AND SUPPRESSEDBY MALIGNANT TRANSFORMATION OF HUMAN KERATINOCYTES
Authors: INKI P LARJAVA H HAAPASALMI K MIETTINEN HM GRENMAN R JALKANEN M
Citation: P. Inki et al., EXPRESSION OF SYNDECAN-1 IS INDUCED BY DIFFERENTIATION AND SUPPRESSEDBY MALIGNANT TRANSFORMATION OF HUMAN KERATINOCYTES, European journal of cell biology, 63(1), 1994, pp. 43-51
THE CYTOPLASMIC DOMAIN OF SYNDECAN-1 IS NOT REQUIRED FOR ASSOCIATION WITH TRITON X-100-INSOLUBLE MATERIAL
Authors: MIETTINEN HM JALKANEN M
Citation: Hm. Miettinen et M. Jalkanen, THE CYTOPLASMIC DOMAIN OF SYNDECAN-1 IS NOT REQUIRED FOR ASSOCIATION WITH TRITON X-100-INSOLUBLE MATERIAL, Journal of Cell Science, 107, 1994, pp. 1571-1581
SUPPRESSION OF TUMOR-CELL GROWTH BY SYNDECAN-1 ECTODOMAIN
Authors: MALI M ANDTFOLK H MIETTINEN HM JALKANEN M
Citation: M. Mali et al., SUPPRESSION OF TUMOR-CELL GROWTH BY SYNDECAN-1 ECTODOMAIN, The Journal of biological chemistry, 269(45), 1994, pp. 27795-27798
INHIBITION OF BASIC FIBROBLAST GROWTH FACTOR-INDUCED GROWTH PROMOTIONBY OVEREXPRESSION OF SYNDECAN-1
Authors: MALI M ELENIUS K MIETTINEN HM JALKANEN M
Citation: M. Mali et al., INHIBITION OF BASIC FIBROBLAST GROWTH FACTOR-INDUCED GROWTH PROMOTIONBY OVEREXPRESSION OF SYNDECAN-1, The Journal of biological chemistry, 268(32), 1993, pp. 24215-24222
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