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Results: 1-8 |
Results: 8
Molecular analysis of the interaction of LCMV with its cellular receptor alpha-dystroglycan
Authors: Kunz, S Sevilla, N McGavern, DB Campbell, KP Oldstone, MBA
Citation: S. Kunz et al., Molecular analysis of the interaction of LCMV with its cellular receptor alpha-dystroglycan, J CELL BIOL, 155(2), 2001, pp. 301-310
Spontaneous remyelination following extensive demyelination is associated with improved neurological function in a viral model of multiple sclerosis
Authors: Murray, PD McGavern, DB Sathornsumetee, S Rodriguez, M
Citation: Pd. Murray et al., Spontaneous remyelination following extensive demyelination is associated with improved neurological function in a viral model of multiple sclerosis, BRAIN, 124, 2001, pp. 1403-1416
HLA-DQ polymorphism influences progression of demyelination and neurologicdeficits in a viral model of multiple sclerosis
Authors: Pavelko, KD Drescher, KM McGavern, DB David, CS Rodriguez, M
Citation: Kd. Pavelko et al., HLA-DQ polymorphism influences progression of demyelination and neurologicdeficits in a viral model of multiple sclerosis, MOL CELL NE, 15(6), 2000, pp. 495-509
Assessment of hindlimb gait as a powerful indicator of axonal loss in a murine model of progressive CNS demyelination
Authors: McGavern, DB Zoecklein, L Sathornsumetee, S Rodriguez, M
Citation: Db. Mcgavern et al., Assessment of hindlimb gait as a powerful indicator of axonal loss in a murine model of progressive CNS demyelination, BRAIN RES, 877(2), 2000, pp. 396-400
Axonal loss results in spinal-cord atrophy, electrophysiological abnormalities and neurological deficits following demyelination in a chronic inflammatory model of multiple sclerosis
Authors: McGavern, DB Murray, PD Rivera-Quinones, C Schmelzer, JD Low, PA Rodriguez, M
Citation: Db. Mcgavern et al., Axonal loss results in spinal-cord atrophy, electrophysiological abnormalities and neurological deficits following demyelination in a chronic inflammatory model of multiple sclerosis, BRAIN, 123, 2000, pp. 519-531
Quantitative ultrastructural analysis of a single spinal cord demyelinatedlesion predicts total lesion load, axonal loss, and neurological dysfunction in a murine model of multiple sclerosis
Authors: Sathornsumetee, S McGavern, DB Ure, DR Rodriguez, M
Citation: S. Sathornsumetee et al., Quantitative ultrastructural analysis of a single spinal cord demyelinatedlesion predicts total lesion load, axonal loss, and neurological dysfunction in a murine model of multiple sclerosis, AM J PATH, 157(4), 2000, pp. 1365-1376
Quantitation of spinal cord demyelination, remyelination, atrophy, and axonal loss in a model of progressive neurologic injury
Authors: McGavern, DB Murray, PD Rodriguez, M
Citation: Db. Mcgavern et al., Quantitation of spinal cord demyelination, remyelination, atrophy, and axonal loss in a model of progressive neurologic injury, J NEUROSC R, 58(4), 1999, pp. 492-504
Quantitative assessment of neurologic deficits in a chronic progressive murine model of CNS demyelination
Authors: McGavern, DB Zoecklein, L Drescher, KM Rodriguez, M
Citation: Db. Mcgavern et al., Quantitative assessment of neurologic deficits in a chronic progressive murine model of CNS demyelination, EXP NEUROL, 158(1), 1999, pp. 171-181
Risultati: 1-8 |