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Results: 1-18 |
Results: 18
hGFAP-cre transgenic mice for manipulation of glial and neuronal function in vivo
Authors: Zhuo, L Theis, M Alvarez-Maya, I Brenner, M Willecke, K Messing, A
Citation: L. Zhuo et al., hGFAP-cre transgenic mice for manipulation of glial and neuronal function in vivo, GENESIS, 31(2), 2001, pp. 85-94
Mutations in GFAP, encoding glial fibrillary acidic protein, are associated with Alexander disease
Authors: Brenner, M Johnson, AB Boespflug-Tanguy, O Rodriguez, D Goldman, JE Messing, A
Citation: M. Brenner et al., Mutations in GFAP, encoding glial fibrillary acidic protein, are associated with Alexander disease, NAT GENET, 27(1), 2001, pp. 117-120
Update on white matter genetic disorders
Authors: Messing, A Brenner, M Johnson, AB Goldman, JE
Citation: A. Messing et al., Update on white matter genetic disorders, PED NEUROL, 25(4), 2001, pp. 347-348
Alexander disease: New insights from genetics
Authors: Messing, A Goldman, JE Johnson, AB Brenner, M
Citation: A. Messing et al., Alexander disease: New insights from genetics, J NE EXP NE, 60(6), 2001, pp. 563-573
Infantile Alexander disease: Spectrum of GFAP mutations and genotype-phenotype correlation
Authors: Rodriguez, D Gauthier, F Bertini, E Bugiani, M Brenner, M N'guyen, S Goizet, C Gelot, A Surtees, R Pedespan, JM Hernandorena, X Troncoso, M Uziel, G Messing, A Ponsot, G Pham-Dinh, D Dautigny, A Boespflug-Tanguy, O
Citation: D. Rodriguez et al., Infantile Alexander disease: Spectrum of GFAP mutations and genotype-phenotype correlation, AM J HU GEN, 69(5), 2001, pp. 1134-1140
Epitope-tagged P-0 glycoprotein causes Charcot-Marie-Tooth-like neuropathyin transgenic mice
Authors: Previtali, SC Quattrini, A Fasolini, M Panzeri, MC Villa, A Filbin, MT Li, WH Chiu, SY Messing, A Wrabetz, L Feltri, ML
Citation: Sc. Previtali et al., Epitope-tagged P-0 glycoprotein causes Charcot-Marie-Tooth-like neuropathyin transgenic mice, J CELL BIOL, 151(5), 2000, pp. 1035-1045
Schwann cell myelination requires timely and precise targeting of P-0 protein
Authors: Yin, X Kidd, GJ Wrabetz, L Feltri, ML Messing, A Trapp, BD
Citation: X. Yin et al., Schwann cell myelination requires timely and precise targeting of P-0 protein, J CELL BIOL, 148(5), 2000, pp. 1009-1020
P-0 glycoprotein overexpression causes congenital hypomyelination of peripheral nerves
Authors: Wrabetz, L Feltri, ML Quattrini, A Imperiale, D Previtali, S D'Antonio, M Martini, R Yin, XH Trapp, BD Zhou, L Chiu, SY Messing, A
Citation: L. Wrabetz et al., P-0 glycoprotein overexpression causes congenital hypomyelination of peripheral nerves, J CELL BIOL, 148(5), 2000, pp. 1021-1033
The Golgi apparatus is present in perisynaptic, subependymal and perivascular processes of astrocytes and in processes of retinal Muller glia
Authors: Gonatas, JO Stieber, A Gonatas, NK Messing, A
Citation: Jo. Gonatas et al., The Golgi apparatus is present in perisynaptic, subependymal and perivascular processes of astrocytes and in processes of retinal Muller glia, BRAIN RES, 855(1), 2000, pp. 23-31
A novel P-0 glycoprotein transgene activates expression of lacZ in myelin-forming Schwann cells
Authors: Feltri, ML D'Antonio, M Quattrini, A Numerato, R Arona, M Previtali, S Chiu, SY Messing, A Wrabetz, L
Citation: Ml. Feltri et al., A novel P-0 glycoprotein transgene activates expression of lacZ in myelin-forming Schwann cells, EUR J NEURO, 11(5), 1999, pp. 1577-1586
Glial fibrillary acidic protein is necessary for mature astrocytes to react to beta-amyloid
Authors: Xu, KX Malouf, AT Messing, A Silver, J
Citation: Kx. Xu et al., Glial fibrillary acidic protein is necessary for mature astrocytes to react to beta-amyloid, GLIA, 25(4), 1999, pp. 390-403
Nonlethal detection of transgene expression in the CNS of founder mice
Authors: Wakabayashi, T Messing, A Brenner, M
Citation: T. Wakabayashi et al., Nonlethal detection of transgene expression in the CNS of founder mice, BIOTECHNIQU, 26(2), 1999, pp. 302
Myelinating Schwann cells determine the internodal localization of Kv1.1, Kv1.2, Kv beta 2, and Caspr
Authors: Arroyo, EJ Xu, YT Zhou, L Messing, A Peles, E Chiu, SY Scherer, SS
Citation: Ej. Arroyo et al., Myelinating Schwann cells determine the internodal localization of Kv1.1, Kv1.2, Kv beta 2, and Caspr, J NEUROCYT, 28(4-5), 1999, pp. 333-347
Analysis of potassium channel functions in mammalian axons by gene knockouts
Authors: Chiu, SY Zhou, L Zhang, CL Messing, A
Citation: Sy. Chiu et al., Analysis of potassium channel functions in mammalian axons by gene knockouts, J NEUROCYT, 28(4-5), 1999, pp. 349-364
Nestin in the liver - Lessons from the brain
Authors: Messing, A
Citation: A. Messing, Nestin in the liver - Lessons from the brain, HEPATOLOGY, 29(2), 1999, pp. 602-603
Specific alteration of spontaneous GABAergic inhibition in cerebellar Purkinje cells in mice lacking the potassium channel Kv1.1
Authors: Zhang, CL Messing, A Chiu, SY
Citation: Cl. Zhang et al., Specific alteration of spontaneous GABAergic inhibition in cerebellar Purkinje cells in mice lacking the potassium channel Kv1.1, J NEUROSC, 19(8), 1999, pp. 2852-2864
Determinants of excitability at transition zones in Kv1.1-deficient myelinated nerves
Authors: Zhou, L Messing, A Chiu, SY
Citation: L. Zhou et al., Determinants of excitability at transition zones in Kv1.1-deficient myelinated nerves, J NEUROSC, 19(14), 1999, pp. 5768-5781
Studies in transgenic mice indicate a loss of connexin32 function in X-linked Charcot-Marie-tooth disease
Authors: Abel, A Bone, LJ Messing, A Scherer, SS Fischbeck, KH
Citation: A. Abel et al., Studies in transgenic mice indicate a loss of connexin32 function in X-linked Charcot-Marie-tooth disease, J NE EXP NE, 58(7), 1999, pp. 702-710
Risultati: 1-18 |