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Results:
1-8
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Results: 8
Authors:
Welsh, DC
Dipla, K
McNulty, PH
Mu, AB
Ojamaa, KM
Klein, I
Houser, SR
Margulies, KB
Citation: Dc. Welsh et al., Preserved contractile function despite atrophic remodeling in unloaded rathearts, AM J P-HEAR, 281(3), 2001, pp. H1131-H1136
Authors:
Kubo, H
Margulies, KB
Piacentino, V
Gaughan, JP
Houser, SR
Citation: H. Kubo et al., Patients with end-stage congestive heart failure treated with beta-adrenergic receptor antagonists have improved ventricular myocyte calcium regulatory protein abundance, CIRCULATION, 104(9), 2001, pp. 1012-1018
Authors:
Flesch, M
Margulies, KB
Mochmann, HC
Engel, D
Sivasubramanian, N
Mann, DL
Citation: M. Flesch et al., Differential regulation of mitogen-activated protein kinases in the failing human heart in response to mechanical unloading, CIRCULATION, 104(19), 2001, pp. 2273-2276
Authors:
Harding, JD
Piacentino, V
Gaughan, JP
Houser, SR
Margulies, KB
Citation: Jd. Harding et al., Electrophysiological alterations after mechanical circulatory support in patients with advanced cardiac failure, CIRCULATION, 104(11), 2001, pp. 1241-1247
Authors:
Margulies, KB
Jaffer, S
Pollack, PS
Ennis, KJ
Citation: Kb. Margulies et al., Physiological significance of early deceleration time prolongation in asymptomatic elderly subjects, J CARD FAIL, 5(2), 1999, pp. 92-99
Authors:
Gaughan, JP
Furukawa, S
Jeevanandam, V
Hefner, CA
Kubo, H
Margulies, KB
McGowan, BS
Mattiello, JA
Dipla, K
Piacentino, V
Li, SY
Houser, SR
Citation: Jp. Gaughan et al., Sodium/calcium exchange contributes to contraction and relaxation in failed human ventricular myocytes, AM J P-HEAR, 46(2), 1999, pp. H714-H724
Authors:
Dipla, K
Mattiello, JA
Margulies, KB
Jeevanandam, V
Houser, SR
Citation: K. Dipla et al., The sarcoplasmic reticulum and the Na+Ca2+ exchanger both contribute to the Ca2+ transient of failing human ventricular myocytes, CIRCUL RES, 84(4), 1999, pp. 435-444
Authors:
Margulies, KB
Dipla, K
Mattiello, JA
Houser, SR
Jeevanandam, V
Citation: Kb. Margulies et al., Atrophy of myocardium and its myocytes by left ventricular assist device -Response, CIRCULATION, 100(9), 1999, pp. 1012-1012
Risultati:
1-8
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