Authors:
Mangione, P
Sunde, M
Giorgetti, S
Stoppini, M
Esposito, G
Gianelli, L
Obici, L
Asti, L
Andreola, A
Viglino, P
Merlini, G
Bellotti, V
Citation: P. Mangione et al., Amyloid fibrils derived from the apolipoprotein A1 Leu174Ser variant contain elements of ordered helical structure, PROTEIN SCI, 10(1), 2001, pp. 187-199
Authors:
Palladini, G
Anesi, E
Perfetti, V
Obici, L
Invernizzi, R
Balduini, C
Ascari, E
Merlini, G
Citation: G. Palladini et al., A modified high-dose dexamethasone regimen for primary systemic (AL) amyloidosis, BR J HAEM, 113(4), 2001, pp. 1044-1046
Authors:
Ferlini, A
Obici, L
Manzati, E
Biadi, O
Tarantino, E
Conigli, P
Merlini, G
D'Alessandro, M
Mazzaferro, V
Tassinari, CA
Salvi, F
Citation: A. Ferlini et al., Mutation and transcription analysis of transthyretin gene in Italian families with hereditary amyloidosis: a putative novel 'hot spot' in codon 47, CLIN GENET, 57(4), 2000, pp. 284-290
Authors:
Torelli, S
Ferlini, A
Obici, L
Sewry, C
Muntoni, F
Citation: S. Torelli et al., Expression, regulation and localisation of dystrophin isoforms in human foetal skeletal and cardiac muscle, NEUROMUSC D, 9(8), 1999, pp. 541-551
Authors:
Obici, L
Bellotti, V
Mangione, P
Stoppini, M
Arbustini, E
Verga, L
Zorzoli, I
Anesi, E
Zanotti, G
Campana, C
Vigano, M
Merlini, G
Citation: L. Obici et al., The new apolipoprotein A-I variant Leu(174) -> Ser causes hereditary cardiac amyloidosis, and the amyloid fibrils are constituted by the 93-residue N-terminal polypeptide, AM J PATH, 155(3), 1999, pp. 695-702